B) Evaluate the Biological Approach’s Explanation of Schizophrenia.

The cognitive explanation acknowledges the role of biological factors in schizophrenia, suggesting that the basis of the condition is abnormal brain activity producing visual and auditory hallucinations. Further features of the disorder emerge as people try to make sense of the hallucinations.

Schizophrenia is classified as a mental disorder that shows profound disruption of cognition and emotion which affects a person’s language, perception, thought and sense of self. The dopamine hypothesis states that schizophrenic’s neurones transmitting dopamine release the neurotransmitter too easily, leading to the characteristic symptoms of schizophrenia. This hypothesis claims that schizophrenics have abnormally high amounts of D2 receptors; receptors that receive dopamine, therefore resulting in a higher amount of D2 receptors binding to the receptors causing more impulses. Dopamine neurotransmitters play a

Physiological Changes Due to Risperdal Hallucinations, delusions, paranoia, psychosis and thought disorder are all symptoms of Schizophrenia and people who suffer from these symptoms seek the treatment of atypical anti-psychotic medications. Those medicines include Risperidone (Risperdal), Clozapine (Clozaril) , Ziprasidone (Geodon) and Quetiapine(Seroquel). The purpose of these medications is to alleviate symptoms of Schizophrenia and lessen the chances of a recurrence. The basic function of Atypical Antipsychotics is to reduce the effects of blockage in the dopamine receptors and serotonin and allow communication between nerve cells. Dopamine is thought to be relevant in Schizophrenic symptoms and

There are two main biological theories which explain the assumed causes of schizophrenia: Genetic and Dopamine Hypothesis.

Psychopathology is the study of mental distress and abnormal maladaptive behaviour, there are four approaches to psychopathology, cognitive, behavioural, psychodynamic and biological. The biological model of abnormality is split into four parts which can all cause abnormal behaviour these are; Genetic, Brain injury, Neurotransmitters and Infection.

Because of the prevalence of copious quantities of dopamine in people who have schizophrenia, most medications attempt to prevent the buildup of dopamine by blocking it in the striatum. Other methods in which drugs for this condition help to counteract the effects of dopamine, however, are more indirect. There are medications that are under development that "interfere with glutamate signals in the brain" (Nauert, 2010) that are targeted towards affecting glutamate, which in turn will then positively influence the levels of dopamine that the brain releases. In both ways then, (direct and indirect), dopamine is the primary focus of schizophrenia medication.

The scientific interpretation of schizophrenia was one that supported a physical basis which was attributed to an imbalance in the cognition of humans. The goal of natural science aimed in finding rational, scientific causes, that would lead to biological

Dopamine is a neurotransmitter associated with attention. Schizophrenics seem to have an increased number of D2 dopamine receptors on receiving neurons. This theory is support by the fact that Phenothiazines bind with D2 receptors and reduce positive symptoms. Another piece of supporting evidence is that Parkinson’s disease drug L-dopa, increase the level of dopamine and causes schizophrenic like symptoms in some people. However excess dopamine can only explain some types of schizophrenia, usually positive rather than negative.

Over the years, experiments have produced evidence to suggest that dopamine plays a role in the development of Schizophrenia (Howes, McCutcheon, & Stone, 2015). Dopamine is a neurotransmitter that is produced in the substantia nigra and ventral tegmental regions of the brain. The belief that dopamine was involved in Schizophrenia arose after multiple studies performed with compounds produced an increase in extracellular concentrations of dopamine (Lieberman, Kane, & Alvir, 1987). The patients that were administered these compounds had similar symptoms to those observed from patients who were diagnosed with Schizophrenia (Lieberman et al., 1987).

Dopamine is one of the main hypothesized physiological causes of schizophrenia (Brisch et. al., 2014). It is involved in movement, pleasure, and cognition: all of which are impacted in schizophrenia. Dopamine is a neurotransmitter or a chemical messenger. It is in the neurotransmitter class of catecholamine and has both inhibitory and excitatory effects (Carlson, 2013). These effects are seen on the postsynaptic neuron, depending on the neuron dopamine can either be inhibitory or excitatory. Within the brain, there are three main systems that use dopamine as their neurotransmitter. These three systems are the nigrostriatal system, mesolimbic system, and the mesocortical system (Carlson, 2013).

Schizophrenia has been termed a heterogeneous group of disorders with varied etiologies (Walker, Kestler, Bollini, & Hochman, 2004) which includes biological, social, cognitive and psychodynamic perspectives. To progress knowledge of schizophrenia, this essay focuses on how the biological and psychological explanations are independent and interdependent and how they may differentiate from one another. This includes: how our biological predisposition, neuro transmitter dysfunction and genetic inheritance, affects how people with schizophrenia respond to social environments, the importance of socio-economic factors

The Dopamine Hypothesis theorizes that the symptoms portrayed in Schizophrenia is can be explained by abnormal function of dopamine in the brain. There have been three versions of the Dopamine Hypothesis. The first version of the hypothesis focuses on the dopamine receptors. Antipsychotic drugs that impact the metabolization and reabsorption of dopamine where found to be effective in treating the symptoms. It was theorized that if the symptoms of a Schizophrenic episode can be treated by the use of dopamine

Schizophrenia is a very serious, long-term disorder that affects about 1% of the world’s population. It affects people anywhere from twenty years old, to forty-five years old. It is known to be one of the most disabling diseases in this age group. Schizophrenia can break down a person’s behaviors, emotions, and thoughts. People who suffer from schizophrenia usually show very inappropriate displays of their actions and feelings. Sufferers have been known to hear voices, even when there is nobody around them. They have problems controlling their thoughts, and sometimes blurt out things that are very inappropriate. This paper will outline the biological, social, and psychological

Many people, including mental health professionals, see how severe psychotic disorders, such as schizophrenia, can be and instantaneously presume it is mostly biological. Now, most do believe in the value of talk therapy, and it is widely recognized that schizophrenia often starts with a stressful trigger. These triggers can include things as normal as a first year of college or as terrible as a trauma such as a sexual assault. Now, I'm not doubting genetic factors or other biological factors exist in those of us with psychotic disorders, but I have a pet theory on what these disorders are. While, I admit the following is more of a personal opinion than science. I, also, doubt it would be considered complete pseudoscience.

Some of these medications include Aripiprazole, Asenapine, Brexpiprazole, Cariprazine, Clozapine, Iloperidone, Lurasidone, Olanzapine, Paliperidone, Quetiapine, Risperidone, and Ziprasidone. Aripiprazole blocks dopamine and serotonin receptors in the brain (Aripiprazole). These two neurotransmitters send messages between brain cells. The medication, however, helps to control dopamine activity in the brain where the dopamine is being overactive (Aripiprazole). In the areas that cause the negative symptoms of Schizophrenia, Aripiprazole stimulates the dopamine levels to make the brain, in those parts, seem as though there is enough dopamine (Aripiprazole). The drug Risperdal also blocks dopamine and serotonin receptors in the brain (Risperdal). At this time the two neurotransmitters send messages between the nerve cells (Risperdal). The drug relieves symptoms of Schizophrenia such as anxiety, depression, and feelings of guilt (Risperdal). The second-generation medications help block receptors in the brain to reduce or get rid of some of the symptoms of