Effective Dental Health And Prevention Of Gum Disease, Gingivitis, Bacterial Carries, Cavities, And Periodontal Disease

     Periodontal disease is more commonly known as gum disease or gingivitis. This infection is serious enough, that it can lead to tooth loss if left untreated. This chronic infection starts around the tooth and it affects the supporting bone and gums. Periodontal disease can affect anywhere from one tooth to all thirty-two teeth. The disease pathology starts with the plaque that builds up on your teeth everyday.

Also commonly referred to as Gingivitis, gum disease is a contagious bacterial infection that not only affects your mouth ecology, but that can have pronounced effects on your overall health. Locally, it can be responsible for eroding and destroying your gums, and in more advanced stages it can lead to tooth loss and even jawbone erosion. Still, left untreated, the inflammation from the bacterial infection can even lead to a variety of ailments ranging from increased allergies to cancer.

The most important cariogenic bacteria are Streptococcus mutans. Other cariogenic bacteria include S. salivarius and S. sanguis as well as various species of Eikenellan and Lactobacillus. Dental caries is a disease of the tooth enamel and the tooth itself, while periodontal disease is a combination of gum swelling and erosion of the ligaments and bone that supports the teeth. Mildest form of periodontal disease is gingivitis, while the most severe form is called acute necrotizing ulcerative gingivitis also known as trench mouth. Poor oral hygiene, unhealthy diet, alcohol and tobacco use may constitute in the development of dental caries and periodontal

Gingivitis is an inflammation of the gum tissues surrounding the teeth. If left untreated, gingivitis can lead to periodontitis, an advanced gum disease. Gingivitis can sometimes be painless and therefore people may be unaware they have

Through research, the bacteria has been found to suppress apoptosis and also reduce the number of tumor suppressors within gingival epithelial cells- encouraging uncontrolled cell division. P. gingivalis is also able to induce gingival epithelial cells to express receptors found in cancerous cells and chronic inflammatory cells. Some proteins produced by the bacteria can have the ability to activate proenzymes that degrade basement membrane near the lymph nodes- potentially causing cancer cell growth to spread to the blood stream and to remote areas.

Gingival herpesvirus infection may promote subgingival attachment and colonization of periodontopathic bacteria similar to the enhanced bacterial adherence to virus-infected cells observed in other infections. Loss of virus-damaged epithelial cells may expose the basement membrane and the

Bacterial plaque that forms on your teeth and around your gums is the culprit that causes periodontal disease. When plaque accumulates on your teeth and around your gums, they soon become inflamed, swollen, and bleed

Gum disease or periodontal disease is caused by a sticky film of bacteria that has formed in tiny pockets around your teeth. The sticky bacteria called plaque. Plaque causes the tissues that support your teeth get infected. Gum disease can cause the loss of teeth, tissue, and bone. Chronic periodontitis affects 47.2% of adults over 30 in the United States” (Gum Disease, MouthHealthy.Org). However, according to the book, A Wellness Way of Life by Robbins, Powers, and Burgess; “Gum disease can increase inflammation and can be prevented by practicing good oral health (i.e. flossing every day and brushing your teeth for two minutes twice a day)” (A Wellness Way of Life,

Yes, previously, it was believed that bacteria played a role in linking gum disease to other diseases; however, research indicates that inflammation may be the contributing factor. Consequently, addressing inflammation may accomplish much more than just helping with the management of gum disease, it may also assist with managing other chronic conditions that cause inflammation.

This stage is an inflammatory response due to the reaction to plaque, which does not show obvious gingival tissue changes in the mouth (Perry, Beemsterboer, & Essex, 2014, p. 71). Stage II gingivitis or early stage shows multiple lesions begin to form between four to seven days after the growth of plaque in the gingival sulcus. During this stage, the gingival tissues appear to be enlarged and red. The patient will lightly to experience bleeding while brushing, flossing, and bleeding on probing (Perry, Beemsterboer, & Essex, 2014, p. 71). Stage III gingivitis or established stage occurs after 15 to 21 days after plaque accumulation. The established lesions from stage II may progress or remain unchanged, which can be reversible once the induce-plaque is removed (Perry, Beemsterboer, & Essex, 2014, p. 71-72). The last stage is stage IV gingivitis or advanced stage, occurs when the inflammation

Gingivitis and periodontitis are among the most common infectious diseases in human beings which results in bone loss, tooth loosening and eventually tooth loss. Although periodontopathic bacteria are the primary etiological agents in periodontal disease, the ultimate determinant of disease progression and clinical outcome is the host's immune response(1). Lymphocyte blastogenesis studies of peripheral blood from patients with periodontal disease also suggested the involvement of cell-mediated immunity(2). The inflammatory response in children is different from that of adults. The most prevalent type of gingival disease in childhood is chronic marginal gingivitis which is characterized by an infiltrate consisting mostly of T lymphocytes and

Next, an article in Medline Plus states that gum disease or periodontal disease is a serious infection and inflammation of the gum.

Chronic periodontitis is common disease of the oral cavity, that can occur over a wide range of age. The major determinant of disease susceptibility is the host immune-inflammatory response to the sub-gingival biofilm. It is paradoxical that these defensive processes result in the majority of the tissue damage leading to the clinical manifestations of disease. The unique anatomy of the periodontium

Periodontitis is a chronic inflammatory disease process which is initiated by bacterial challenge and characterized by destruction of tooth supporting tissues. It is the most prevalent form of bone disease in humans and a modifying factor of systemic health of patients 1. However recent evidence suggests that mere presence of putative periodontal pathogens is not sufficient for initiation of disease process 2. It is rather persistent host inflammatory response against those pathogens that leads to periodontal tissue destruction 2.

This hypothesis links inflammatory periodontal disease to CVD, such as atherosclerosis, without necessarily invoking bacteria or their products interacting with the cardiovascular system (38).