Cerebral air embolism following pigtail catheter insertion
Abstract:
Pleural pigtail catheter placement associated with many complications including pneumothorax, hemorrhage, and chest pain. Air embolism can rarely be a complication of pigtail catheter insertion and has a high risk of occurrence with positive pressure ventilation (PPV).
Cerebral air embolism is a very rare complication but lethal. We report a case of patient of cerebral air embolism as a complication of placement of pigtail pleural catheter placement while a patient on (PPV).
Case presentation:
A 50 years old male who has bilateral pneumonia, was found to have pneumothorax while on mechanical ventilation CPAP/PS mode. While placing the pigtail catheter, the patient
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Keywords:
Cerebral air embolism-Pigtail catheter-Pneumothorax- Chest Tube
Introduction:
Placement of a chest tube indicated for pleural effusion and pneumothorax. There have been two kinds of chest tubes being used, including a Pigtail catheter and chest tube thoracotomy.
The pigtail catheter drainage is widely used as it is easier and less invasive
Cerebral air embolism is a rare complication that can be induced by pulmonary barotrauma, the trauma of the chest or head and iatrogenic causes such as invasive procedures or surgery. This risk increases if the patient is on positive pressure ventilation and while the pressure in the airway increased.
We reporting a case of cerebral air embolism associated with pigtail catheter insertion for treatment of pneumothorax in a patient who was on positive pressure mechanical ventilation.
Case Report:
This is a 50 years old male with no significant past medical history presented initially with shortness of breath and hypoxia and was transferred to the ICU. He was treated for bilateral pneumonia that required prolonged mechanical ventilation via a tracheostomy. He has necrotizing pneumonia and he has been in the hospital for 6 weeks due to the development of multi-organ failure. He was weaned from mechanical ventilation to the point he was tolerating a CPAP/PS mode. Later on, it was noticed that he
Throughout my clinical rotation, the only concern for this patient was pain management and discomfort from the chest tube site. The patient was given Ketorolac for a pain of 4/10 using the numeric pain sale. The patient was also at risk for pneumonia and pressure ulcers from immobility and not coughing/deep breathing. Nursing interventions were getting the patient up to the chair and using the incentive spirometer ten-times per hour. Another concern was SOB and fatigue with activities.
his neurologic intensive care unit (NICU) stay, Y.W. was intubated and placed on mechanical ventilation, had a feeding tube inserted and was placed on tube feedings, had a Foley catheter to down drain (DD), and had multiple IVs inserted. He developed pneumonia 1 month after admission.
Chest tubes are used to drain fluid or remove air from the patient’s chest area. My patient had a chest tube to drain his pleural effusion 26mL
At CTPA study performed at the time excluded any pulmonary emboli and the report made comment of a moderate sized right-sided pleural effusion with compressive atelectasis. There was no comment on the report of any parenchymal infiltrate and I have not cited the images myself. CRP was 113. He was given a presumptive diagnosis of pneumonia with parapneumonic effusion and commenced an Augmentin Duo Forte and doxycycline. In
38. American Journal of Respiratory care and critical care Medicine, Volume 175, issue 7, pages 698 – 704
Pt approached staff 2200 stating, she was having a hard time breathing. Pt also stated her tongue was swollen from an allergic reaction. Mild tongue swelling noted. After assessing the patient, she had bilateral audible wheezes and o2 stat at 96%. No s/s of respiratory distress noted. Pt received a nebulizer treatment at 2205 and was fine after tx, stating "my breathing improved." Prn Bendaryl was also given after a swallow evaluation. No further medical complaints. Slept well through the
Tarbox, A., & Swaroop, M. (2013). Pulmonary embolism. International Journal of Critical Illness and Injury Science, 3(1),
In 1974 Ward reported an incidence of 3% of symptomatic pneumothorax after ISB by paraesthesia blind technique. The pneumothorax was almost certainly produced by the prior attempt to carry out a supraclavicular block, as it is difficult to imagine the apex of the lung reaching as high as C6, the level of an
CBC: Hgb 14.4; Hct 42.1; WBC 0.4(L);RBC 4.59, Aterial blood gas: FI02 .44; pH of 7.38, PO2 97, PCO2 31 and HCO3 21.3 showed hypoxemia persisting and slightly alkaline, with decreased pCO2 of 31, suggesting some level of hyperventilation. EKG revealed sinus tachycardia and no specific S-T-T wave. CXR revealed bibasilar alteletasis. No pneumothorax or significant pleural effusion. The patient had initially been started on a bi-nasal cannula on 6L/min, but patient was not able to get O2 up so physician ordered a Bipap. While the patient
All patients were followed with pulmonary artery catheters and invasive blood pressure. After orotracheal intubation, patients were ventilated with intermittent positive pressure with a tidal volume of 8 mL / kg, final expiratory positive pressure of 5 to 8 cmH2O and FiO2 of 60 to 100% to maintain arterial oxygen saturation above 95%.
In this paper, we will cover multiple things. I will cover what IV stands for, what peripheral intravenous catheters are and what they are used for. I will even discuss sites for peripheral IV’s. I will explain what a central venous catheter is, what it is used for, and the types of catheters out there. I will explain the procedure on how to insert one, how to change the dressing, the safety guidelines to follow for insertion, and how to discontinue a central venous catheter. Discussion of what a patient needs to know about having a central venous catheter is included. Equipment to have ready for insertion is vital to know. It will explain why sterile procedure is used for insertion and what to monitor when a patient has a central venous
Air embolism is a rare but potentially fatal consequence of air entering the vascular system. It can result from a wide range of procedures, including those related to vascular access in interventional radiology, in addition to open surgical procedures. We set out to review all cases of air embolism at our institution over a 25-year period, including analysis of cause, clinical signs and symptoms, treatment and prognosis.
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