Carmonic-Tonic Seizure: A Case Study

In this scenario, we have Debbie Young who has been a special education teacher and assistant principal in a school district in the south prior to becoming a high school principal. One day Debbie is approached by parents of a tenth-grade student named Jonathan, who has several disabilities that require constant care by a trained nurse. Jonathan is profoundly mentally disabled and suffers from spastic quadriplegia as well as having a seizure disorder. Principal Young turns down the parents’ request because of the expense of having a student with these conditions at the school and the view that the school is not the most appropriate placement for Jonathan.

It was believed a seizure in the brain caused by the electric current would stimulate the release of neurotransmitters, and would allow the brain to reorganize to function correctly (Nasar). Neurotransmitters are chemical substances that transmit signals that facilitate cell to cell communication throughout the nervous system, and play a key component in the biological paradigm for treatment of schizophrenia. At this early stage of understanding schizophrenia’s pathology, it was believed these chemicals were imbalanced and shocking the brain would force a rebalance of these vital chemicals. Once rebalanced, it was hypothesized the patient would no longer suffer from schizophrenia (Tharyan, 2005). The insight into treating neurotransmitter imbalances in patients diagnosed with schizophrenia began the treatment path to present day antipsychotic pharmaceutical drug therapy. While the actual effects of electroshock therapy on patients with schizophrenia are unclear, it is still used today to treat schizophrenic patients who are resistant to pharmaceutical drug therapy. Electroshock therapy was the utilization of insulin coma therapy and electroshock therapy to treat schizophrenia were successful partly due to the required hospital admittance and doctor administered methodology. In both treatments the patients were required to be admitted into a hospital and were not responsible for administering the treatment themselves. While both treatment methods were viewed as inhumane and

MR. David likewise takes atorvastatin (Lipitor); 10 mg every day, for hypercholesterolemia (hoisted LDL cholesterol, low HDL cholesterol, and raised triglycerides). He has endured this medicine and holds fast to the day by day plan. Amid the previous 6 months, he has likewise taken chromium picolinate,

There are many current drugs and therapies for seizure illnesses. One example of these drugs is, carbamazepine (epitol). Epitol, according to webmd.com, works in the sense of

Status epilepticus are seizures lasting >20 minutes, or repeated seizures without regaining consciousness [71]. Physicians should be particularly careful with it, since the background may be other than epilepsy. Infection, haemorrhage, trauma, hypoxia, ischemic or haemorrhagic stroke, hypoglycaemia, hyponatremia, drug toxicity and alcohol withdraw should be ruled out. In any of the above, the provoking cause should be treated. If the cause is epilepsy, the treatment of choice is benzodiazepines and the second choice is phenytoin/ fosphenytoin, but with caution as rapid infusion may induce cardiovascular problems. For resistance in those two drugs, the Neurocritical Care Society’s Status Epilepticus Guideline

Aidan is an11 year old boy in the 6th grade at Valley Central Middle School. Aidan was initially referred to CSE in November 2010 during kindergarten. Aidan was born premature at 33 weeks and had difficulty breathing upon birth. He was prescribed medication during infancy to mature his lungs. Aidan had significant communication delays and it was noted that Aidan utilized gestures to communicate. Aidan’s academic testing indicated very low reading and math skills. His cognitive testing indicated that Aidan’s overall cognitive skills are in the very low range. Aidan’s speech evaluation results indicated receptive and expressive language skills in the very low range. Aidan demonstrated difficulties with pencil grasp and did not demonstrate a

The amplitude of the fluctuations, the frequency spectrum and duration of RMS changes are the main causes of flicker phenomenon.

The patient is a 53-year-old African American female who was recently hospitalized from 1/19 -1/22, had a complete workup for seizure disorder, was discharged home and she represented to the ED several times on the 23rd complaining of seizures. She present after having a single isolated seizure which lasted 20 minutes witnessed by her family. She also presented earlier in the evening of the previous evening because she continued to have seizures. She is a history of CVA, hypertension, bipolar disorder. The patient has required intubation in the past. Her laboratory work is essentially unremarkable, phosphatase of 26, AST of 72 and ALT of 75. Dilantin levels are not reported at this point. The case was discussed with Dr. Amr Kahf. I

This is 36 year old AAF. Patient reprots she has a history of seizure disorder, and currently taking carbamaaazepin 200 mg QID. Patient has NC Medicaid. Disusse with the patient to talk with ARMS Medicaid/Medicare Specialist. Patient was told to return to the clinic in 2 months for carbamazepine level. Also patient is to sign the consent for medical records from NC PCP. Patient reports generalized weekness and tireness, requestign elevator pass at her group home. Patient denies chest pain, SOB, n/V/D,or

If you are with someone having a tonic-colonic seizure (a generalized seizure in which the muscles suddenly stiffen and the person may fall), you should try to:

Many forms of statins are present in the markets including atorvastatin (Lipitor), lovastatin (Mevacor, Altocor) and simvastatin (Zocor). Inhibition of 3-hydroxy-3-methyl glutaryl coenzyme A reductase (HMG-CoA reductase), a key enzyme in the cholesterol biosynthetic pathway is the mode of action of most of the statins (2).

Imagine what it would be like to live through unpredictable seizures. At any given moment, having to fight through health problems that have no proven cure. Syndromes of this sort are fatal and can be overwhelming to one 's being. Epilepsy in this case, has effected and changed the lives of millions throughout the world.

Benign epilepsy with centrotemporal spikes (BECTS) is one of the more common epileptic disorders in children. It is also known as Benign Rolandic Epilepsy. Onset occurs between the ages of 3 to 13 and BECTS will run its course by adulthood.

Seizures are described into two major groups of seizures: primary generalized seizures and partial seizures. The main difference between both of these seizures is how it begins. Partial seizures begin in one area of the brain and affect the part of the body being controlled by that area of that brain. Generalized seizures affect the whole area of the brain and occur at the same time. They begin and spread rapidly making it impossible to identify the origin.

Partial seizures also referred to as focal seizures, have abnormal excited neuron activity on one part of the brain and in some cases it can increase to other parts of the brain. These specific areas are called the seizure focus. As the activity typically stays around where it started from, there is the potential for it to spread due to the failure of inhibitory mechanisms. Within the seizure focus, neurons experience depolarization, which is then followed by a line of action potentials. The activity involved is defined as proximal depolarizing