Alcohol Essay

The pathophysiology of alcoholism begins after alcohol the ingestion of alcohol. It is absorbed and unaltered through the stomach and intestines. Next it is distributed throughout the body through the blood and absorbed by all tissues and fluids (Huether, 2012, p. 72). Furthermore, in the liver the alcohol blood content is metabolized into acetaldehyde by the enzymes process of alcohol dehydrogenase (ADH), the microsomal ethanol oxidizing system (MEOS), and catalase (Huether & McCance, 2012). Next, the metabolizing effects the central nervous system (CNS) and exhibits a depressant action. It is first expressed in the subcortical structures of the brain. This has an effect of disorientation of motor skills and intellect. With an increase in blood alcohol concentration, the medullary centers become depressed and as a result affects respiration (Huether & McCance, 2012). In addition, the effects of alcoholism encourage hepatic and gastric changes. The hepatic effect is caused by acetaldehyde, in which, induces inflammation, fatty deposits and enlargement of the liver (Huether & McCance, 2012).

Alcohol impairs judgment, memory, concentration and coordination, as well as inducing extreme mood swings and emotional

Alcohol is very damaging when taken, it can effect the processes of the brain and all of its neurotransmitters around it. When taking alcohol the brain loses its key things needed to get around daily life. Things like memory loss, nausea, danger with judgement, sleep patterns change, and much more can occur when taking alcohol. There are two common operating systems in our brain one that

Alcohol interferes with the brains communication pathways such as motor loss, memory loss, and impaired judgement.

Alcohol has been considered by many to be a powerful, and sometimes deadly, drug. It is often created during the breakdown process of sugar into ethanol and carbon dioxide during the fermentation of fruits or grain. The consumption of this substance has been restricted by age in most countries, as it has been linked to the harm of the kidneys, brain, and other parts of the human body. Alcohol has been a large presence in the media for centuries, mainly due to its sometimes damaging effects on consumers. However, there are those who believe that consuming alcohol could actually prove to have health benefits as well. A study by Dutch scientists suggests that a low-to-moderate intake of alcohol could actually reduce the risk of dementia in elderly victims from age fifty-five and up.

The ethanol helps the neurons in the brain resist Alzheimer’s and dementia later in life. In France there was a study that found moderate drinkers to have a 75% lower risk from Alzheimer’s disease and an 80% lower risk for senile dementia. Also older people who drink in moderation generally suffer less mental decline.

Alcohol is known to produce many chronic abnormalities that are attributable to parietal lobe dysfunction. The self becomes aware of the body being a distant object but it feels a strange attachment feeling (Miller et al, 1991). Considerable laboratory research indicates that different levels of alcohol consumption contributes to overall behavioural and cognitive functions and therefore the reason for carrying out this study is to test the effects of alcohol on human coordination. According to Allum et al (1998) Alcohol effects information received by brain receptors causing the working ones to compensate for the defective information processed by

L., Wang, Yoast, and Zeigler, (2005) study suggests that alcohol consumption is shared among teenagers and grownups; furthermore, there is a misconception that minors who consume alcohol heavily recuperate from the lethal consequences of alcohol. These persons often undergo detrimental consequences compared to grown up and this is due to diminished body mass, heavy consumption of alcohol, including binge drinking, in addition to primarily absence of tolerance to alcohol (Dickinson et al, 2005). Alcohol consumption contributes to impairment and mortality in teenagers; furthermore, it is related to intensified physical and health illnesses (Dickinson et al, 2005). Alcohol consumption during adolescence enhances the danger of development to alcohol use illnesses (Dickinson et al, 2005). Animal research established that teenage brains are more vulnerable than mature brains to neurochemical instabilities, lifelong instabilities in practical actions, neurodegeneration, and injuries in spatial recollection execution (Dickinson et al, 2005). Additionally, research has established that binge interactions with alcohol throughout puberty intensifies vulnerability to the memory damaging as a result of alcohol (Dickinson et al, 2005). Investigations on humans validates that teenage alcohol consumption is linked with neurodegeneration; fluctuations in functional brain action; and neurocognitive damages, which include shortages in

Research testing the role of alcohol in T2D has provide conflicting conclusions, with some researchers predict an escalation in risk of diabetes in alcohol consumers, while other researchers have predicted the opposite view. Even though there are a number of extensive investigations and assessments, the specific role of alcohol consumption in the occurrence and workings of T2D are inadequately known. The target of this research is to look at the connection of alcohol use as it relates to diabetic pathology and cognitive behavior. (p. 811)

According Roehrs and his colleagues of their research, “alcohol's sedative and performance-disruptive effects can be attenuated by enhancing the basal level of alertness after alcohol consumption” (Roehrs). It means that after drinking we would experience sleepiness that makes an individual dangerous to drive. Alcohol affects has the same severity to some neurotransmitter systems and sedative drugs do (Roehrs). “For example, alcohol facilitates GABA-mediated inhibition (acts as GABA agonist) and reduces the release of acetylcholine (acts as acetylcholine antagonist). Consequently, alcohol could mimic the effects of other sedative drugs” (Roehrs). Alcohol affects serotonin and glutamate; these two transmitters regulate sleep and wakefulness. Serotonin is a neurotransmitter that is in charge of of sleep. Glutamate is in charge of wakefulness. When alcohol have sedative effects, would make it hard for a person to stay awake which is why law prohibits us to drink and drive, because when we cannot stay focus driving under influence, it is not safe for the driver and everyone else on the road (Roehrs).

The drinking habit is ubiquitous in human societies, and notable for the best and worst reasons. More and more evidence that documents the effects of alcohol on the human organism continues to be gathered by scientists. As is usual with prolific research about one specific subject, there are some surprising and seemingly counterintuitive findings that leave scientists scratching their heads in confusion. One of the unexpected discoveries about the effects of alcohol is the seeming improvement of memory recall of recently learned information (specifically before the drinking event). This ironic observation contrasts with the prevailing literature documenting the detrimental effects of the substance to neurological health.

The consumption of alcoholic beverages dates back to approximately 10,000 years ago when ‘viticulture' (the selective cultivation of grape vines for making wine) is said to have originated in the mountains between the Black and Caspian Seas. (1) It is one of the most commonly used psychoactive drugs in the world. Alcohol interacts with gamma amino bultyric acid (GABA) receptors in the brain, this is the main inhibitory neurotransmitter in humans and is synthesized from glutamic acid. (1a)

At the age of eighteen the human brain is still developing and alcohol consumtion destories brain cells when consumed under the age of twenty-five. While there are many components of the brain suffering from alcohol throughout the adolescent years there are two areas of the brain that are very sensitive to alcohol throughout this era. The hippcampus cells are responible for learning and memory, throughout the

The consumption of alcohol affects youth adults brain more than it would to an adult. It can have many damaging effects that can affect their everyday life. In fact, it is said that a person’s brain is not fully developed till there in his or hers mid-20s.

Alcoholism is a chronic brain disease of addiction. Diseases such as Cancer, Diabetes, HIV Aids, affect some type of cell in the human body. Every organ is made up of multiple cells that work together to allow proper functioning of the organ. These diseases may affect one or multiple organs in the body. There is a concern amongst researchers that alcoholism has a particular affect on brain cells. The disease of Alcoholism impairs a human being’s brain functioning, similarly to how other diseases cause impairment in organ functioning within the body. In order to fully understand this disease, one must be familiar with the brain and how the body processes alcohol. Alcohol stays in the body and waits to become metabolized, suppressing normal brain functioning when settled for an extended period of time after extreme consumption. To personal belief, alcoholism is a disease of the brain caused by excessive drinking. If