Atrial Septal Disorder Case Studies

Atrioventricular Canal Defect is an abnormality that causes the mixing of blood. There is a hole in center of heart where the wall between the upper and lower chambers meet. The tricuspid and mitral valves aren’t formed properly and one large valve crosses the defect. The defect lets oxygen rich blood pass to the heart’s right side and mix with deoxygenated blood, then go back to the lungs. Another abnormality is Atrial Septal Defect (ASD), where the walls of the upper chambers of the heart don’t close completely, causing a left to right movement of blood due to the higher pressure. The mixing of oxygenated and deoxygenated blood may cause the right atrium and ventricle to enlarge due to the higher volume of blood.

Ventricular septal defect cause the blood flow move from high pressure, left ventricle, into the right ventricle, which has lower pressure. Oxygenated blood instead of going to the systemic circulation enters into the left ventricle and goes into the pulmonary artery and to the lungs. As the result return of the blood from the lungs to the left atrium and eventually into the left ventricle, will increase and cause dilation and hypertrophy of the chambers, increases end-diastolic pressure, and raise the pulmonary venous pressure. By increasing the pulmonary blood flow, the pulmonary capillary pressure also rises and that can increase the interstitial fluid in the lungs, and cause pulmonary edema. Finally, as blood goes through the gap, from the LV to the RV, away from the aorta, case the cardiac output to drop,

A ventricular septal defect or VSD is a heart defect. It’s congenital, this means that a baby is born with it. It is an opening or hole in the dividing wall called the Septum, between the two lower chambers of the heart, right and left ventricles. Ventricular septal defects are the most common type of congenital heart defects. A VSD allows oxygen-rich blood to pass from the left ventricle through the opening in the septum. Then it mixes with oxygen-poor blood in the right ventricle. There are different types of ventricular septal defects, the type your child has depends on which part of the wall between the ventricles is involved. Also the size of the opening or hole varies. A ventricular septal defect may occur more often in some families, this is because of gene defects. But most of the time, the cause is unknown. Similarly to ventricular septal defect, an Atrial septal defect is a birth defect of the heart in which there is also a hole in the wall in the septum, but instead of dividing the lower chambers it divides the upper chambers of the heart (atria), here a hole can vary in size and may close on its own or may require surgery as well. An atrial septal defect is one type of congenital heart defect, in which also the baby is born with it.

If the heart beats too fast, too slow or irregularly, it may not be able to pump enough blood to all of the body.

Atrial Septal Defect affects the cardiovascular system of a person’s heart and affects a person’s life. The understanding of the heart and the process it completes in our bodies. As we look into the difference of what the heart does and the challenges you go through when your heart is not healthy. When we our born we our born with heart murmur’s in which they are supposed to close throughout your growing process. Atrial Septsl Defect is a hole in your heart that should be fixed in order for the person to live longer or a full life.

Congenital heart disease include many different types of defects. Some of these defects are simple, such as a hole in the septum. Others, are more complex and severe that include combinations of simple defects, problems with the location of blood vessels leading to and from the heart and other serious problems with the development of the heart. The different types of defects ranging from simple to complex are holes in the heart (septal defects or atrial septal defects-ASD), Patent Ductus Arteriosus (PDA-abnormal blood flow occurs between the aorta and pulmonary artery), Narrowed valves

The normal flow of blood through the heart consists of the entry of blood to the right atrium through to the right ventricle and then the blood passes through the tricuspid valve entering the lungs and then through to the left atrium and to the left ventricle via the mitral valve and finally into the body. During fetal development, however there are the risks for congenital anomalies. These congenital heart disorders consist of Atrial septal defect (ASD) and Ventricular septal defect (VSD). ASD is one of the common congenital heart defects that requires interventional measures or repair. The condition occurs when the foramen ovale the opening between the atria prior to birth does not close and there

Mrs. Schafer completed her pulmonary function test prior to the appointment time and she was evaluated by Dr. Theodore J Standiford. Mrs. Schaefer provided an acute report of her injury and symptoms she was experiencing. Dr. Standiford replied that Mrs. Schaefer lung capacity was reduced by a third. (1/3). I inquired if it was the result of the injuries in the MVA. Dr. Standiford replied that they are a contributing factor i.e. flail chest and fluid in her lungs from aggravated congested heart failure. Dr. Standiford noted left crackles breath sounds on his examination and recommended that Mrs. Schaefer return to her cardiologist and that a high resolution CT scan of her lungs be obtained prior to the next appointment to determine if she has

Congenital left ventricular outpouching (LVO) is a rare disorder in which a confined protrusion arises from the left ventricular (LV) free wall. The terms LV aneurysm, LV diverticulum, double-chambered LV and LV accessory chamber have been used interchangeably in the literature [1–9], however anatomical differences do exist between these terms [10]. LV aneurysm is an expansible thin-walled, usually fibrotic myocardium and pericardium, pouch contracting in asynchrony with the LV and has a wide communication with the LV cavity. Conversely, an LV diverticulum has three cardiac wall layers (endocardium, myocardium and pericardium), contracts in synchrony with the LV and has a narrow connection to LV. LV accessory chamber presents an entity that combines some criteria of both aneurysm and diverticulum. Its wall is similar to a diverticulum but it has a wide communication with the LV as an aneurysm. Lastly, double-chambered left

Malpositioned simply means that the aorta opens inside the heart in the wrong place. In a normal, healthy heart, the aorta is connected to the left ventricle, allowing oxygen rich blood to be pumped throughout the body. In patients suffering from tetralogy of Fallot, the aorta is slightly shifted to the right. This slight shift places the artery directly over the ventricular septum defect, which is typically located at the tip of the ventricles. As previously stated, the VSD allows oxygen rich and oxygen poor blood to mix together. With the aorta being directly over the VSD, this allows the mixture of blood to be pumped through the rest of the body, again contributing to the cyanotic presentation of the

Congenital heart disease (CHD) is a problem with the heart's structure and function that is present at birth (Weinrauch, 2015). Congenital heart disease is divided into two types, cyanotic (turns the skin blue due to a lack of oxygen) and non-cyanotic. Cyanotic heart diseases include Ebstein’s anomaly, tricuspid atresia, tetralogy of Fallot, Truncus arteriosus, transportation of the great vessels, pulmonary atresia, hypoplatstic left heart, and total anomalous pulmonary venous return. Non-cyanotic heart diseases include aortic stenosis, pulmonic stenosis, patent ductus arteriosus (PDA), arterial septic defect (ASD), coarctation of the aorta, ventricular septal defect (VSD), and atrioventricular canal (endocardial cushion defect).

An atrial septal defect (ASD) is a hole in the heart. This hole is located in the thin tissue (septum) that separates the two upper chambers of the heart, the right and left atrium. This hole is present at birth (congenital). A few minutes after birth, this hole normally closes so that blood is not able to go between the right and left atrium.

The prognosis of Ectopia Cordis generally depends on three factors: Location of the defect, the extent of cardiac displacement, and the presence of any abnormalities (Obstet 1958). In most cases the prognosis is most commonly poor. Most babies are stillborn while other babies expire after shortly after delivery due to hypoxemia, cardiac failure, and infection. The poor prognosis is related to the difficulty of returning the heart back into a small thoracic cavity (Amato JJ, 1995).

Atrial Septal Defect is a common and interesting disorder. Atrial Septal Defect is mostly know as “a hole in the heart.” This disorder is birth defect. It can happen to male, female, adult, and kids. This disorder mostly happens to kids and infant. Atrial Septal Defect is when there is a hole in your heart that doesn’t allow oxygen to flow through. This can causes difficulty breathing, heart murmur, and e.t.c... There three different types of level. These level are called ostium secundum, ostium primum, and sinus venosus. The most severe level is ostium secundum which is the true atrial septal. Then is ostium primum, and last sinus venosus. This genetic disorder is typically diagnosed by noticing .symptoms.

Patent ductus arteriosus (PDA), one of the more common cardiac defects at birth, is the persistence of an opening between the pulmonary artery and the descending thoracic aorta. This opening is as a result of failure of the physiological fetus ductus arteriosus to close, which normally occurs soon after birth. This hole allows for oxygenated blood from the aorta to mix with oxygen-depleted blood from the pulmonary artery. As a consequence, significant strain is placed on the heart and pressure within the lungs’ arteries is dramatically increased.