COPD disease is caused by disorders of the respiratory tract with obstruction of the trachea caused congestion of the lungs as a result, the internal resistance of the pharynx increased while exhaling. The patient with dyspnea that is difficulty breathing because of the force required to breathe more. The patient was suffered from COPD especially in a chronic elderly patient to perform activities or do activities of daily living were decreased. In addition the length of hospitalization for long time, the cost of treatment is high, the loss of income that inability to work normally and when the disease get worsened which these are result in patients suffering, disillusionment and depression. The lack of control exacerbations on COPD potentially
| This is important because we need to look at the relevant data and realize that she seems to be in distress and first take care of that. Also realize that she seems to have an infection. With this information we are able to prioritize
History of Present Illness: Mr. Magnuson is a very pleasant 77-year-old gentleman who was previously seen in this office by Elvira Aguila, MD for COPD and hypoxic respiratory failure. He is here today for routine followed up. He was last evaluated in January 2015. Since that time, he states that his dyspnea is worse. He feels that it is related to the weather. He does state that he works around the house, although he does have significant functional limitations because of shortness of breath. His wife also confirms that he is able to do less and less. He has a stable, minimal cough. He is using 4L of oxygen at night as well as, as needed throughout the day. He continues to smoke three to four cigarettes on
History of Present Illness: Ms. Crouthamel is a very pleasant 76-year-old woman who I saw in April for a COPD and hypoxic respiratory failure. She has had stable symptoms of shortness of breath. She denies any significant cough. She does state that the humid weather can make breathing difficult at times. She remains active by participating in work in her garden on a daily basis. She is on supplemental oxygen 24 hours a day. She does also admit to occasional tobacco use.
Mr. Bishop is here for routine followup of his chronic illness. He is treated with Alvesco 160 mcg two puffs twice daily, Atrovent two puffs three times daily and Ventolin as needed for his COPD. He reports good compliance and uses these inhalers as prescribed. He generally uses his Ventolin with exercise. He reports that he is running 1-2 miles a day and also doing a step tape daily and reports good exercise tolerance. He does not wake at night coughing or feeling short of breath. For his hypertension, he takes hydrochlorothiazide 25 mg, and amlodipine 5 mg, and simvastatin 20 mg for his hyperlipidemia. He takes these as prescribed and denies any side effects. He denies
This is a case study on a 76 year old man.Mr Alan Chari(pseudonym used to protect the identity of a patient),was admitted over night in my department.He is a divorcee who stays with son.He is a retired teacher and his son is permanently employed by a local company as an electrician.He is independent with activities of daily livings but is occasionally limited by his ill health.He used to be a heavy smoker .After realising the burden COPD has on general New Zealand population ,affecting about15% of the adult population over the age of 45 years according to asthmanz( 2010) ,l took this case study to gain in-depth understanding.
The effectiveness of Family Support Caregiver in improving the Functional Status of Client with Pulmonary Diseases.
This case involves the assessment of the client’s problems. GL is a 62-year-old retired correctional officer she has come to the emergency room with SOB on exertion and worsening fatigue for the last few months. She was diagnosed with chronic obstructive pulmonary disease (COPD) as her symptoms have become more troublesome over the past month. She was diagnosed with COPD twenty-four months ago. GL was doing well on inhaled albuterol as needed but for the past 2 months has had a persistent cough and is more breathlessness. GL is a 62 year old retired, Hispanic, catholic, female who complains of shortness of breath on exertion and worsening fatigue for the last couple of months. She has not complained of chest pain or tightness. She has noticed slight foot and ankle edema and has gotten worse over past months. GL’s medical
Your topic is very interesting, when I practice as an ICU nurse I nursed many patients admitted with COPD exacerbations due to different etiology including unknown causes. I believe you bring up a very good point about not only obtaining an informed consent, but also offer education to the patients willing to participate in this research project. This is such a great intervention as the inform consent does not meet the educational needs required to provide full understanding of why this research needs to be completed and the benefits, and the impact that may have among this population. I am earger to read about your findings!! Great
D.Z., a 65-year-old man, is admitted to a medical floor for exacerbation of his chronic obstructive pulmonary disease (COPD; emphysema). He has a past medical history of hypertension, which has been well controlled by Enalapril (Vasotec) for the past 6 years. He has had pneumonia yearly for the past 3 years, and has been a 2-pack-a-day smoker for 38 years. He appears as a cachectic man who is experiencing difficulty breathing at rest. He reports cough productive of thick yellow-green sputum. D.Z. seems irritable and anxious; he complains of sleeping poorly and states that lately feels tired most of the time. His vital signs (VS) are 162/84, 124, 36, 102 F, SaO2 88%. His admitting diagnosis is an acute
Bronchitis COPD is a productive cough for at least 3 months along with airway obstruction. This condition would likely result in the clinical finding of crackles, wheezing, sputum production, and hypoxia and abnormally elevated carbon dioxide retention at rest. Bronchitis COPD involves inflamed airways obstructed by mucus whereas emphysematous COPD involves destruction of terminal airways leading to decreased space for gas exchange. In emphysematous COPD, one would have shortness of breath, dyspnea and may appear barrel chested. Also, hypoxia and hypercarbia aren’t as prominent in emphysematous COPD as in bronchitis COPD.
First of all, congratulations on decreasing the amount of cigarettes you are smoking everyday. There are quite a few options available to you that will help you as you strive to improve your health. As a nurse, I am here to help you become the healthiest you can be. In this letter, I outline your condition, factors in your environment that help and harm your condition, as well as identify goals and interventions to help you be at your best. I look forward to working with you!
The World Health Organization (WHO) recognizes dementia as a major public health priority and a significant cause of disability.[1] With the aging of the Canadian population, dementia as a serious public health issue will continue to grow. Several research reports point to a potential link between COPD and the development of cognitive decline.[2-5] Depending on the population group and methodology, about 10 to 61% of COPD patients have cognitive impairment.[2,6] The literature indicates that cognitive impairment in COPD patients may be associated with increased adverse outcomes such as more prolonged hospitalization, difficulty with the instrumental activities of daily living (IADL),[7,8] and trouble with managing their disease and adherence to treatment.[9-11] Moreover, the cognitive impairment could affect the success of pulmonary rehabilitation[12] and smoking cessation programmes[13] in patients with COPD.
Some of the laboratory testing used to identify COPD are arterial blood gas and increased hematocrit levels. Arterial blood gas testing will reveals hypoxemia and hypercarbia due to the retention
Chronic Obstructive Pulmonary Disease, also known as COPD, is the third leading cause of death in the United States. COPD includes extensive lungs diseases such as emphysema, non-reversible asthma, specific forms of bronchiectasis, and chronic bronchitis. This disease restricts the flow of air in and out of the lungs. Ways in which these limitations may occur include the loss of elasticity in the air sacs and throughout the airways, the destruction of the walls between air sacs, the inflammation or thickening of airway walls, or the overproduction of mucus in airways which can lead to blockage. Throughout this paper I am going to explain the main causes, symptoms, diagnosis, and ways to reduce COPD.
I will analyse the prevalence of the condition and what the potential causes may be. My interests have been directed to pre hospital care and community lead treatment packages, which are potentially available to the patient, as this is the acute environment, which I will have contact with in my employment as a paramedic. The initial reading was to understand COPD as a chronic condition, what is COPD? and its prevalence in the population. The (World health organisation, 2000), states that one in four deaths in the world are caused by COPD. In 2010 (Vos T Flaxman etal, 2012), says globally there were approximately 329 million, which is 4.8% of the population who are affected by this chronic condition, In the UK (NICE, 2010), have estimated that 3 million people suffer from COPD, with more yet to be diagnosed. This information about the amount of people living with this condition was surprising, as I little knowledge of its existence. During the early 1960’s (Timothy Q. Howes, 2005), says the term COPD had been designated as a single term unifying all the chronic respiratory diseases. Since then the term COPD, has been sub divided in to three umbrella areas, Bronchitis, Emphysema and Chronic asthma, which are separate conditions, which I have been previously aware of as their individual conditions. The 58 year old patient who we visited,