Production of a small subgenomic RNA in flavivirus-infected cells is this an example of virus-encoded molecules modifying signal transduction pathways to promote replication?
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Production of a small subgenomic RNA in flavivirus-infected cells is this an example of virus-encoded molecules modifying signal transduction pathways to promote replication?
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- Transduction: Describe the process of generalized transduction as a type of horizontal transfer. In your response be sure to address the following:a. What is the role of the bacteriophage (bacterial virus) is in this scenario?Question:- Transformation of host cells by several DNA viruses requires inactivation of the cellular proteins pRB, CKI ( cyclin-dependent kinase inhibitor) and p53. give an example of how each of these cellular proteins can be inactivated by DNA viruses. ( one example/cellular protein)What low molecular weight protiens are produced by a hot cell in response to a viral infection that protects a pre-infected cell from a viral infected
- Is the Degradation of Xrn1 in poliovirus-infected cells an example of virus-encoded molecules modifying signal transduction pathways to promote replication?Cytopathic effect on susceptible cells is an important way to detect viral production. Regarding the topic, describe the cytopathic observations of at least three viruses (Human papillomavirus infection, Cytomegalovirus and Herpes simplex).The transcriptional complement of the DNA strand with the code 3’ TAA-CAT-GCT 5’ is
- Explain the role receptor-mediated endocytosis (RME) plays in virus replication (lytic cycle). If you worked for a pharmaceutical company and wanted to develop a vaccine to prevent viral replication, how could you use your knowledge of RME to your advantage?Discussions on the relationship between amino acid sequence and protein structure, why is testing known inhibitors of viral proteases from other viruses a logical first step in identifying drugs that have efficacy against the Sars-CoV-2 main protease? https://www.nature.com/articles/s41467-021-20900-6please help explain Influenza A virus M2 protein triggers mitochondrial DNA-mediated antiviral immune responses Cytosolic mitochondrial DNA (mtDNA) activates cGAS-mediated antiviral immune responses, but the mechanism by which RNA viruses stimulate mtDNA release remains unknown. Here we show that viroporin activity of influenza virus M2 or encephalomyocarditis virus (EMCV) 2B protein triggers translocation of mtDNA into the cytosol in a MAVS-dependent manner. Although influenza virus-induced cytosolic mtDNA stimulates cGAS- and DDX41- dependent innate immune responses, the nonstructural protein 1 (NS1) of influenza virus associates with mtDNA to evade the STING-dependent antiviral immunity. The STINGdependent antiviral signaling is amplified in neighboring cells through gap junctions. In addition, we find that STING-dependent recognition of influenza virus is essential for limiting virus replication in vivo. Our results show a mechanism by which influenza virus stimulates mtDNA release…
- Effects of BPA on phosphorylation of MAPKfamily in RAW264.7 cells conclusionSerotonin pathway of tryptophan transformation.You are studying a caspase cleavage site in an N protein from a new coronavirus you make a D/E site-directed mutation at this cleavage site and find that the Mutant protein is uncleaved during virus-induced apoptosis. Thus the aspartic acid is: a negative regulator of cleavage irrelevant for cleavage necessary for cleavage sufficient for cleavage