Atrial Fibrillation Research Paper

Maureen shows clinical manifestations such as hypotension (BP 80 mmHg systolic), tachycardia (HR 120 bpm and irregular), tachypnea (Resps 28 bpm), SaO2 unreadable, capillary refill time >4secs, temp 36.5°C (core) indicating the signs of hypovolaemia (Perner & Backer, 2014, p. 614). With the reference of Mrs. Hardy’s medical condition, such as arthritic knees and atrial fibrillation (INR 2.7), she is under diclofenac Acid 50mgs PO BD and warfarin 2mgs PO mane respectively (Jordan, 2010, p. 567; Zacher, et al., 2008, p. 930). Diclofenac is a

Patient complained she was short of breath and experiencing severe pain between her shoulder blades. She stated that she has been feeling nasuseated for the past 3 hours. She states she has a history of stable angina and is currently taking medication as needed. She states she did not take the nitroglycerin because she was not experiencing chest pain, just back pain. She states that her last check-up with the Pulmonologist showed that her EKG did not show any changes since her last visit. She denies episodes of syncope. The patient does report that she tripped over something on the floor, which resulted in her falling and hitting her back on a large table. In addition, she states that her heart rate has been ranging from 130/ 90 to 140/92. Patient states her Primary care physician placed her on blood pressure medication 2 months ago due to the increase.

In transport, patient received O2 at 4 liters via nasal cannula, baseline EKG, Normal Saline IV started in left hand, 325 mg aspirin by mouth (po). Patient complained she was short of breath and experiencing severe pain between her shoulder blades. She stated that she has been feeling nasuseated for the past 3 hours. She states she has a history of stable angina and is currently taking medication as needed. She states she did not take the nitroglycerin because she was not experiencing chest pain, just back pain. She states that her last check-up with the Pulmonologist showed that her EKG did not show any changes since her last visit. She denies episodes of syncope. The patient does report that she tripped over something on the floor, which resulted in her falling and hitting her back on a large table. In addition, she states that her heart rate has been ranging from 130/ 90 to 140/92. Patient states her Primary care physician placed her on blood pressure medication 2 months ago due to the increase.

African American male that is seen today for followup post hospital discharge. He is a 48-year-old gentleman with complicated cardiac history as well as neurological history including congestive heart failure. History of strokes 01/2017, possible sick sinus syndrome. He has an implanted pacemaker that was placed in 06/2017, as well as hypertension. He was taken to the Central Hospital on 09/01 with presentation of chest pain, noted to be around his pacemaker site. He identified being in seizure and suddenly felt chest pain with shortness of breath, and was offered nitro, he developed headaches and dyspnea post nitro treatment, of note is that the EKG that was obtained during that process, did not identify any pacemaker spike despite having a

You are the nurse working in an anticoagulation clinic. K.N. is a patient who has a longstanding irregularly irregular heartbeat (atrial fi brillation, or A-fi b) for which he takes the oral anticoagulant warfarin (Coumadin). Recently, K.N. had his mitral heart valve replaced with a mechanical valve. You know that there are different PT/INR (prothrombin time/International Normalized Ratio) goal recommendations based on the indication for anticoagulation. (NOTE: PT has now been replaced by or is reported, in most cases, with INR [International Normalized Ratio], an international value that allows for laboratory standardization. PTT is more properly written

Patient 2: The patient would receive anticoagulant medication and wear SCDs to prevent DVT, monitor oxygen levels and intervene when it is below the patient’s normal level, and keep the patient from exerting herself during activities, such as getting up to walk around.

Atrial fibrillation (AF) is a cardiac arrhythmia. It is the most common arrhythmia and it has implications for patients and anaesthetists alike. The anaesthetist must take into consideration the physiological and pharmacological implications of this common arrhythmia.

When one begins to discuss issues that arise with an intrinsic organ, things get serious. Unfortunately, vital organs like the heart and liver that are causing problems can’t be removed like the appendix or the kidney and “cure” people of issues such as Appendicitis or constant dealings with gallstones. This is true when things begin to go awry within the heart and conditions begin to arise. Atrial Fibrillation (A-fib) is the most common type of arrhythmia, or issue with the heart rate or rhythm. Though A-fib is not a condition that is life-threatening, depending on the severity and lack of treatment, this condition leads to more serious issues like heart disease, and even heart attacks and strokes, which are usually what end up being the cause of death in individuals diagnosed with the condition. Because of A-fib leading to other more detrimental problems, such as the ones listed above, it is difficult to identify and pinpoint if someone has it or had it, which is why it is known as one of the contributors to the “silent killer,” also known as the heart attack. In this paper, gaining a deeper understanding of Atrial Fibrillation will be accomplished through the exploration of: causes, diagnosis, symptoms and treatment, the variation of types, what all is affected within the body as a result, what happens physically, and what occurs at the cellular level when diagnosed with this disorder. All in all, Atrial Fibrillation should be taken seriously and further research is

This report is an analysis of an anonymous patient’s medications and how they relate to his health issues. Please note that the patient will be referred to as patient Afib in order to protect his privacy. Patient Afib is a 58 year old male with a recent onset of atrial fibrillation (AF) that has been cured using ablation and antiarrhythmic medications. This report discusses how physicians treated patient Afib’s AF, the medications patient Afib is currently taking and their mechanism of action, and how these medications relate to the patient’s disease. Medications discussed in this analysis include Proair HFA, Multaq, Toprol XL, and Coumadin. Proair HFA, a rescue inhaler used to treat asthma, was prescribed to patient Afib in order to determine if AF or underlying asthma caused his shortness of breath. Multaq is an antiarrhythmic drug used to keep patient Afib in a normal sinus rhythm. Toprol XL is a medication used in the treatment of hypertension that also exhibits rate controlling properties which prove beneficial for patient Afib. Coumadin, an anticoagulant, is used to reduce the risk of stroke for patient Afib should his AF ever spontaneously return without his knowledge. Patient Afib does not like the regime of pills he is required to take but understands their necessity.

I would like to express many thanks for my advisor Daryl Sieg for all of the support. I would also like to thank Dr. Klug for her guidance in the statistical aspect of this paper. Thank you to Allison Frank at the coagulation clinic in Fargo for taking the time to discuss my paper with me. Her view on these medications was very beneficial.

On average a healthy adult during state of rest has a heart rate of 80 beats per minute, that’s 4,800 times per hour, 115,200 times per day, and over the course of a year about 42,048,000 times. Each beat has the important job of pumping blood and keeping you alive. Every so often the extraordinary complexity of the human body has its flaws, for some heart rates are not always steady but rather rapid with erratic electrical pulses. An example of a dangerous flaw that could occur within the human body is Ventricular Fibrillation. Ventricular Fibrillation is the name given to the condition when the pumping chambers in your heart quiver uselessly instead of pumping blood which causes cardiac arrest.

According to a new study found that people with abnormal heart rhythm called atrial fibrillation that commonly used painkillers can increase the risk of bleeding and blood clots.This risk is even higher among those who are taking blood thinners with pain medications, such as anti-inflammatory drugs, which is not Dr. Nath Islamic Guinea (Gunnar Gislason) said that if you add NSAIDs on blood thinners, you have double the risk of bleeding.He noted that people with atrial fibrillation taking blood thinners to reduce the risk of ischemic stroke. Atrial fibrillation causes the upper recording rapid heart contractions and irregular. Twisted contract will form a blood clot, which can lead to ischemic stroke if it's from the heart and go to the brain.Dr.

Atrial fibrillation is a common disorder associated with significant morbidity, mortality, and economic costs. Radio-frequency catheter ablation (RFCA) of the distal pulmonary veins and posterior left atrium is increasingly being used by cardiac interventional electrophysiologists to treat patients with atrial fibrillation. The success of RFCA is highly dependent on a preprocedural understanding of the complex three-dimensional (3D) anatomy of the distal pulmonary veins and posterior left atrium. Neither fluoroscopy nor echocardiography can adequately depict this anatomy.

Presently, the evolving comprehensive research labors toward comprehending atrial fibrillation(Afib) is guided by epidemiologic results by gaining a better comprehension the natural history of the disease. As most diseases being researched there appears to be a growing "epidemic" and Afib is among those diseases that has substantial influence on morbidity and mortality in addition to the socioeconomic outcomes in association to hospital admissions, chronic disease management and disabilities(Chugh et al, 2014). Therefore, gaining a solid grasp of the epidemiology and natural history of AFib is critical to the future management of this disease.

Considering Joe’s CAD and atrial fibrillation, a multiple drug therapy approach could be implemented using aspirin, clopidogrel and Warfarin. Genetic testing would help elucidate dosing for Warfarin and prevent bleeding complications.