Diagnosis And Treatment Of Renal Calculi

Injury to the glomerulus and the tubules presents the onset of Intra-renal failure (Matzke, 2011). Some of the frequent causes for Intra-renal failure are glomerulonephritis; pyelonephritis; and tubular injury. Post-renal failure develops from things like ureteroliths, tumors, or anatomic impediments. Opposite of the acute form, the chronic form has a slow onset that has no early stage symptoms. It is important to know that following an acute episode a chronic renal episode often follows, and at this juncture the damage is irreversible. Glomerulonephritis and pyelonephritis combined, has been reported to be the forerunner in as much as half the cases from acute to chronic renal failure. Diabetes mellitus, renal vascular disease, such as atherosclerosis, hypertension, polycystic kidney disease, drug damage, and nephrolith are all examples of other causes of CKD (Pradeep, 2014). Biopsies of kidneys that suffered with CKD reveal smaller kidneys with scarring on the tubules.

     Kidney stones are hard, crystalline deposits in the kidney. They are usually hexagonal, eighty percent of which are made of calcium. These calcium stones are two to three times more common in men, and are most likely to reoccur. The calcium oxalate versions most likely result from eating specific food. One percent of stones are cystine stones, which have to do with

Laboratory Data: Hemoglobin 14.6, hematocrit 43.6, and WBC 13,000. Sodium 138, potassium 3.8, chloride 105, C02 24, BUN 10, creatinine 0.9, and glucose 102. Urinalysis was negative.

Medical History: A 57-year-old Caucasian male, with a history of end-stage renal disease, secondary to biopsy proven IgA nephropathy, history of liver cirrhosis secondary to alcohol abuse, hypertension, thrombocytopenia, leukopenia, who presented for evaluation for candidacy for a kidney transplant. The patient was evaluated at Tampa General Hospital. He went through a full medical work up. He was not listed due to lack of financial clearance per their center.

A CT of the abdomen and pelvis without contrast dated 07/27/2017 revealed no evidence of a kidney stone. There as a tiny fat-containing ventral abdominal wall hernia just to the right of the midline.

Renal insufficiency, proteinuria. The patient has seen a nephrologist in the past for a kidney lesion. I do think it would be a good idea to check in with the nephrologist, once again. She will get me his name and we will go ahead and set this up. When I did this.

This 60 year old Hispanic male presents at the clinic today with a chief complaint of urinary frequency, decreased urine flow, increased nocturia, slight terminal dysuria and low grade fever. The patient was experiencing these symptoms for the past two years, but they had increased a whole lot more during the last two weeks. Upon assessment, it is noted that the patient has a

M.T. has a 1-year history of atrial fibrillation. She takes warfarin daily, and her INR is within the therapeutic range, between 2 and 3. Her creatinine level is 1.3 mg/dL, and a normal level is 0.6-1.2 mg/dl for women. This indicates that she has slightly reduced renal function at this time, possibly due to her diabetes, medications, age, or urinary tract infection (UTI). She has been diagnosed with acute uncomplicated UTI. The 24-hour history of dysuria, urinary frequency, and urgency combined with the urinalysis result

Past Medical History: The patient has a history of end-stage renal disease secondary to IgA nephropathy, hypertension, alcohol abuse, biopsy proven liver cirrhosis, history of right leg cellulitis,

This is an 82-year-old gentleman, who was referred from Dr. Gretchen Marsh’s office because the patient’s BUN and creatinine were high, as he has acute kidney injury. The patient went to Dr. Marsh’s office yesterday and was having generalized weakness. He does not have any vomiting. No diarrhea. No fever. No cough with expectoration. No sharpness. No wheezing. No headache. No dizziness. No passing out. No rectal bleeding. No hematemesis. No abdominal pain. No sore throat. No stuffy nose. No cough with expectoration. No burning, frequency, or

blood urine from bleeding in your kidneys or bladder, blood in stool due to bleeding in

Mr. Newman is a 49 year old male who has hematuria, fever and severe flank pain. He also has bilateral lumbar tenderness, bilateral renal enlargement, liver enlargement, ankle and facial edema, skin pallor, and lung sounds suggest pulmonary edema.

On the same date an abdominal x-ray was also completed which revealed no small bowel dilation, no free gas, with a few small pelvis phleboliths (venous calcifications) noted. My involvement and care of the patient occurred Feb 22nd. On this date her lab work was: BUN 6.3, Cr 64, Na 138, K+ 3.5, Cl 107, C02 22, WBC 8.2 (15.4 Feb 15, 4.1 Feb 17), Hgb 93 (106 Feb 15, 80 Feb 16, 73 Feb 17), Hct .291, Plts 305. On assessment the client was still on contact plus isolation, but both a stool C+S and C-Diff tests were negative. Vitals: T 35.8, P 68 reg, RR 18, BP 94/52, Spo2 98% room air, Pain 0/10 (did receive 650mg acetaminophen @ 0630 for chronic Lt arm/hand pain).

Chronic Kidney Disease (CKD) is a disease that is described as a loss of kidney function gradually over time. As kidney function decreases, the waste collection in the body’s blood becomes high and makes the individual feel sick. This disease can lead to other complications in the body such as anemia, poor nutritional health, high blood pressure, and nerve damage. These complications will begin to progress and show as CKD progresses to advanced stages. Early detection of this disease is essential when it comes to treatment. If CKD is diagnosed early enough the disease progression can be slowed down and managed. This disease will eventually lead to failure of the kidneys

For the purpose of confidentiality, the patient will be identified by the initials A. S. A.S was a 52- year old African American woman who was admitted to the hospital when she started to experience severe urinary retention and shortness of breath. She has three adult children and eight grandchildren, but recently lost her husband of 25 years to diabetes. The patient appears to be very independent because she lives alone in her home and is aware of the disease process. She has a past medical history of acute renal