Bovine Spongiform Encephalopathy

The medicine is applied from the neck, all the way down and must be left on for at least eight hours. You can also kill the mites from using oral medications. Some medicines are Permethrin cream, Lindane lotion, Crotamiton also know as Eurax, and Ivermectin also known as Stromectol. Crotamiton is available as a cream or a lotion and it's applied once a day for two days. As for Ivermectin, doctors usually prescribe this oral medication for people who have altered immune systems, or for the people who don't get results from the prescription lotions and

Animal or zoophilic infections are usually periodic and restricted to the areas where the host animal is found. Microsporum canis, the cat and dog ringworm, is the commonest of the zoophilic infections worldwide and spread occurs directly from an infected animal and, probably, from contaminated furniture, floors and carpets in the home environment. I contrast, Anthropophilic dermatophytes are more common in the community. In some cases there is evidence to support the existence of localized epidemics of

The meats that consumers eat are slabs of a cow’s muscle tissue and the infectious meat is only found in the nerve tissue. Of course there is the possibility of having some of the contaminated meat passed on to the meat that consumers eat when the cows are cut, but the chances are very slim. Since this disease is contracted through eating contaminated meat, the site of infection should be in the digestive tract, yet this disease eats away at the brain and nervous system.

The word encephalitis is derived from two Greek words. Enkephalos, which means brain, and itis, which means inflammation. Encephalitis was defined by Sir John Burton Cleland, a pathologist and naturalist. When he defined it, it was named ‘Australian X disease’.

     Mad Cow Disease, scientifically referred to as (BSE) Bovine Spongiform Encephalopathy, is a disease that affects those humans who eat the meat from infected cows. Mad Cow Disease is one of several fatal brain diseases called (TSE) Transmissible Spongiform Encephalopathy. (USDA) There was evidence of a new illness resembling the sheep disease scrapie. It was technically named BSE but quickly acquired the mad cow tag because of the way infected cattle behave. (CNN) In 1997, there was an award given to Stanley Prusiner, for concluding that a distorted protein called a prion was responsible for Mad Cow Disease, noted the long incubation period made it difficult to distinguish (Bryant).

The effects of mad-cow disease on cattle are very obvious. This disease is not one in which you see a cow grazing and it looks perfectly fine, but a disease in which the cow looks as if it has no control over its own body and mindlessly wanders around. That is because having no control over its own body is exactly what happens.

Variant Creutzfeldt-Jakob disease (vCJD) is a rare and fatal human neurodegenerative condition falling under the category of Transmissible Spongiform Encephalopathy (TSE) because of having characteristic of spongy degeneration of the brain that it causes and its ability to be transmitted. First it was described in the United Kingdom in March 1996 and it has been connected with exposure to a TSE of cattle known as Bovine Spongiform Encephalopathy (BSE) sometimes called Classical BSE, having been reported first in 1986 in the United Kingdom.

What is Mad Cow Disease? Mad Cow Disease "is a normally extremely rare neurological disorder that affects the central nervous system of an animal and a human. The disease infested brain of the animal or human is slowly, progressively, and severely damaged. It has been categorized as a disease, which falls into the category of a disease known as Spongiform Encephalopathy. Having Bovine Spongiform Encephalopathy is an over-dramatized way of specifying that one has diseased brain tissue which is; spongy, porous, and no

Lyme disease is caused by the bacteria Borrellia burgdorferi, and it is the most common vector borne illness in the United States (CDC, 2014). Lyme disease is transmitted by the blacklegged ticks and they live on animals like dogs and cats, around stone walls and wood piles. These ticks also live in wooded and bushy areas with high grass and leaf litter. These ticks hold onto leaves and grasses by their lower legs, where they get picked up by a passing host and that is one of the ways through which they make their way to the human body.

According to Tonya Colpitts and associates, through some research it has shown that the virus has the ability to infect macrophages, which help facilitate the virus across the blood brain barrier where it can then begin causing neurological symptoms (Colpitts 5). Yet, according to the article written by Emilie Donadieu and colleagues, “the mechanisms by which West Nile Virus enters the central nervous system is not fully understood” (Donadieu 9). Not enough research has been done to confirm the hypothesis that it is most likely that the virus affects the central nervous system via the blood brain barrier, which helps to protect the brain from pathogens and being exposed to immune mediation or activation (Donadieu 9-10). I it is known however, that once the virus crosses the blood brain barrier it leads to neuronal death via lesions on the brain (Donadieu 6). Neurological symptoms seem to develop more frequently in horses than in humans. According to the article written by Antoine Garwah, 1 in 150 infected people develop severe symptoms of the virus, which include but are not limited to: high fever, headache, stupor, tremors, seizures or convulsions, and paralysis (Garwah 2). Meanwhile, according to the Merck Manual article, West Nile Encephalomyelitis in Horses, approximately 10-39% of infected horses show signs and the neurological signs

"Mad Cow" disease or BSE has never been detected in the United States. No definitive links have been found between BSE and a human form of the illness.

Mad Cow Disease Bovine spongiform encephalophathy ( BSE), which is mainly known as mad cow disease has infected the society and has put the lives of individuals at risk. This was very frightening to the people that ate meat during that time. The terrified people would not even want to eat hamburgers at MacDonald, steak at restaurant and much more. What also alarmed the people was the fact that there was a possibility of getting the human form of mad cow disease. This was called variant Creutzfeldt-Jakob disease (vCJD) which is through tainted blood, but this was only a theory which

Scalp ringworm (tinea capitis) is a fungal infection of the skin on the scalp. This condition is easily spread from person to person (contagious). Ringworm also can be spread from animals to humans.

Trichinosis also referred to as trichinellosis is caused by ingesting the larvae of the parasitic roundworm species called Trichinella. The infection commonly occurs after eating raw or undercooked meat from animals such as domestic pigs or wild boar, horses, bear or cougar. The Trichinella larvae will form a cyst within the striated muscles of these animals and once ingested will mature into adult worms in the small intestine after 1-2 days. These adult worms will mate and produce eggs, which will then develop into larvae that cross into the blood system and migrate to striated muscles where they form cysts. The clinical presentation of Trichinosis varies

It is considered as a hypersensitivity reaction to the migrating larvae of stomach worms Habronema microstoma, H. muscae and Dreschia megastoma. Both the stable and house fly deposit their larvae on manure, which injest the stomach worm larvae and serves as intermediate host. The adult flies deposit stomach worm larvae wherever they feed like mucocutaneus junction, wounds and other moist regions of the animal’s body. The larvae deposited near mouth or lips complete their life cycle and become adult in the stomach. The larvae can penetrate wounds and moistened skin lesions and may reach to dermal and subcutaneous tissues and eventually die (Rebhun, 1996; Yarmut et al.,2008). The disease occurs more in the fly season with warm weather and regress during winters. Calcification and deposition of sulfur granules are the characterstic for stomach worm lesion (Pusturela et al.,2003). The larvae deposited on the mucous membranes or on injured tissue do not complete their life cycle and induce a local inflammatory reaction causing cutaneous (summer sores) or ophthalmic habronemiasis (Traversa et al.,