arguementative research paper

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Psychology

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Feb 20, 2024

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11/17/2018 Professor Koski FYW 1000C Adam McGowan Argumentative research paper As the brain deteriorates, the most common causes of this being age, brain damage, and Alzheimer's disease ( progressive mental deterioration that can occur in middle or old age, due to generalized degeneration of the brain. It is the most common cause of premature senility) , which causes memory loss, weakening the synapses in the brain. Without proper treatment the aging brain has a difficult time controlling the various part of the body. The brain begins to create fewer neurons, sending less impulses, significantly slowing down the various parts of the brain such as the prefrontal cortex, visual cortex, and the frontal cortex, causing neural plasticity through the use of BDNF occur. Brain derived neurotrophic factor (BDNF) is a protein found all throughout the body, with high concentrations found in the spine, brain and lower leg muscles ( Gomez-Pinilla, F.) . BDNF is a mechanism type protein found in the brain and spinal cord. BDNF can be used to assess changes in the brain. The rate of BDNF production can be increased by, cognition training, and various exercises that affect the brain, and physical exercise.

Because of these devices, many researchers believe that exercise can influence Alzheimer’s disease positively. Based off of this claim, the opinion of this paper has leaned on exercise positively affecting Alzheimer’s patients due to research done on three main objectives; how exercise affects the brain directly, how exercise affects brain devices such as Neural plasticity and the BDNF protein, and how exercise affects memory, seeing as Alzheimer’s main symptoms is memory loss, and cognitive disfunction. Research done shows that when exercise is done, there is a spike of neural activity in the brain, and an increase of growth and development of neurons and synapses ( Adlard, P. A., & Cotman). The research also shows that exercise increases BDNF levels, and the BDNF increases the length of synapses and strengthens neurons. Research done on the effect of BDNF on Alzheimer’s disease shows that BDNF releases inhibitors of the enzyme acetylcholinesterase. This inhibitor is crucial for treating Alzheimer’s disease (Leyhe, Stransky, Eschweiler, Buchkremer, & Laske, 2007). Research was also done to observe the effect of BDNF in schizophrenia patients. The research showed that the elevation of BDNF levels in the body decreased the pathogenesis of schizophrenia ( Vinogradov, S., Fisher, M., Holland, C., Shelly, W). With knowledge, scientists have concluded that using exercise to enhance BDNF, neural plasticity, and hippocampal neurogenesis, will positively affect the brain, and can drastically improve brain health. The question being tried to resolve is whether different types of exercise such as cardiovascular exercise can have a different effect on BDNF levels than endurance exercise. Based on this data, it can be determined that in certain cases, exercise has influenced Alzheimer’s disease. The study shows that exercise has a positive influence on BDNF levels, and therefore, the brain can benefit from exercise Just one study is not enough to cement a singular claim of exercise being beneficial for brain devices. Therefore, another study done in 2008 was to research the effect that exercise had

on BDNF, and if it can promote brain activity in the brains of patients with Alzheimer’s disease. Their data showed the BDNF levels in rats that exercise, by about 50% (Buchkremer, & Laske, 2007). A similar study done to demonstrate that voluntary exercise improves learning and neurogenesis in aged mice found slightly similar results (Buchkremer, & Laske, 2007). This study took multiple groups of mice, including two control groups of mice in order to test as the title states, the benefits of exercise on learning and neurogenesis. The study showed that the young running group of mice took the shortest time to complete the maze and the fastest running time for the wheel compared to the other groups including the young control group. (Buchkremer, & Laske, 2007) This means that according to how the data was analyzed, the exercise increased the levels of BDNF. The data also showed that the young running group had the most enhancement of neurogenesis in comparison to the old running group by their speed of completing the wheel and the maze. This shows that exercise is an excellent enhancer for neurogenesis. Neurogenesis deals directly with memory and brain function, therefore, exercise has a positive effect on Brain functions and memory as well. A similar study done to demonstrate that voluntary exercise improves learning and neurogenesis, (a key factor in this argument) in aged mice found slightly similar results (Buchkremer, & Laske, 2007). This study took multiple groups of mice, including two control groups of mice in order to test as the title states, the benefits of exercise on learning and neurogenesis. The study showed that the young running group of mice took the shortest time to complete the maze and the fastest running time for the wheel compared to the other groups including the young control group. (Buchkremer, & Laske, 2007) This means that according to how the data was analyzed, the exercise increased the levels of BDNF. The data also showed that the young running group had the most enhancement of neurogenesis in comparison to the old

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