COPD: A Systemic Disorder Analysis

The positive outcome of the acute treatment of the patient helped me feel more confident in communicating with patients. The patient commented on his appreciation of having the ambulance service available, in his time of need. I told the patient this was my first week on the road, and he said that he was happy with my performance. My paramedic mentor gave me positive feedback on my ability to communicate well with the patient. The treatment package contributed to a good understanding of how the therapeutic respiratory drugs worked and how quickly they became effective. I found out that COPD patients should only permitted to have increased oxygen levels for no longer, than six minutes as stated in (section 27 of B R O’Driscoll, etal

This is a case study on a 76 year old man.Mr Alan Chari(pseudonym used to protect the identity of a patient),was admitted over night in my department.He is a divorcee who stays with son.He is a retired teacher and his son is permanently employed by a local company as an electrician.He is independent with activities of daily livings but is occasionally limited by his ill health.He used to be a heavy smoker .After realising the burden COPD has on general New Zealand population ,affecting about15% of the adult population over the age of 45 years according to asthmanz( 2010) ,l took this case study to gain in-depth understanding.

R.W. appears with progressive difficulty getting his breath while doing simple tasks, and also having difficulty doing any manual work, complains of a cough, fatigue, and weight loss, and has been treated for three respiratory infections a year for the past 3 years. On physical examination, CNP notice clubbing of his fingers, use accessory muscles for respiration, wheezing in the lungs, and hyperresonance on percussion of the lungs, and also pulmonary function studies show an FEV1 of 58%. These all symptoms and history represented here most strongly indicate the probability of chronic obstructive pulmonary disease (COPD). COPD is a respiratory disease categorized by chronic airway inflammation, a decrease in lung function over time, and gradual damage in quality of life (Booker, 2014).

COPD is the continued tightening of the airways, causing a blockage to the airflow to the lungs, which causes shortness of breath. It chiefly comprises of emphysema and chronic bronchitis. Both are typically caused by smoking, or less frequently, by work-related exposure to dusts or

History of Present Illness: Mr. Magnuson is a very pleasant 77-year-old gentleman who was previously seen in this office by Elvira Aguila, MD for COPD and hypoxic respiratory failure. He is here today for routine followed up. He was last evaluated in January 2015. Since that time, he states that his dyspnea is worse. He feels that it is related to the weather. He does state that he works around the house, although he does have significant functional limitations because of shortness of breath. His wife also confirms that he is able to do less and less. He has a stable, minimal cough. He is using 4L of oxygen at night as well as, as needed throughout the day. He continues to smoke three to four cigarettes on

The main symptoms of COPD are long lasting cough, mucus that come up when you cough, and when you exercise (or even just walk up the stairs) shortness of breath can get worse. When COPD gets worse, it gets harder to eat or exercise, and breathing takes much more energy. People often lose weight and get weaker. Symptoms might even flare up and get much worse. This is called COPD exacerbation. An exacerbation can range from mild to life threatening. The longer you have this disease, the more severe the flare up can get.

COPD is a disease that depletes a person of air. This disease is the fourth top cause of death in the United States. COPD describes several lung diseases including emphysema, chronic bronchitis, refractory asthma, and other forms of bronchiectasis. There is no average case, as every case is different from the next. This disease is long term but treatable.

Chronic Obstructive Pulmonary Disease (COPD) is an umbrella term used to portray dynamic lung sicknesses including emphysema, constant bronchitis, unmanageable (non-reversible) asthma, and a few types of bronchiectasis. This malady is described by expanding shortness of breath (What is COPD?). Many individuals botch their expanded shortness of breath and hacking as an ordinary piece of maturing. In the early phases of the infection, you may not see the side effects (What is COPD?).

The effectiveness of Family Support Caregiver in improving the Functional Status of Client with Pulmonary Diseases.

Let us first discuss your recent diagnosis of COPD. You may have wondered what COPD is and what caused it. Excellent questions! COPD stands for chronic obstructive pulmonary disease (Mayo Clinic Staff, 2016). Chronic inflammation obstructs your airflow, resulting in trouble breathing and excess mucus production. So what exactly causes that chronic inflammation? Cigarette smoke and other noxious gases cause this inflammation in your lungs and airway (Mayo Clinic Staff, 2016). Not everyone who smokes gets COPD. Also, not everyone who has COPD smokes. Workplace exposure to noxious gases can also lead to COPD. Additionally, there is a genetic disorder that causes an underproduction of a lung protecting protein (Mayo Clinic Staff, 2016).

COPD is a multidimensional illness, with a few systemic signs and relationship with various comorbid maladies. The undoubtedly connect amongst COPD and these extrapulmonary conditions is an overflow of provocative arbiters from the lung, as systemic irritation is related with skeletal muscle squandering and cachexia and also with cardiovascular, metabolic, and bone illnesses. More research is expected to comprehend the connections between these illnesses and to scan for regular treatable segments. It appears to be likely that medications, for example, statins, that are as of now used to oversee cardiovascular and metabolic ailments may likewise give an advantage in COPD patients, in spite of the fact that it is critical that randomized fake treatment controlled trials be led to affirm this probability. It is critical to consider how the presence of a comorbid infection may influence the administration of the patient who additionally

This assignment will explain the pathophysiology of the disease process chronic obstructive pulmonary disease (COPD). It will examine how this disease affects an individual looking at the biological, psychological and social aspects. It will accomplish this by referring to a patient who was admitted to a medical ward with an exacerbation of COPD. Furthermore with assistance of Gibbs model of reflection (as cited in Bulman & Schutz, 2004) it will demonstrate how an experience altered an attitude. In accordance with the Nursing and Midwifery Council, (NMC) Code of Professional Conduct (NMC, 2005) regarding safeguarding patient information no names or places will be divulged. Therefore throughout the assignment the patient will be referred to

Chronic Obstructive Pulmonary Disease, also known as COPD, is the third leading cause of death in the United States. COPD includes extensive lungs diseases such as emphysema, non-reversible asthma, specific forms of bronchiectasis, and chronic bronchitis. This disease restricts the flow of air in and out of the lungs. Ways in which these limitations may occur include the loss of elasticity in the air sacs and throughout the airways, the destruction of the walls between air sacs, the inflammation or thickening of airway walls, or the overproduction of mucus in airways which can lead to blockage. Throughout this paper I am going to explain the main causes, symptoms, diagnosis, and ways to reduce COPD.

A review of his medical record indicates a medical history of COPD-chronic, CHF-chronic and NIDDM-chronic. His medical record indicates that on 10/11/16 he saw Dr Mustafa for a complaint of SOB, cough, congestion and wheezing. He was prescribed a Medrol dose pak and ABT Azithromycin. On 10/14/16 again he saw Dr Mustafa for wheezing and productive cough because he did not obtain the previously prescribed medication from the pharmacy and again on 10/21/16 he saw Dr. Mustafa for SOB, weak and dizziness at which time he was referred to palliative care.

Some of the laboratory testing used to identify COPD are arterial blood gas and increased hematocrit levels. Arterial blood gas testing will reveals hypoxemia and hypercarbia due to the retention