A Short Note On Systemic Sclerosis ( Ss ) Based On The Joint Committee Of The Acr

DIAGNOSTIC DATA: White count was 13.4, hemoglobin and hematocrit 15.4 and 45.8, platelets 206, with an 89% shift. Sodium 133, potassium 3.7, chloride 99, bicarb 24, BUN and creatinine are 18 and 1.1, respectively. Glucose 146, albumin 4.3, total bilirubin 1.7. The remainder of the LFTs is within normal limits. Urinalysis reveals trace ketones with 100mg per decilitre protein and a small amount of blood. CT scan was performed revealing evidence of acute appendicitis with pericecal inflammation, as well as, dilatation of the appendix and

REVIEW OF SYSTEMS;;12 point ROS was preformed and is negative except noted in above HIP, PMH and PSH. Careful attention was paid to endocrine, integumentary, pulmonary, renal and neurological exam

HISTORY OF PRESENT ILLNESS: This 46-year old gentleman with past medical history significant only for degenerative disease of the bilateral hips, secondary to arthritis presents to the emergency room after having had 3 days of abdominal pain. It initially started 3 days ago and was a generalized vague abdominal complaint. Earlier this morning the pain localized and radiated to the right lower quadrant. He had some nausea without emesis. He was able to tolerate p.o earlier around 6am, but he now

T.B. is a 65-year-old retiree who is admitted to your unit from the emergency department (ED). On arrival you note that he is trembling and nearly doubled over with severe abdominal pain. T.B. indicates that he has severe pain in the right upper quadrant (RUQ) of his abdomen that radiates through to his mid-back as a deep, sharp boring pain. He is more comfortable walking or sitting bent forward rather than lying flat in bed. He admits to having had several similar bouts of abdominal pain in the last month, but “none as bad as this.” He feels nauseated but has not vomited, although he did vomit a week ago with a similar episode. T.B. experienced an acute onset of pain after eating fish and chips

History of Present Illness: The patient is a 27-year-old male complaining of right lower-quadrant abdominal pain, nausea, and

The patient complained of right lower quadrant pain and of feeling faint. Dr. O'Donnel documented a chief complaint, a brief history of present illness, and a systemic review of the gastrointestinal system and respiratory system. Dr. O'Donnel also documented a complete examination of all body systems, which included all required elements. Medical decision making was of moderate complexity.

History of Present Illness: The patient is a 27-year-old male complaining of right lower-quadrant abdominal pain, nausea, and

This patient presented to the emergency department (ED) with pain in his upper right quadrant and flank. He reported experiencing abdominal distention

Multiple Sclerosis is an autoimmune disorder which is characterized by multiple areas of scarring to nerve fibers of the CNS. It often affects people between 20 to 40 years. Currently there are about 1.1 million people suffering from multiple sclerosis. Like most of the autoimmune diseases females are more affected than males in this disease. Multiple Sclerosis effects the central nervous system (brain and spinal cord). The disease commences with a discrete attack known as CIS (clinically isolated syndrome), which may attack any area of the CNS but mostly attacks either the brain stem, optic nerve or spinal cord.

Multiple Sclerosis (MS) is an autoimmune disease of the Central Nervous System, which interferes with the communication between the Central Nervous System (brain and spinal cord) and other parts of the body. The immune system attacks the myelin on the nerve fibers; resulting in damaged myelin that forms sclerosis. Overtime, many nerve fibers and myelin sheath will be damaged or even destroyed, resulting in the nerve impulse being interrupted. MS can be mild, moderate, or severe, therefore, it may cause disability as well as death. Interestingly enough, more women are affected than men. Common signs and symptoms of multiple sclerosis include: fatigue, difficulties with walking (gait), spasticity, numbness/tingling, weakness, vision problems, pain, vertigo, depression, emotional changes, cognitive changes, bladder/bowel problems, and sexual problems. Less common signs and symptoms include: speech/swallowing problems, seizures, tremor, breathing problems, headache, pruritus, and hearing loss. There a four different types of Multiple Sclerosis: Relapsing-remitting MS (RRMS), Secondary Progressive MS (SPMS), Primary-progressive MS (PPMS), and Progressive-relapsing MS (PRMS). Relapsing-remitting Multiple Sclerosis is the most common type of MS; about 85% of people are diagnosed with this type of Multiple Sclerosis. Relapsing-remitting Multiple Sclerosis is characterized by worsening neurologic function attacks throughout the disease. The attacks are also known

Thus allowing me to form a differential diagnosis and rule out certain causes, such as; constipation, and indigestion. Subsequently, the physical examination enabled me to confirm a diagnosis of acute abdomen. As the patient was not experiencing any worrying (red flag) symptoms associated with abdominal emergencies, such as; appendicitis or pancreatitis. However, I did forget certain aspects of the physical examination and had to be prompted by the MO. Although with more practice such incidence would be reduced.

It is believed that in the year 1754, Dr. Carlo Curzio of Naples first discovered this disorder in a young female patient of his. The disorder only affects the skin in most cases, but can harm blood vessels, internal organs, the digestive tract, and many more structures causing it to become life-threatening. There are several symptoms for each structure affected by Scleroderma. The skin experiences hardening and tightening, while the fingers and toes show signs of numbness,

Systemic Sclerosis (SSc) is an autoimmune connective tissue disease of unknown etiology resulting in a complex interplay of vasculopathy, inflammation, and fibrosis.[1] More recently, lymphatic microangiopathy was also described.[2,3] Lymphatic vessel involvement seems to start in an early phase of SSc and progresses to a significant loss of lymphatic capillaries in later stages.[3,4] This lymphatic microangiopathy in addition to increased vascular permeability may underlie the de pathophysiology of the early edematous phase frequently observed in SSc, which is most often observed on hands. In patients with diffuse SSc, this feature might be extensive and severe cases of lymphedema have been described.[5,6] The unusual feature of this case

Due to thickened skin from build- up collagen, motor skills will be greatly affected negatively; such as delay occurs in reaching, moves, walks, flows and paces. Also, weakening of stabilization, grips, lift, walks, transport, endures, calibrates and paces. As well as, malfunction can occur depends on the severity in grip, stabilizes, moves.

Systemic Lupus Erythematosus is a chronic autoimmune disease which causes inflammation of your joints, tissues, and organs. The inflammation presents itself as heat, pain, swelling and redness. SLE is a variable disease that doesn’t take any one particular course; therefore its unpredictability makes it even more devastating. No two people will experience the same disease symptoms or severity level. As S.L.E progresses there will be periods of very subtle to no symptoms at all called remission or an exacerbation of symptoms called flares.