Color Blindness and Testing in Children Essay

A baby’s genes are determined at conception. If something is faulty at this stage, this can have a huge impact on the child’s development. This may be physical or intellectual. If a mother takes drugs or smokes when pregnant, there is an increased chance of delivering an underweight baby at birth. It can also lead to cognitive problems for the child as they get older.

Sometimes special blue lights are used on infants whose levels are very high. This is called phototherapy. These lights work by helping to break down bilirubin in the skin.

When children come into the world they are not born hating anyone, in fact they are born completely helpless and dependent on another person to care for them. Children are also dependent on others to learn. They come into this world needing to feel protected and loved, so why do we teach them to hate? Why not instead teach them to love? There are many things that will need to change in our society to dismantle racism; however it will need to start with our children. My paper will show who is oppressed, who is oppressing, and will compare and contrast what has happened in history, and to where we will need to start to end racism.

as a intangible factor. Colored children not having the ability to study, to engage in discussions and

colorblind practices can be inequitable. I then draw on normative principles from social theory to defend

Colour blindness can is often carries the mutated gene on the X chromosomes that it passed 50% of this over to the child, the mother may not have had colour blindness but it would most likely be carried on one of the X chromosomes, If the mother is the carrier and the father doesn’t have colour blindness there is a one in two chance that the son may have the disease while for the daughter there is a one in two chance that she would be the carrier of the disease and there being no chance in her getting it. (National Eye Institution, Accessed 22.08.2015). If both of the Parents are carriers that means that there is a one in four chance that the child would most likely have the disease, one in two that the child is a carrier of the disease or a one in four chance that the child isn’t a carrier of the disease and also doesn’t have the disease although it is most likely that one of the children would end up getting colour blindness if one of the parents happen to have it (National Eye Institution, Accessed

How eyes see color is pretty simple, our retinas approximately six million cone cells. Each cell in the retina is assigned to a certain color. There are three different types of cone cells, blue, green, and red, which correspond to the three primary colors. Being colorblind is more likely to happen to boys than to girls. By reading the article after the colorblind test I also learned that people who suffer from being color blind are not blind to a color, but have trouble seeing a certain shade of a color. Another, thing that I learned is that being colorblind can be inherited genetically or if you damage one of genes “photopigments” you also can become color blind. If I had never read this article, I would have never learned all this interesting and great knowledge to

Living in a society where being colorblind is now a trend, it is hard for most individuals to believe that racism still exits, and still taunts minorities through laws. Although transitioning away from slavery and the civil rights movement has assisted African Americans’ in longer being suppressed in society— another obstacle of suppression is a live, and runs through the criminal justice system. In this paper you will see how and why drug laws were created and its effects, how law inequality is supported by racial discrimination; criminal justice policies and its leading to the rise of incarceration rates, the promotion of the Rockefeller drug law by African Americans’, and Clinton’s reaction toward the 1994 law.

Shading vision issues may make it harder for kids to learn and read, which can prompt poor schoolwork and low self-regard.

The article “Gene therapy for red- green colour blindness in adult primates” by Katherine Mancuso and her colleges is about the possibility of curing color blindness. This test was done on adult squirrel monkeys that were missing the L- opsin gene. Out of the three cones humans have (short (S), long (L), or the middle (M) photoreceptor) only the L-or-M cone is responsible for red- green colour blindness. Many female squirrel monkeys have the ability to access all three photoreceptors giving them the ability for trichromatic color vision, but males are dichromats meaning they are missing the L-or- M gene causing them to be color blind. In order to correct this color deficiency a third type of pigment was added to the monkeys retinas to provide them with the receptors that are necessary in order obtain trichromatic color vision. Over the span of a year the scientists observed that before the treatment the monkeys couldn’t decipher between blue green and red violet. After they started to develop a new pigment (due to the injection) in the cone photoreceptors scientists discovered that the monkeys now reacted to the colors they couldn’t see before. The scientists concluded primates benefited from the injection and that they were able to see colors they were invisible to them before, and that the findings in this experiment could

Introduction: The gene that codes for red-green color blindness, the most common form of color blindness in humans, is found on the X chromosome. One in 12 males is color blind. In females, however, color blindness affects only about 1 in 200. Why is

In able to know for sure of albinism, tests may be run. Tests for vision could

Red-green colorblindness is usually inherited in an X-linked recessive way and it affects an approximated 6 percent of men in the world. This means that, the disorder is usually passed to the affected persons through the X chromosomes thus making the condition to be experienced more by men than

The gene carrying the defect that produces albinism is recessive, which means that both parents must carry this recessive gene in order to produce a child with the condition. When both parents carry the gene (and neither has albinism), there is a one in four chance with each pregnancy that their child will have albinism. The inheritance pattern of ocular albinism is alittle different. This condition is X-linked, meaning that the recessive gene for ocular albinism is located on the X chromosome. X-linked ocular albinism appears just about only in males who inherit the condition from their mothers.

That type affects one out of every sixty-thousand males around the world, though it is much less common in females. Nearly one out of every seventy people are a carrier for the albinism gene. That means that even though they carry the albinism gene that does not mean they show any outward sign of having it. To get albinism both parents must have the gene, and the child has a twenty five percent chance of having albinism, a fifty percent chance of being a carrier, and a twenty five percent chance of having two normal genes.