Dm1 Research Paper

Type 1 Diabetes effects millions of people around the world (Michel & Montada-Atin, 2014). Most often, a person is diagnosed before their 30th birthday, with diagnosis commonly being between 11 and 13 years of age (Michel & Montada-Atin, 2014). Diabetes is a disease of the pancreas. More specifically, the breakdown of β cells in the pancreas causes the production of insulin to cease (Michel & Montada-Atin, 2014). Once the body is unable to

At 1646 hours, this writer phoned (202) 727-9099 Command Information Center and spoke with Mr. Charles of the Metropolitan Police Department and requested Telephone Surveillance Unit be paged and have them to contact this writer.

Type 1 diabetes is a chronic illness characterized by the body’s inability to produce insulin. Onset most often occurs in childhood, but the disease can also develop in adults in their late 30s and early 40s. The classic symptom of type 1 diabetes is: The need to urinate frequently, excessive drinking as a result of thirst, excessive hunger and abnormally large intake of solids by mouth. Unexplained weight loss, Other symptoms may include fatigue, nausea, and blurred vision. The onset of symptomatic disease may be sudden and is a classic sign of diabetes mellitus that is under poor control or not under treatment

Type 1 diabetes, previously called juvenile diabetes, is an incurable condition that is normally diagnosed in children and young adults. Type one diabetes (T1D) is an autoimmune disease with an unknown cause. Symptoms can be subtle and if gone undiagnosed and untreated, T1D can be fatal. The treatment of T1D requires daily injections of insulin to break down excess sugar in the blood. Treatment costs of diabetes are high and the complications associated with T1D only increases those costs. Historically people with a diagnosis of type 1 diabetes (T1D) did not have long life expectancy. Today with advancements in treatment, commitment to managing insulin levels and living a healthy life style, T1D patients can lead a long an full life.

Type I Diabetes was once referred to as Juvenile Diabetes or Insulin Dependent type Diabetes Mellitus (IDDM). Type 1 diabetes, is an incurable but treatable disease which can occur at any age but is mostly found in children. Couch et al. (2008) states, “Juvenile diabetes affects about 1 in every 400-600 children and more than 13,000 are diagnosed yearly” (pg. 1). A 2014 CDC fact sheet concerning diabetes, showed that, “In adults, type 1 diabetes accounts for approximately 5% of all diagnosed cases of diabetes in the United States.” It seems that we all know someone who is affected by diabetes; either type 1 or type 2. Diabetes seems to be such a common-place illness and is much more prevalent in today’s society. However, this hasn’t always been the case. Gale (2002), states, “At the start of the 20th century, childhood diabetes was rare… (pg. 3353).” In this paper, I will provide information about Juvenile or Type I diabetes in order to educate the reader about this illness.

The signs and symptoms of DM1 or DM2 can be very different based on the individuals, even among members of the same family. Many people will not develop all of the symptoms tied to DM1 or DM2. DM1 and DM2 have many symptoms that overlap, but there are also symptoms unique to each disorder, making it easier to identify them in individuals and making them distinct disorders. DM1 can develop at birth (congenital form), during childhood (juvenile form) and during adulthood (adult form). The adult form is the most common form and usually begins in a person’s 30s. Generally, the signs and symptoms of these disorders can be seen in a person slowly over a long period of time. The onset of DM2 is typically in a persons 30’s, but anywhere between their

Type 1 diabetes, also referred to as Insulin Dependent Diabetes Mellitus (IDDM) or Juvenile Diabetes, can be caused by a genetic disorder. It can occur at any age, but it is most often diagnosed in children, adolescents, or young adults around 20 years old or before a person is 30 years of age. Insulin is a hormone produced by special cells, called the beta cells, in the pancreas, an organ located in the area behind the stomach. Insulin is needed to move blood sugar (glucose) into cells, where it is stored and later used for energy. In type 1 diabetes, these cells produce little or no insulin. Without enough insulin, glucose builds up in the bloodstream instead of going into the cells. The body is unable to

Type I diabetes mellitus (DMII) was previous referred to as juvenile diabetes, as it was known to affect mainly children and young adults. Typically individuals diagnosed with this chronic illness find out at an earlier age and treatment as well as plan of care begin and adjust, as they get older. According to the Americans Diabetes Association, only 5% of people diagnosed with diabetes mellitus have this form, compared with type II that is usually associated with obesity and older age. With type I diabetes your body or specifically, your pancreas produces very little or no insulin whatsoever, in contrast to type II diabetes when the body doesn’t produce enough insulin or may become resistant, which in turn will effect the breakdown of carbohydrates (Mayo Clinic, 2016). Our body uses organic compounds such as protein, lipids and carbohydrates to produce energy; carbs also referred to as sugar or glucose is our main source of energy amongst the three components. “Insulin is a hormone that the body needs to get glucose from the bloodstream into the cells of the body”, (American Diabetes Association, 2016). So without insulin, sugar cannot enter our cells for energy and the high levels remain in the blood, known as hyperglycemia. There is no known cause of this chronic illness, but research does believe genetics has some role in the disease process. The immune system of individuals with DMI mistakenly destroys insulin-producing cells, after a certain period of time

Diabetes mellitus (DM) is a group of metabolic diseases characterized by hyperglycemia resulting from defects in insulin secretion, insulin action or both. The chronic hyperglycemia of diabetes is associated with long term damage, dysfunction, and failure of various organs especially the eyes, kidneys, nerves, heart, and blood vessels. Type 1 DM once known as juvenile diabetes or Insulin dependent diabetes, is a chronic condition in which the pancreas produces little or no insulin. Although type 1 diabetes can develop at any age, it typically appears during childhood or adolescence (1).

Type One Diabetes (T1D) is an autoimmune disorder that is characterized by destruction of insulin-producing pancreatic beta cells. The disease results in a deficiency, and eventually, a complete lack of insulin that must be

Type 1 is also known as Juvenile Diabetes or Insulin Dependent Diabetes. The exact cause is unknown. We do know that since the body’s immune system kills viruses and bacteria, it could have mistakenly destroy cells. For sure, we know it happens when the pancreas produces little or no insulin. (Mayo Clinic Type 1 Diabetes)

Type 1 diabetes is an autoimmune disease that begins when the pancreas quits making insulin. Insulin plays a key role by letting glucose enter the body’s cells, and then uses it for energy. When the body doesn’t get the

There are different types of diabetes; however, this paper strictly focuses on Type 1 and Type 2 diabetes. Type 1 diabetes is an insulin dependent diabetes mellitus; it is often called Juvenile or onset diabetes because it is often found in children or young adults. (Readers Digest 1996 pg120) Type 1diabetes has a common effect on the destruction of the pancreatic betta cells. Over the years, there has been a connection between inheritance factors and the disease. Environmental factors, diet and lifestyle can also contribute to it as well, although diabetes can develop without a cause.

Type 1 Diabetes Mellitus is a disease that occurs when insulin-producing beta cells of the pancreas are damaged or are being destroyed (Mahan, Escott-Stump & Raymond, 2012). The gradual destruction of the beta cells of the pancreas usually leads to complete insulin deficiency, which ultimately manifests itself in the following symptoms at clinical onset: “high blood glucose (hyperglycemia), frequent urination (polyuria), excessive thirst (polydipsia), and a significant amount of weight loss, dehydration, electrolyte disturbances and ketoacidosis”, requiring dependency of exogenous insulin in order to promote optimal glycemic control in order to prevent further more severe complications; such as, ketoacidosis, microvascular diseases, macrovascular diseases, neuropathy, or worse – Death (Mahan et al., 2012). Type 1 Diabetes Mellitus can result from two existing forms: 1. Immune-mediated diabetes Mellitus, which is characterized by autoimmune destruction of the beta cells of the pancreas and an idiopathic (unknown cause) commonly seen in ethnic groups of African or Asian descent (Mahan et al., 2012). Of all diagnosed diabetes cases, prevalence of Type 1 Diabetes Mellitus accounts for 5%-10%, and although onset may present itself at any age, 70% of cases are diagnosed in young adults (younger than age 30), with a peak in occurring incidences seen mostly in children and adolescents (Mahan et al., 2012). “In the United States approximately 1 in every 400 to 500

Type 1 DM is caused by autoimmune disorder where the T cell mediated disease damage the pancreatic beta cell (Huether & McCance, 2012). At the onset of the disease process, lymphocytic infiltrates of macrophage activating CD4+ cells with cytokine secreting, cytotoxic CD8+cells surround the necrotic B cells (McPhee & Hammer, 2010) (p.507). This is a gradual process that happens over several years. However, the production of glucagon increases due to the damage to the beta cells of islet