Ryan case study
1. Discuss the pathophysiology of atrial septal defect?
A septal defect is the connection between left and right side of the heart. The opening in the atrial septum permits left to right shunting of blood. The opening may be small, as when the foramen ovale fails to close, or large, as when the septum may be completely absent.
2. What is the incidence and etiology of this congenital heart defect?
Congenital defects of the atrial septum are common, accounting for approximately 13 percent of congenital heart disorders, with a reported birth prevalence of approximately 2 per 1000 live births. Every child is born with an opening between the upper heart chambers. It's a normal fetal opening that allows blood to detour away from the lungs before birth. After birth, the opening
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8. Priorities for Ryan’s preoperative care:
Important priorities Ryan needs right before surgery include a careful history and physical examination to detect the presence or potential development of any acute illnesses. Assess the child’s behavioral patterns, cardiac function, respiratory function, weight, and fluid status.
9.Potential complications associated with open-heart surgery performed on an infant: Infection: signs include fever, excessive incisional pain, spreading erythema around the incision, and wound drainage beginning 3 to 4 days post-op Arrhythmias: auscultate apical pulse to detect an irregular heart rate or bradycardia; both indicate a reduced cardiac output, which requires immediate intervention.
Pneumonia or fluid in pleural space: assess the chest and lungs for breath sounds, respiratory effort, and signs of distress. Impaired tissue perfusion: check pulse ox, capillary refill, extremity warmth, pedal pulses, LOC, and urine output. Reduced urine output is a sign of decreased cardiac
Atrioventricular Canal Defect is an abnormality that causes the mixing of blood. There is a hole in center of heart where the wall between the upper and lower chambers meet. The tricuspid and mitral valves aren’t formed properly and one large valve crosses the defect. The defect lets oxygen rich blood pass to the heart’s right side and mix with deoxygenated blood, then go back to the lungs. Another abnormality is Atrial Septal Defect (ASD), where the walls of the upper chambers of the heart don’t close completely, causing a left to right movement of blood due to the higher pressure. The mixing of oxygenated and deoxygenated blood may cause the right atrium and ventricle to enlarge due to the higher volume of blood.
2. The defect in Caleb’s heart allows blood to mix between the two ventricular chambers. Due to this defect would you expect the blood to move from left-to-right ventricle or right-to-left ventricle during systole? Explain your answer based on blood pressure and resistance in the heart and great vessels. It goes left to right during systole. The difference is normally, oxygen-poor (blue) blood returns to the right atrium from the body, travels to the right ventricle, and then is pumped into the lungs where it receives oxygen. Oxygen-rich (red) blood returns to the left atrium from the lungs, passes into the left ventricle, and then is pumped out to the body through the aorta. But when an infant has ventricular septal defect it still allows oxygen-rich (red) blood to pass from the left ventricle, through the opening in the septum, and then mix with oxygen-poor (blue) blood in the right ventricle. (ROCHESTER.EDU) but instead when systole occurs the blood gets mixed because of the septum therefore heart needs to pump harder to ensure that enough blood with oxygen reaches the body.
For both Atrial and Ventricular septal defect the size of the opening or hole affects how severe the child’s
The right side of heart will increase in size as the muscle builds up and the heart rate will increase due to compensation for the left ventricle. With auscultation, the lungs will sound crackly from the liquid in the alveoli and interstitial space and when assessing respiration rate it is likely to be above thirty breaths per minute (Lewis et al., 2014). The patient may report that they are experiencing dyspnea, nocturia, fatigue, and a productive coughing. They may appear anxious and their skin may look pale (Lewis et al., 2014).
Atrial Septal Defect affects the cardiovascular system of a person’s heart and affects a person’s life. The understanding of the heart and the process it completes in our bodies. As we look into the difference of what the heart does and the challenges you go through when your heart is not healthy. When we our born we our born with heart murmur’s in which they are supposed to close throughout your growing process. Atrial Septsl Defect is a hole in your heart that should be fixed in order for the person to live longer or a full life.
Congenital left ventricular outpouching (LVO) is an uncommon perinatal diagnosis. Various terms used in the medical literature to describe the LVO are a source of perplexity. The novel classification developed in a recent review by Rad et al attempting to provide a clear differentiation of the mixed definitions based on anatomical and cardiac imaging data. Our report presents a case series of prenatal diagnosis of LVO. The application of Rad et al original classification and its impact on the patients’ outcome are discussed.
Atrial Septal Defect(ASD) is a very large problem concerning the heart in its overall function. When the heart, being the core of the cardiovascular system, has issues; it effects the rest of the body as a result. The core of the problem resides in the atrial septum. Normally the heart is divided into four separate chambers. But a person with atrial septal defect has an atrial septum that allows the blood from the left side of the heart back into the right side. This results in increased pulmonary blood flow and diastolic overload of the right ventricle. By having this constant left-to-right shunt, it can alter the pulmonary vascular resistance leading to hypertension or even the reversal of the shunt itself.
A ventricular septal defect (VSD) is one of the most common congenital heart defects. During fetus development, a gap occurs in the heart’s lower wall between two ventricular chambers and allows blood to pass through from the left to the right side of the heart. The blood rich with oxygen, pumped back to the lungs instead of out to the body and cause the heart to work harder than usual.
An atrial septal defect (ASD) is a hole in the heart. This hole is located in the thin tissue (septum) that separates the two upper chambers of the heart, the right and left atrium. This hole is present at birth (congenital). A few minutes after birth, this hole normally closes so that blood is not able to go between the right and left atrium.
Patent ductus arteriosus (PDA), one of the more common cardiac defects at birth, is the persistence of an opening between the pulmonary artery and the descending thoracic aorta. This opening is as a result of failure of the physiological fetus ductus arteriosus to close, which normally occurs soon after birth. This hole allows for oxygenated blood from the aorta to mix with oxygen-depleted blood from the pulmonary artery. As a consequence, significant strain is placed on the heart and pressure within the lungs’ arteries is dramatically increased.
If the heart beats too fast, too slow or irregularly, it may not be able to pump enough blood to all of the body.
Third, thoraco-abdominal, constitutes 20% of the total cases. The heart is located in the area between the thorax and the abdomen. Lastly, abdominal, constitutes 10% of the total cases. The heart is located in the abdomen of the infant. In which the heart passes through a defect in the diaphragm to enter the abdominal cavity. To conclude, Thoracic and thoraco-abdominal ectopia cordis constitute the vast majority of known cases (Meena, 2016).
The normal flow of blood through the heart consists of the entry of blood to the right atrium through to the right ventricle and then the blood passes through the tricuspid valve entering the lungs and then through to the left atrium and to the left ventricle via the mitral valve and finally into the body. During fetal development, however there are the risks for congenital anomalies. These congenital heart disorders consist of Atrial septal defect (ASD) and Ventricular septal defect (VSD). ASD is one of the common congenital heart defects that requires interventional measures or repair. The condition occurs when the foramen ovale the opening between the atria prior to birth does not close and there
According to the text approximately 1, 966 babies are born with Atrial Septal Defect. People who have this disorder are dealing with this everyday of their life. Since Atrial Septal Defect is a heart condition, the symptoms and treatment have an impact on everyday life.
Congenital heart disease (CHD) is a problem with the heart's structure and function that is present at birth (Weinrauch, 2015). Congenital heart disease is divided into two types, cyanotic (turns the skin blue due to a lack of oxygen) and non-cyanotic. Cyanotic heart diseases include Ebstein’s anomaly, tricuspid atresia, tetralogy of Fallot, Truncus arteriosus, transportation of the great vessels, pulmonary atresia, hypoplatstic left heart, and total anomalous pulmonary venous return. Non-cyanotic heart diseases include aortic stenosis, pulmonic stenosis, patent ductus arteriosus (PDA), arterial septic defect (ASD), coarctation of the aorta, ventricular septal defect (VSD), and atrioventricular canal (endocardial cushion defect).