Comparing The Data And The Treatment Plan For Initial Management Of Svco

The registered nurse must be observant of any clinical signs that require further assessment such as the signs and symptoms stated earlier in this paper. Previously, nurses were taught to use the Homan’s sign to assess for a DVT. After extensive research, it has been found that the Homan’s sign is not as effective as previously documented. Therefore, after additional research, the registered nurse can use the Wells model when assessing a patient for a DVT. The Wells model is a numerical scale similar to other scales used within the nursing practice and consists of a scoring system that predicts the likelihood of a DVT. The Wells model assesses for the following criteria: if a patient has active cancer, any paralysis, if the patient has been bedridden for more than 3 days or had a major surgery within the last 4 weeks, Is the patient experiencing any local tenderness, any swelling or pitting edema in the leg or calf, or is there an alternative diagnosis that is more likely than a DVT. These areas are scored with a number 1 if positive and a 0 if negative. A score of 3 or more would indicate a high probability for a DVT. A score of 1 or 2 would indicate a moderate probability, and a score of 0 would indicate a low probability of a DVT. When doing an assessment, the registered nurse can quickly use the Wells model to determine if further assessment is

Physical Examination: General: The patient is an alert, oriented male appearing his stated age. He appears to be in moderate distress. Vital signs: blood pressure 132/78 and pulse 68 and regular. Temperature is 38.56 oC (101.4 oF). HEENT:Normocephalic, atraumatic. Pupils were equal, round, and reactive to light. Ears are clear. Throat is normal. Neck: The neck is supple with no carotid bruits. Lungs: The lungs are clear to auscultation and percussion. Heart: Regular rate and rhythm. Abdomen:Bowel sounds are normal. There is rebound tenderness with maximal discomfort on palpation in the right lower quadrant. Extremities: No clubbing, cyanosis, or edema.

Also, a cardiac ECHO with Definity study was scheduled for 5/22/17 to reevaluate the LV thrombus, pending which a decision for anticoagulation was to be made. Inpatient Neurology evaluated him on 5/20/17, and also mentioned in their consult note that the patient had an acute CVA in April 2017, and recommended continuation of ASA and Plavix, along with considering initiating statins, and that MRI brain can be done after 6 weeks of stent placement if there were no contraindications (Although per the Neurologist’s note – the patient informed that he had PTCA 1 month ago, while in reality, his stent was placed in 3/2017; and a MRI of the brain was never subsequently done during this admission).

The blood vessel that Dr. Eltahawy was concerned about was thin and looked as though it would collapse in on itself. First, Dr. Eltahawy tried a balloon catheter. At the top of the catheter was a small balloon that could inflate to maintain a shape or structure of a blood vessel. However, because the blood vessel was so thin, the balloon catheter was not very effective. Instead, Dr. Eltahawy installed a stent. A stent is a wire mesh tube. It is placed in a blood vessel permanently to maintain the shape of the vessel and to allow for the free flow of blood. Throughout this entire independent study, I realize that shadowing in the medical field is less about learning about medicine and more about discovering about whether becoming a doctor is the right step. This independent study has been a step in the right

Rose and Eddy have a strong bond which each of them stands up for each other with issues in the family

Few days back, the patient had a CABG surgery and was send home under stable conditions. Family member noticed SOB and weakness from the patient and was directed to attend the ED. As they got to the ED, the emergency department nursing staff noticed SOB with pericardial hematoma and immediate drainage was necessary. A chest tube was placed as a treatment option.

Venous thromboembolism refers to the formation of a blood clot in a blood vessel. While clots can form in an artery or a vein, this article focuses only on clots that occur in a vein ("," 2015). Critically ill patients are at an increased risk of a venous thromboembolism (VTE) due to VTE can manifest as a deep venous thrombosis (DVT) or a pulmonary embolism (PE). Risk factors include venous stasis, vascular injury, and hypercoagulable disorders. A majority of ICU patients carry at least one risk factor for VTE; additional risk factors are considered to have a cumulative effect…it is impossible to predict which patients will experience a

There were several dependent variables which were significant predictors of complications. First females older than 70 with HTN and renal failure were identified. Second the presence of a venous sheath was found to be an important indicator and finally female gender with a high BMI stood out as well for a higher complication

The obstruction in Room 40’s leg was discovered using a diagnostic arteriogram. An arteriogram is an image of the interior of blood vessels, such as arteries, by the use of electromagnetic waves. During this procedure, a dye is injected into the artery revealing any bulges, weakness, lesions or occlusions of the blood vessel. The ability to see any evidence of a a plaque formation or obstruction within a vessel makes an arteriogram an effective method for detecting atherosclerosis (Johns Hopkins Medicine, 2015).

Venous Thromboembolism (VTE) is a disease that embodies both pulmonary embolism (PE) and deep vein thrombosis (DVT). On the side of research, VTE is the most common preventable cause of hospital-associated mortality and is both the second most common medical complication and cause of excess length of hospital stay (LOS). Furthermore, with appropriate evidence-based prophylactic measures, the occurrence of hospital-associated VTE (HA-VTE) can be reduced by as much as seventy percent. Subsequently, there are approximately between 200,000 and 300,000 VTE-related deaths annually, which is greater than the yearly rate of mortality for breast cancer,

Pt also has a history of deep vein thrombosis (DVT) with an inferior vena cava filter to capture any lose blood cells. The chest pain he is experiencing is no cardiac secondary to chest wall pain which is symptoms of his small cell carcinoma of the lung. Patient is required to use oxygen as needed for his SOB secondary to his lung cancer. No evidence of a compression fracture was found in the spine, patient has prior laminectomy postoperative changes in the lower lumbar secondary to previous falls.

There were no differences in age and education between the OSA group and the non-OSA group. There were higher rates of male patients (67% vs. 20%, p< 0.03) compared to those in the non-OSA group. Further, among those in the highest quartile of ischemic microvascular burden (highest quartile of both DWMH and PVH volumes), 80% of them were in the OSA group. Moreover, patients in the OSA group had a higher rate of having advanced lesions, such as beginning confluence of foci or large confluent areas, compared to those in the non-OSA group (t=2.96, p<0.001).

A Deep Venous Thrombosis (DVT) will propagate when there is either stasis of blood flow, endothelial injury, or if the patient is in a hypercoagulable state. Under physiological conditions, a dislodged DVT can predictably settle in the pulmonary arteries causing a pulmonary embolism. However, a DVT in the presence of a intracardiac shunt or PAVM can paradoxically cause an embolism in branches of the aorta. The pathophysiological mechanism varies depending on the etiology of the paradoxical embolism. For instance, in a paradoxical embolism due to a PFO, a DVT gets dislodged and enters the right atrium where a transient increase in right atrial pressure during a Valsalva maneuver can force the embolism through the PFO and into the left atrium

A: Janie is a 60 year old Female with PMH of A-Fib, COPD, Hypothyroidism, HTN, Lung Cancer and recently diagnosed Pulmonary Embolism. Janie presents to ER for evaluation on SOB, cough with greenish sputum, sore thoart, hoarseness and generalized weakness. Janie lives at home with her husband, use to smoke ½ pack per week, but quit many years ago, denies alcohol or drugs. Family history is non-contributory. Allergies: NKDA. Differential diagnosis includes worsening Lung Ca, PE, COPD and CHF. Janie uses home O2 at 4 L/NC. V/S: T=98.7, HR=89, R=16, B/P=132/56, O2 sats=100% on 4L/NC, Pain=6/10. Labs: WBC=7.6, H&H=8.5/27, Na=141, Troponin=0.08/0.06, BNP=495, INR=4.2, UA=3+ protein, 1+ blood and 6-10 RBC. CXR: Impression:1). COPD with nonspecific coarsening of the basilar interstitium. 2). Mild cardiomegaly with borderline cardiac compensation. 3). Right

The study hoped to find an acceptable correlation with abnormal or normal Doppler ultrasound and venography. The results were not sufficient to determine that Doppler ultrasound alone was an adequate tool to determine stent malfunction consistently. Future objectives are for non-invasive Doppler testing to be sensitive and reproducible enough to detect early stent malfunction even before symptoms occur.29 Boyer,23 reports that Doppler ultrasound has sensitivity and specificity rates from 70%-100% in cases of stent stenosis, but it is still not ideal. Ultrasound requires a technologist and radiologist with experience and must be conducted with consistent parameters to obtain dependable results. Most of the trials recommend follow up studies including some or all of the following: venography, Doppler ultrasound combined with two-dimensional ultrasound, angiography, magnetic resonance imaging and computerized