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Alzheimer's Childhood

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Studies have indicated that exercise improves memory and cognition in older adults and we know nutrition at all stages of life has an important role. All of these ways to keep the brain healthy and even improve its health are, in a sense, external. What can the brain do to help itself? At what point in development should it start? This is not a question a parent thinks about for their child when contemplating their welfare. It is not something one thinks about as a child, adolescent, young adult or even mature or senior adult. There are more “important” things to consider than to try speculating on a future so far ahead as to whether a person is going to develop Alzheimer’s disease (AD) or dementia. To a family affected by AD or dementia, …show more content…

Their first step in their research was locating local people using birth records from 1921 but they were unsuccessful. The researchers discovered that in 1932, the Scottish Council for Research and Education (SCRE) conducted the Scottish Mental Survey (SMS32). This survey tested the childhood mental ability of all school children born in 1921. Access was given to the University of Aberdeen in Scotland to discover if there were childhood origins of late onset disease and disability, more specifically AD. They were also given access to another survey conducted in 1947 (SMS47). Both surveys gave researchers their baseline data for cognitive and dementia …show more content…

They were able to locate 354 local residents who did not have known dementia. They located 506 local people in 1999 from SMS47. “The Aberdeen cohorts were studied from ages 77 to 88 (SMS32) and from age 64 to 68 (SMS47)” (Whalley et al., 2011). The researchers wanted to identify major childhood influences in risks for dementia and they believed that because the proposed risk factors for AD were found equally in childhood (e.g. years of fulltime education, childhood intelligence, socioeconomic adversity, death of a parent, family history of dementia, APOE e4 carrier status) as compared to in late life (e.g. deficiencies of the vitamins B6, B9 and B12, head injury, sedentary lifestyle, risk factors for vascular disease). Data would be required from a long life course to build a full model of Alzheimer risk. They were able to use the surveys to adjust the data for the contribution of childhood mental ability to compare to the adult behavior patterns. This gave them the estimation of lifetime change. The recognized that the lack of exposure of data, between the surveys and information gathered in the adult interviews, was an important limitation to the study design. This can be eliminated with current longitudinal studies being

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