A Brief Look at Gangrene

The septicemic version is defined as a fast growing bacteria which if not treated properly and fast enough would eventually lead to death. What would happen inside the body would be that the bacteria would disable your body’s ability from being able to clot blood. The infected would become very itchy all over their body to the point where they made themselves bleed from everywhere. Eventually the victim would have massive blood loss leading to

Gangrene is a term that is used to identify when a large amount of tissue undergoes cell death (necrosis). Gangrene essentially means death of tissues due to lack of blood supply and invasion of deeper tissues with infection (Porth & Gaspard, 2015). There are two main classifications of gangrene, dry type and wet or moist type (Porth & Gaspard, 2015). There are several other types of gangrene that are rarer, however, all types of gangrene manifest either as dry or wet form (Porth & Gaspard, 2015).

Risk factors for infection can be anything that suppresses the ability of the host to resist the infectious pathogen, including the following: drugs, such as steroids and chemotherapy; disorders that affect the innate and adaptive immune responses, such as cancer, leukopenia, and HIV/AIDS; and a breakdown of the first line of defense, such a trauma, broken skin, or surgical wounds. Patients are at risk for developing sepsis and septic shock if they are very old or very young and have and inability to fight off the infection. Some other risk factors for the

The lives can be cut short if internal organs start to blister. Also those who have EB are more likely to have skin cancer than those without EB. Another way that life could be cut short is because EB can cause sepsis which is when bacteria is spread all over the body via blood stream.

Is aggressive widespread inflammatory response to infection (sepsis) or perceived invader. Also, ischemia, infarction and injury.

It 's bad or infectious bacteria that cause illness as they rapidly reproduce and produce a toxin that kills or mutates cells, bacteria is also self sufficient.

Neisseria is a Gram negative bacteria that has fimbriae that allow attachment for colonization. This means that the bacteria has colonized , so there are multiple Neisseria cells present. Gram negative bacteria are endotoxins, which could explain why her symptoms have worsened. Endotoxins only excrete when the cells die, so when you take antibiotics it kills off the cells quicker than them just dying by themselves. The more cells that die the more severe the execration , the worse the symptoms. Endotoxins can cause your blood to clot causing tissue death or can cause septic shock, which could explain why half the people

Bacteria affected 2 million people from antibiotic resistance bacteria and 23,000 die per year due to the infections. Bacteria works by entering the body in various ways, they can enter past nose, mouth or ears as they are small, they can be inhaled from the air, eaten from food as well as through cuts/ open wounds.

In S. pyogenes-associated necrotizing fasciitis, the bacteria first invade host fascia before spreading and causing more invasive damage. Initial GAS invasion can occur through three primary pathways: 1) preceding superficial infection; 2) direct inoculation; or 3) hematogenous seeding from a previous injury site. After colonization, GAS spread through the typically sterile fascial planes that separate muscle groups, which are only loosely held together by connective tissue. The streptococci then release several proteases and other virulence factors that cause localized tissue damage. Simultaneously, this dissemination prompts the arrival of acute inflammatory host cells; the response of polymorphouclear leukocytes (PMNs), in particular, can also contribute to the tissue damage characteristic of necrotizing fasciitis. Once the

Initially diagnosis of herpes simplex was entertained and herpes cultures were taken but the appearance of the lesions, the very marked pain that was associated with them, and the occurrence of these lesions in the setting of rheumatoid arthritis and diabetes all suggested a diagnosis of pyoderma gangrenosum. He subsequently, was hospitalized because of worsening respiratory failure and subsequently, was treated for pneumonia and what is probably some congestive failure. He improved from intensive treatment of these problems but developed further ulceration on his right medial thigh with two large undermining lesions. He now is currently being treated with prednisone at 20 mg per day down from previous 40 mg. He continues to have marked hyperglycemia with difficulty controlling his blood sugars partially because of the prednisone dosage. He is being treated now with a collagen wound healing ointment and he is making excellent progress. While hospitalized for his pneumonia and pulmonary decompensation, he was treated with Levaquin and clindamycin. But cultures from the skin lesions were never positive. A biopsy of the lesions has shown a lymphocytic, histocytic and neurophilic infiltrate without signs of

| |This is a result of the misshapen blood cells gathering in clumps and getting stuck in joints in|

On his death certificate they declared his cause to be Sepsis by an unknown origin. Sepsis is defined as a systemic inflammatory response that occurs within the body due infection within the bloodstream. Common sites for infection that cause sepsis include pneumonia, kidney infections, or abdominal infections. The infection can be either bacterial, viral, or fungal. The most common gram positive organisms include staphylococcus aureus and enterococci. Common gram negative organisms include pseudomonas species and E. coli.

Most people naturally carry the bacteria in their intestines, away from the oxygen that would kill them. Given that our intestines produce different types of bacteria, so if we take antibiotic

Sepsis is a life threatening illness where the patient’s body responds improperly to an infection. The patient’s immune system goes into overdrive as a response to an infection. Sepsis develops with the chemicals that the immune system releases into the blood stream as the body fights the infection. The infection causes inflammation throughout the entire body (systemic) and can be very difficult to treat. Sepsis is also known as Systemic Inflammatory Response Syndrome (SIRS). There are three stages of Sepsis: Sepsis, severe Sepsis, and Septic shock. As Sepsis spreads though out the body via the blood stream, the inflammation spreads. Once a patient gets to the Septic shock stage, the patient is in grave danger and the patient’s bodily functions

Since the disease can be rather opportunistic, it can remain in an individual’s system for quite a long time, and as long as the immune system is strong, chances of infections will be less. However, once the body weakens, either because of other infections, and especially terminal diseases, the bacteria become active and infectious.