Advantages And Disadvantages Of Piracetam

All antidepressants work in a similar way, though there are various types of antidepressants—often called “families”—that each work a bit differently. They all, however, increase the brain’s concentration of various neurotransmitters. Antidepressants are psychiatric medications given to patients with depressive disorders to alleviate symptoms. They correct chemical imbalances of neurotransmitters in the brain which probably cause changes in mood and behavior. Antidepressants may be used for a wide range of psychiatric conditions, including social anxiety disorder, anxiety disorders. Antidepressants were initially developed in the 1950s. Their use has become progressively more common over the last twenty years. Generally speaking, anti­psychotic

Chronic intake, the delayed onset of action, drug resistance and numerous side effects force the researchers to look for the new, safe antidepressant strategies (1, 2) with rapid onset and longer time of action.

The solution is often to raise the dosage. The trouble is that Phentermine can simply be handled an optimum dosage

Addiction to these can happen if taken over a long period of time. Also, irritation of the stomach, liver damage and sleep disturbances as some

One of the issues that always occurs when someone is trying to recover from an illness of some kind is the side effects that the treatments can give someone. Almost no NPT treatments have side effects besides emotional distress, but pharmacological treatments do have side effects. Joseph F. Goldberg, a clinical professor of psychiatry at Icahn School of Medicine, explains, Pharmacological treatments can have side effects ranging

Here’s a list of medications that are known to interact with this drug. There may be others not listed. If you have any questions, ask your

The minority of physicians prefer non-stimulants as a solution. 38% of 365 physicians who completed a survey concluded that they preferred to prescribe a nonstimulant (Collingwood 2). The primary issue with non-stimulants is the fact that they’re more liable to cause depression and suicidal thoughts, especially in teenagers, which is primarily why non-stimulants aren’t the popular choice. There are two types of non-stimulants: norepinephrine reuptake inhibitors, such as Strattera, which affect the behavioral control in the brain, and alpha-adrenergic agonists, such as Intuniv, which were developed to lower high blood pressure (Boorady 5). These two types of medications are similar in side effects concerning loss of appetite, drowsiness, and irritability. According to a survey on Treato, Intuniv received a review of very satisfied patients while patients using Strattera were moderately satisfied. A vast majority of those who used Strattera had great concern about the formula causing a feeling of depression while those using Intuniv were primarily concerned with weight gain and drowsiness caused by the lowering of the blood pressure. Around the age of 15, my parents sent me back to my physician to test a non-stimulant, and, due to the positive results, we chose to try Intuniv. Luckily, with the proper dosage, it actually worked, and to this day I still take it; of course, there are still some unwanted

If an individual is prescribed aminophylline, he or she should inform the doctor of any allergies that they might have and if he or she is taking any other medication and the time. This way the doctor would be better able to access the situation and provide any information or solution that might be needed to prevent any unwanted reactions. Also, medication such as over-the-counter or cold medicines can contain ephedrine, phenylephrine or pseudoephedrine which can have adverse and unwanted reactions when taken together with aminophylline. A user should check the labels of medications carefully

Melinda, I also have the same concern when it comes to side effects and furthermore allergies. This issue can definetely be drawback as it can contribute to non-complience and perhaps even life threathening related to allergies. I think it would be difficult to determine which drug could be the trigger for such reactions. If I was to be the health care provider prescribing this pill I would take into consideration, if the patient had taken any of this drugs separatly prior, to make an assessment if weather it would be a good candidate for the polypill.

Methaqualones were first introduced in India in 1955. This drug was then sent to Europe and Japan as a safe barbiturate substitute. By 1965 it had become the most commonly prescribed sedative in England. It was also at this time that it was gaining popularity as a street drug, under the names Mandies or

Gamma-Aminobutyric acid commonly referred to as GABA, and glutamate make up around 80 percent of the neurotransmitters found in brain. GABA dampens the activity in the brain while

Consult your doctor if you have any or other medical conditions, especially if any of these applies:

Regardless of your opinion when it comes to stimulants, one of the big concerns when taking any prescription drug should be the potential for interactions with other substances

Adverse reactions to this medication are migraine, speech disorders, rhinitis, sinusitis, hyperglycemia, elevated liver function, elevated serum creatinine level, pancytopenia, bronchitis, dyspnea, toxic epidermal necrolysis, anaphylaxis, elevated creatine kinase, generalized pain, and infection. Nursing considerations with this medication is to have the patient swallow the whole tablet and not to chew. Watch for aspiration while watching the patient take the medication. Educate the patient about the medication and inform them to notify a physician if bleeding

The linkage of serotonin to depression has been known for the past five years. From numerous studies, the most concrete evidence of this connection is the decreased concentration of serotonin metabolites like 5-HIAA (5-hydroxyindole acetic acid) in the cerebrospinal fluid and brain tissues of depressed people. If depression, as suggested, is a result of decreased levels of serotonin in the brain, pharmaceutical agents that can reverse this effect should be helpful in treating depressed patients. Therefore, the primary targets of various antidepressant medications are serotonin transports of the brain. Since serotonin is activated when released by neurons into the synapse, antidepressants function at the synapse to enhance serotonin activity. Normally, serotonin's actions in the synapse are terminated by its being taken back into the neuron then releases it at which point "it is either recycled for reuse as a transmitter or broken down into its metabolic by products and transported out of the brain." As a result, antidepressants work to increase serotonin levels at the synapse by blocking serotonin reuptake (2).