Alcohol Consumption And Metabolic Syndrome

Thesis: Despite the numerous factors such as genetic mutations, smoking, radiation, viruses, cancer-causing chemicals known as carcinogens, hormones, and lifestyle choices, today’s main culprit of many types of cancers is diet, specifically the American or Western diet which is characterized by a high intake of red meat, refined sugars, and saturated fat.

The aim of this study revealed an interested trend when comparing IL-6 results to the different metabolic diseases listed as top Ten Global Health Disparities. This study showed that CR will benefit situations where there is a socioeconomic disadvantage, even factor that include health disparities among race and age would show benefit because CR is suitable for all disadvantage groups. Caloric Restriction (CR) has proven to be non-expensive, beneficial agent against age related, racial, and socioeconomic disadvantage, associated with health disparities. The results for IL-6 in each case indicated that CR could improve endothelial function in rats with different metabolic diseases possibly through reduction in body weights resulting in reduction in fat mass and concomitant reduction in the production of the proinflammatory cytokines. IL-6 is expected to be reduced in Colorectal cancer (CRC) rats under

Birt, D. F.; Pelling, J. C.; Nair, S., and Lepley, D. Diet intervention for modifying cancer risk. Prog Clin Biol Res. 1996; 395:223-34.

Alcohol and hypertension are two rising concerns in national and global health. According to the Substance Abuse and Mental Health Services Administration (2014), 56.9% of adults in the United States have participated in drinking in the last month. Furthermore, 24.7% adults reported to participating in heavy or binge drinking. The World Health Organization (WHO) reports that in 2012, 3.3 million deaths or 5.9% of all global deaths were linked to alcohol consumption and within the United States 88,000 people died from alcohol related causes making it the fourth leading preventable cause of death in the United States (Stahre, Roeber, & Kanny, 2014). The abundance of alcohol consumption has shown to lead to high rates of hypertension.

Cancer is the leading cause of death among Asians Americans, and rates vary with age, acculturation and location. The stated differences may represent the influence of environment, such as diet and health habits, and therefore may be modifiable. When it comes to liver and stomach cancer, Asian Americans are tree times more likely to develop liver cancer than non-Hispanic whites and twice as likely to develop liver cancer. In the Chinese, liver cancer is associated with Hepatitis B (precursor to liver disease and cancer) and in whites, liver cancer is primarily associated with alcohol. For colon cancer, it is suggested that diet is related, so is other environmental risk factors. Breast cancer is the most commonly diagnosed cancer among Chinese

In Whiting S. Albert’s article “Alcohol Use Is Harmful” he stated that alcohol is a chemical that contains hydrogen and carbon. Whiting stated further that the active ingredient of alcohol is ethyl alcohol, which is also known as ethanol. Alcohol is one of the few things that human being consume that do not need digestion. Alcohol appears the bloodstream immediately, thus, its intoxicating effects are felt instantly (1). “High levels of mortality, morbidity, and social malaise are associated with abuse of alcohol, and increasing numbers of women and youth are abusing alcohol” (Ferreira and Darryn 1). “Alcohol slows down the function of all living cells, especially those in the brain” (Whiting 1), which causes thinking to be impaired. Alcohol is probably the most used drug worldwide. “Three in ten adults 18 years of age and over have had alcoholism and/or engaged in alcohol abuse at some point in their lives …” (“Impact of Alcoholism” 1). According to Centers for Disease Control and Prevention,

In the United States alone, 40 million adults are addicted to cigarettes (“Current Cigarette Smoking Among Adults in the United States”), 14 million are dependent on alcohol (“Drug Addiction Statistics – Alcoholism Statistics and Data Sources”), and 219 million people are overweight or obese (“Overweight and Obesity Statistics”). Worldwide, these statistics triple, double, and nonuple respectively. However, at least in the United States, cigarettes and alcohol are both regulated by age, so the numbers of adolescent smokers and alcoholics are limited. Sugar, on the other hand, is not controlled nearly as strictly as cigarettes and alcohol. Consequently, there are far more overweight or obese adolescents than there are underage drinkers and smokers. The health risks that come with such early exposure to a substance as toxic as sugar include obesity, heart disease and heart failure, kidney disease and kidney failure, liver disease, high blood pressure, depression, social withdrawal, and anxiety, among other physical and mental disorders. These health problems take a toll on the government health care system, which in turn can cause irreparable damage to the rest of the economy. In addition to the negative effects it has on physical health, in terms of addiction, sugar is comparable to nicotine, alcohol, and cocaine. Sugar production, purchase, and consumption should be regulated the same way other addictive substances are regulated – for example, through taxes, age restriction,

The World Gastroenterology Organization (WGO) has recently declared NALFD as most common cause of liver disease. Obesity is an increasing problem not only in the UK but worldwide. An estimated 26% of the adults in the UK are considered to be obese 1, 2. Obesity induced metabolic syndrome may lead to NAFLD, which can progress to Nonalcoholic Steatohepatitis (NASH), and in turn to advanced fibrosis and an increased risk of Hepatocellular Carcinoma (HCC) 2, 3.

It has been question on whether alcohol contains high amounts of calories and carbohydrates. Is there a correlation between alcohol, calories and carbohydrates? Does in increase in the amount of alcohol cause an increase in calories and carbohydrates?

We hear many different things about how alcohol affects the brain and body, most notably that it is a depressant. That’s only part of the story. Alcohol is a depressant, but it’s also an indirect stimulant, and plays a few other roles that might surprise you.

In that case the individual and the family of the individual often resort to social care and community health care services (Berkman, 2000).

Alcohol consumption is well entrenched in the social fabric of many adult populations across the world, virtually constituting a behavioral norm. It is cheap, legal and readily available. Regular and extensive alcohol consumption is a brain-centered addictive behavioral disorder that crosses all boundaries of gender, race, age, social class and, in many patients, might lead to alcoholic liver disease (ALD).1-3 Not only does heavy drinking significantly increases morbidity and mortality from infectious disease4, it also risks the development of brain, pancreatic, cardiovascular, oncological, cerebral and renal diseases.5

One of the alcohol’s effect on the major organ systems in the body is that alcohol affects the gastrointestinal system in a variety of ways. This system is the route by which alcohol enters the body and is absorbed. It is where the first steps of metabolism take place. Moderate amounts of alcohol can disturb the normal functioning of this system. Chronic heavy use of alcohol often raises havoc. Alcohol can have both direct and indirect effects. Direct effects occur as a consequence of the initial, direct effects. In addition, both acute and chronic use of alcohol stimulates the stomach lining’s secretion of hydrochloric acid and irritates the lining of the gut in general. It also inhibits the muscular contractions, called peristalsis, that

In addition, their plasma was checked levels of triglycerides and hormones. The results of the first study show that in the mice on ketogenic diet lost as much weight as the mice on the calorie restricted diet, but the group on the high carbohydrate-high fat diet (which ate just as many calories as the ketogenic diet group) showed an average increase of 6 lbs and twice the increase in body fat composition (as shown via DEXA measurement). The average body fat composition of all animals in other groups remained at around 3.4 lb per animal. Although both groups lost the same amount of weight, the metabolic rate (gathered by the CLAMS apparatus) of the ketogenic diet mice was 15% greater than the mice on the calorie restricted diet. Microarray analysis showed suppression of certain genes (e.g. FAS) involved in fatty acid synthesis in animals on the ketogenic diet, whereas in the high carbohydrate-high fat group, these genes were significantly

The majority of the studies done on the link between diet and cancer indicate that there is a relationship between high fat intake and an increased risk