Ev Packaging Essay

Packaging involves designing and producing the tin or bottle for a product. Packaging contains two levels. The primary packaging is the container for each individual product. For instance, A&W uses bottle with variety of sizes to fill the root beers. Plastic bottle can prevent leakage problem and can keep it once couldn’t finish it at once. The secondary packaging contains the quality of the product. A&W uses different colours and designs to wrap the bottle to differentiate the function of products. For example, white is for “Diet Root Beer with Cream Soda” and golden is for “Root Beer with Cream Soda”. The difference of packaging designs conveys the characteristics of the brand. Besides, this packaging can provide correct and useful information

Cancer, medically called ‘tumorigenesis’ (Thaker, Lutgendorf, & Sood, 2007, p.430) occurs when cells in the body orient themselves for malignant growth. Such cells show ‘self-sufficiency in growth signals’, are ‘insensitive to anti-growth signals’ and have ‘limitless replicative potential’ (Thaker, Lutgendorf, & Sood, 2007, p.430). Once a particular set of cells become malignant, the malignancy can spread to other set of cells in different organs due to ‘crosstalk’ between the affected cells and their surrounding ‘tissues’ and ‘micro-environments’(Thaker, Lutgendorf, & Sood, 2007, p.430).

I reviewed your memorandum to Governor Jerry Brown, outlining a “Plan to Increase Demand for Electric Vehicles.” Your message to the California government is clear about its intent. You offer solid arguments. You urge the government to develop electric vehicles to improve air quality in California, and clearly identify the burden to develop and market the electric car within the private sector. To improve your paper, you could strengthen your arguments with more concrete support, develop more seamless transitions, and include associated counter-arguments.

disease in which abnormal cells divide without control and can invade tissues nearby. Cancer can also spread to other parts of the body through the blood and lymph systems. The National cancer Institute and the World Health Organization (WHO) report that in 2012, there were 14 million new cancer cases and 8.2 million cancer related deaths. The number of new cases is expected to rise by about 70% over the next two decades. WHO also reports that in 2012 the most common cause of cancer death was lung cancer with 1.59 million deaths. The two main types of lung cancer are Small cell lung cancer (SCLC) and Non-small cell lung cancer (NSCLC). NSCLC makes up about 85% to 90% of lung cancers. Treatments for NSCLC can include surgery, radiation therapy, chemotherapy, immunotherapy and targeted therapies. One example of targeted therapy is angiogenesis therapy. Angiogenesis is the formation of blood vessels. Tumors need a blood supply to grow beyond a certain size and will give off a chemical signals to stimulate angiogenesis. One method of preventing this is to find ways to block tumor angiogenesis. This can be done through angiogenesis therapy, which is also called antiangiogenic agents. Angiogenesis is unique in fighting cancer because it focuses on blood vessels rather than tumor cells. Because angiogenesis is unique, arguments can be made for and against this type of treatment. This goal of this paper is to provide pros and cons for angiogenesis therapy.

Both of these events occur because of interactions between the cancer cells and the stromal microenvironment (Weinberg et al., 2014). The degradation of extracellular matrix (ECM) and regulation of cell mesenchymal integrity are require steps (Bonnomet et al., 2010; Woessner, 1991). As known that matric metalloproteins (MMPs) are play an important role on ECM degradation. Type IV collagenase and gelatinase (MMP-2 or MMP-9) are major proteases for ECM degradation leading to migration and intravasation (Martin et al., 2007). Several junction proteins, such as E-cadherin and Vascular-Endothelial cadherin (VE-cadherin), are regular the integrity of cell-cell content and cell-mesenchymal contact (Fleming et al., 2000; Micalizzi et al., 2010). During cancer cells leave the original tumor organ to migrate to the target metastasis organ process, adhesion molecules (e.g., integrin-1 and E-selectin) play a key role in regulating the adhesion of tumor cells to endothelium cells (Yates et al., 2014; Okegawa et al., 2002). On the other hand, when cancer cells leave the original tumor organ to migrate to circulate system by intravasation, then they extravasation into the circulatory system to migrate to the target metastasis organ. The regulation of migration, invasion and adhesion may be an effective strategy for improving the prognosis of

Well, fucoidan has been found to also have a role in this. It stops a tumor cell from being able to anchor to any surface in the body and without being able to anchor, there is no growing it can do. These are just some of the ways in which this compound is amazing, but science is still working out what else it can do and how it can be worked into treatments.

Distant spread of primary malignant cells to local and distant sites was explained by several models. Cumulative research shows a pivotal role of tumor microenvironment in the process of malignant cell dissemination. This role occurs through angiogenesis and epithelial-stromal interactions, via the paracrine secretion of growth factors by stromal cells, which induce an epithelial-mesenchymal transition in neighboring epithelial cells. This result in the increased migration of epithelial cells [3].

Angiogenesis, the growth of new blood vessels, provides a tumor with the nutrients it needs to grow and spread to new tissues in the body. In clear-cell renal carcinoma a mutation in the tumor suppressor gene VHL leads to deregulation of vascular endothelial growth factor (VEGF). This action enables angiogenesis and leads to disease progression. Bevacizumab is a humanized monoclonal antibody that acts by binding to VEGF in the blood. This prevents VEGF from binding to

The research in Dr. Langenfeld’s lab focuses on the genes that regulate lung cancer. The experiment looks closely at the non-small cell lung carcinoma (NSCLC) and immortalized bronchial epithelial cells. Careful study of the carcinoma and epithelial cells has revealed that the mRNA of bone morphogenetic proteins 2 and 4 (BMP-2/4) was highly expressed in the carcinoma. Past studies have shown that BMP-2/4 have certain properties that allow them to activate the differentiation, growth, and migration of cancerous cells in the embryo. After studying these morphogens in lung carcinoma is was revealed that the BMP-2, in its mature state, is significantly more expressed in NSCLC than BMP-4 in cancerous lung tissue, but did not show much effect in normal lung tissue or benign lung tumors. The BMP-2 that was exposed in vitro to the A549 and H7249 human lung cancer lines stimulated significant migration and invasiveness. In vivo trials showed that the growth of tumors of A549 cells in nude mice was highly enhanced. Tumor growth in NSCLC was attempted to be reduced with recombinant through the exposure to noggin or the anti-BMP-2 antibody. Results showed a significant reduction in the tumor growth[1].

There are many different types of illnesses and diseases that are culprits for taking the lives of people worldwide. Of the different diseases, cancer is one of the most frequent causes of death. Cancer is defined as unregulated growth of cells that deviate from usual cellular activities. This includes the development of tumors and eventually the invasion of other tissues. There are different stages of cancer (0-4) that mark its progression. Depending on the state of the tumor, the types of treatments will vary, which determines its curability as well. During the initial stage (0), it is a state where the tumor cells are localized in one area and remain static. At this stage of cancer, the

Cancer is one of the leading causes of death worldwide as it can develop in almost any organ or tissue. Significant advances in understanding the cellular basis of cancer and the underlying biological mechanisms of tumour has been vastly improved in the recent years (Jiang et al. 1994). Cancer is a genetic disease which requires a series of mutation during mitosis to develop, its characteristics can be associated with their ability to grow and divide abnormal cells uncontrollable while in the mean time invade and cause nearby blood vessels to serve its need. Even though many people are affected by cancer today, the abilities which cancer cells have make it hard to find a single effective treatment for cancer. The focus of research now lies

These days tumours are an exceptionally disputed issue, a lot of individuals have diseases involving tumours. Here I want to talk about the different sorts of medicines used to decrease growth and which is the best medicine among them. First and foremost I want to present some fundamental information about cancer. A tumour is an ailment of the cells, which are the body's essential building pieces, cancer happens when unusual cells develop in an uncontrolled way (MedicineNet.com 2014). These unusual cells can harm or attack the encompassing tissues, or spread to different parts of the body, bringing on additional harm. The growth of these cells, in some cases, starts in one piece of the body before spreading to different areas. This

Due to their role in preventing apoptosis in cancer cells, anti-apoptotic proteins BCL-2 and BCL-XL were investigated as potential drug targets: it was proposed that inhibiting these anti-apoptotic proteins could promote cancer cell death.

Also, mutations in apoptosis complex or suicide that is activated by p53, a tumor suppressing protein, and this results in the complete disabling of replication and, thus, there is prevention of cell destruction when the alarms responsible for self-destruction are set off. The above factors, when combined, lead to the unlimited cell proliferation and this unchecked cell division and malignant tumors release chemicals that promote the growth of abnormal capillaries leading to the feeding of these capillaries with the critical nutrients and this leads to the destruction of the surrounding cells. Some cells even detach themselves from the cell mass and then travel through to the bloodstream and then form tumors in some other body parts through metastasis.

Packaging is an important aspect in marketing and transcends all products regardless of size, market and other aspects in marketing. While packaging in literal terms refers to the technology used in enclosing and protecting products during distribution, sales or storage, the meaning goes beyond this when dealing with products such as vehicles. Considering that buyers make purchase decisions on what they see, packing plays a significant role in such decisions. However, in motor vehicles, packing comes in a different perspective because unlike a smart phone, a car cannot be packed but is displayed in areas such as show rooms. This does not mean vehicles do not get ‘packaged’ because it comes in the form of attractive and functional features.