Table of Contents I. Introduction to Type 1 Diabetes Mellitus a. What it is b. Prevalence and demographics c. Treatment II. Cognitive Outcomes: Mood and Memory a. Memory i. Neural Basis of Memory ii. Cognitive Outcome of TIDM on Memory III. Structural Brain Changes: MRI Studies a. The Hippocampus i. Purpose and Function of the Hippocampus ii. Previous studies on T1DM and Hippocampal Volume iii. The Cognitive Effects of T1DM on Hippocampal Volume b. Other Brain Areas IV. Conclusion a
Maggie Roman MED 2056 Cohort FT31 Diabetes Mellitus Mrs. Annabelle June 29, 2015 The human body achieves homeostasis through the coordination of organs and different systems throughout the body. In particular, the endocrine system plays a functional role in regulating the body’s physiological activities via chemical substances, known as hormones. The endocrine cells secrete hormones in response to body signals in a negative feedback loop, which is a self-regulatory response intended to
Type 1 insulin dependent diabetes mellitus is most commonly diagnosed in children and adolescents but can sometimes be diagnosed in older age. It is defined as a chronic condition in which the pancreas does not produce insulin which is needed to allow glucose, known as the bodies source of fuel, to enter the cells. Type 1 diabetes does not have a cure but can be managed with proper treatment of insulin therapy. Type 2 non-insulin dependent diabetes mellitus is commonly caused by genetics, obesity
Type one diabetes mellitus (T1DM) is a genetic autoimmune metabolic condition that requires diligent self-management and monitoring. Poorly managed or untreated T1DM can lead to serious complications and even death. Type 1 Diabetes occurs when the insulin producing beta cells also know as islet cells, produced in the islet of langethan, are attacked in the pancreases resulting in little to no insulin production. This leads to elevated levels of blood glucose (more than 8mmol/L) and if left uncontrolled
Topic Intensive glucose control of patients with type 1 diabetes mellitus to maintain blood glucose levels (BGLs) near normal range Introduction Type 1 diabetes is an autoimmune disorder which can occur from insufficient insulin secretion. Insulin is a hormone which is secreted by the pancreas and regulates BGLs (Loghmani, 2005; Nussey & Whitehead, 2001). Type 1 diabetes is also known as insulin dependent diabetes mellitus (IDDM). In this, the body produces no, or very little, insulin which can
This report investigates children living with the Chronic condition of Type 1 Diabetes Mellitus (T1DM). Chronic illness is a disease or condition that is persistent, with long term effects for an individual. Chronic is applied, when a condition last for a duration of three months or greater. According to the World Health Organisation (2017), Diabetes is one of the leading causes of mortality in the world, representing 60% of all deaths, along with heart disease, stroke, cancer and chronic respiratory
General Hospital for treatment of diabetic ketoacidosis (DKA) and new onset type 1 diabetes mellitus. DKA is an emergency situation that results in 100,000 hospitalizations in the US yearly, a 9% mortality rate, and treatments of reportedly 1 billion dollars per year (Katsilambros, Kanaka-Gantenbein, Liatis, Makrilakis, & Tentolouris, 2011). Presenting to the emergency room with DKA is the first manifestation of type 1 diabetes in 30% of cases (Katsilambros et al., 2011). This paper will examine Mr
Diabetes Mellitus Type 1 is a condition that results from the autoimmune destruction of insulin producing beta cells in the pancreas. This type of Diabetes Mellitus accounts for about 5% of cases of diabetes. It affects males and females equally, and is usually diagnosed under the age of twenty. I was diagnosed with Juvenile Diabetes when I was eleven. I had been experiencing the common symptoms of extreme thirst, frequent urination, weight loss, and sugar cravings for a few weeks. I was taken to
Introduction: Type 1 Diabetes Mellitus is a disease that occurs when insulin-producing beta cells of the pancreas are damaged or are being destroyed (Mahan, Escott-Stump & Raymond, 2012). The gradual destruction of the beta cells of the pancreas usually leads to complete insulin deficiency, which ultimately manifests itself in the following symptoms at clinical onset: “high blood glucose (hyperglycemia), frequent urination (polyuria), excessive thirst (polydipsia), and a significant amount of weight
Type 1 diabetes mellitus is an autoimmune disease that results in insulin dependence due to failure of secretion of the hormone insulin by beta cells in the pancreas. Type 1 diabetes usually manifests in childhood, < 20 years of age – the reason for which it was formerly termed juvenile diabetes, and is more prevalent in Caucasians then in any other ethnicity (Diabetes Basics, n.d.). The key to proper management is early detection, patient education, and most importantly patient compliance. The