The patient is a 61-year old female who has a history of Chronic Obstructive PulmonaryDisease (COPD), hypertension, Coronary Artery Disease (CAD), and anxiety. The patient is familiar with the diagnosis and illnesses that have been contracted over the years and is knowledgeable about the disease processes, medications, and signs and symptoms of each disease. The patient came into the emergency department presenting with dyspnea, chest pain, weakness, and severe wheezing. The patient was diagnosed with an onset of a COPD exacerbation. During the hospital, stay chest x-ray and respiratory panel was done, showing positive for pneumonia. The patient continued to stay at the hospital until oxygenation status improved and the course of IV antibiotics
October 2014 had some unusually high temperatures for Southwestern Pennsylvania. While most residents were enjoying the summer like weather, I came to find out that not all were. Extremes in weather can trigger COPD exacerbation. COPD symptoms, such as cough, phlegm production, and shortness of breath, tend to get worse for some patients when the air is very cold or when it is hot and humid. The body is always working to try to maintain a normal body temperature, which is about 98.6 F. When exposed to extreme temperatures, such as during the heat of summer, the body uses extra energy trying to cool itself down in order to maintain normal body temperature. The use of extra energy causes the body to demand more oxygen. People with COPD often
This is a case study on a 76 year old man.Mr Alan Chari(pseudonym used to protect the identity of a patient),was admitted over night in my department.He is a divorcee who stays with son.He is a retired teacher and his son is permanently employed by a local company as an electrician.He is independent with activities of daily livings but is occasionally limited by his ill health.He used to be a heavy smoker .After realising the burden COPD has on general New Zealand population ,affecting about15% of the adult population over the age of 45 years according to asthmanz( 2010) ,l took this case study to gain in-depth understanding.
The most likely bacterial causes of infective exacerbation of COPD are: Haemophilus influenzae, Moraxella catarrhalis, Streptococcus pneumonia, Haemophilus parainfluenzae, Staphylococcus aureus, Pseudomonas aeruginosa, gram-negative Enterobacteriaceae, Klebsiella pneumonia and Mycoplasma pneumoniae (S.Sethi, 2004; T. Kawamatawong et al, 2017; King et al, 2013).
The effectiveness of Family Support Caregiver in improving the Functional Status of Client with Pulmonary Diseases.
This assignment will explain the pathophysiology of the disease process chronic obstructive pulmonary disease (COPD). It will examine how this disease affects an individual looking at the biological, psychological and social aspects. It will accomplish this by referring to a patient who was admitted to a medical ward with an exacerbation of COPD. Furthermore with assistance of Gibbs model of reflection (as cited in Bulman & Schutz, 2004) it will demonstrate how an experience altered an attitude. In accordance with the Nursing and Midwifery Council, (NMC) Code of Professional Conduct (NMC, 2005) regarding safeguarding patient information no names or places will be divulged. Therefore throughout the assignment the patient will be referred to
Your topic is very interesting, when I practice as an ICU nurse I nursed many patients admitted with COPD exacerbations due to different etiology including unknown causes. I believe you bring up a very good point about not only obtaining an informed consent, but also offer education to the patients willing to participate in this research project. This is such a great intervention as the inform consent does not meet the educational needs required to provide full understanding of why this research needs to be completed and the benefits, and the impact that may have among this population. I am earger to read about your findings!! Great
The World Health Organization (WHO) recognizes dementia as a major public health priority and a significant cause of disability.[1] With the aging of the Canadian population, dementia as a serious public health issue will continue to grow. Several research reports point to a potential link between COPD and the development of cognitive decline.[2-5] Depending on the population group and methodology, about 10 to 61% of COPD patients have cognitive impairment.[2,6] The literature indicates that cognitive impairment in COPD patients may be associated with increased adverse outcomes such as more prolonged hospitalization, difficulty with the instrumental activities of daily living (IADL),[7,8] and trouble with managing their disease and adherence to treatment.[9-11] Moreover, the cognitive impairment could affect the success of pulmonary rehabilitation[12] and smoking cessation programmes[13] in patients with COPD.
During the second week of my clinical rotation, I had the privilege of being with the respiratory department, as a result of that my research of the Continuity of Care topic will be based on the topic of COPD (Chronic Obstructive Pulmonary Disease). COPD patients are usually readmitted due to acute exacerbations also known as (AECOPD). (Laverty et al., 2015). In this paper, we explore the COPD discharge care bundles which have been developed by different authors. The care bundle consists of a short list of certain evidence-based practices to be utilized or implemented before discharge for all patients who have been admitted with COPD, based on reviewing national guidelines, evidenced based practices, expert opinion, other relevant literature, peer-reviewed journals and patient consultation. (Hopkinson et al., 2012).
As a result of R.S.’s chronic obstructive pulmonary disease (COPD), he’s going to be experiencing the many consequences of his type B COPD, otherwise known as chronic bronchitis. Copstead & Banasik (2013) states that the pathogenesis for chronic bronchitis has changes in the airways that include swelling and chronic inflammation of the bronchial mucosa which can lead to scarring. In addition, there is hypertrophy of mucosal glands and goblet cells, which leads to increased mucosal production. In chronic bronchitis, there’s also a hypersecretion of bronchial mucous (Copstead & Banasik, 2013, p. 483). In normal conditions, the mucus produced by the mucus glands aids in the protection of the lungs by capturing the foreign particles that enter into the lungs. Bellamy & Booker (2004) report that when long-term smokers such as R.S., who are already developing chronic airflow obstruction, the excess of hypersecretion of mucus contributes to the decline of lung function. Long-term production of mucus may cause the patient to suffer lower respiratory tract infection (p. 19).
Based on a case study for a 76 year old female, Betty White, presenting to a medical ward with an acute exacerbation of chronic obstructive pulmonary disease (COPD), this paper will firstly outline a brief summary of COPD and discuss the associated risk factors. Secondly, the patient’s information will be summarised. From the perspective of the primary Registered Nurse, this paper will detail steps of an initial clinical assessment of the patient. In doing this, the priorities and considerations involved in order to provide best patient care for this scenario will be addressed. A discussion of information and suggested interventions will be integrated as to how the nurse shall develop a plan of care. Furthermore, it will outline
The positive outcome of the acute treatment of the patient helped me feel more confident in communicating with patients. The patient commented on his appreciation of having the ambulance service available, in his time of need. I told the patient this was my first week on the road, and he said that he was happy with my performance. My paramedic mentor gave me positive feedback on my ability to communicate well with the patient. The treatment package contributed to a good understanding of how the therapeutic respiratory drugs worked and how quickly they became effective. I found out that COPD patients should only permitted to have increased oxygen levels for no longer, than six minutes as stated in (section 27 of B R O’Driscoll, etal
All over the world, chronic obstructive pulmonary disease (COPD) is a very significant and prevalent cause of morbidity and mortality, and it is increasing with time (Hurd, 2000; Pauwels, 2000; Petty, 2000). Due to the factor of COPD being an underdiagnosed and undertreated disease, the epidemiology (Pauwels, Rabe, 2004) is about 60 to 85 % with mild or moderate COPD remaining undiagnosed (Miravitlles et al., 2009; Hvidsten et al., 2010).
Pathological changes characteristic of COPD are found in the airways , lung parenchyma and pulmonary vasculature .These include chronic inflammation, with increased numbers of CD8 lymphocytes in different parts of the lower respiratory system and structural changes which result from repeated injury . Inflammatory and structural changes increase with disease severity and smoking .(43)
Smoking can take a serious toll on your health. If you have been smoking for several years, you could be a risk for developing COPD. Fortunately, you can slow the progression of COPD. Quitting smoking takes a tremendous amount of will power, but you can kick the nicotine habit. Many people increase their caffeine intake to get through the withdrawal stage, and water can also be used to kick the tobacco habit. Increasing your water intake will flush toxins out of your body. Most people start to notice positive changes within one week of quitting.
COPD is a lung disease that makes a person hard to breath. This can cause damage to the lung over the years and this is usually caused from smoking. COPD is a mix of two diseases chronic bronchitis is the airway that carry air to the lung that causes the lung to inflame and make a lot of mucus. This can cause or block the airways that will make it hard to breath. Then there is emphysema where this is a healthy person, Tiny air sacs in the lungs and look like balloons. As the person breathe when inhaling and exhaling. Emphysema are air sacs that damage and lose their stretch. This will cause for less air to get in and out the lungs, in which it will make you feel out of breath.