Exophthalmic Goiter

Graves ' disease is an immune system disorder that results in the overproduction of thyroid hormones (hyperthyroidism). The thyroid gland is an important organ of the endocrine system. The gland is located at the front of the neck above where the collarbones meet. This gland releases the hormones thyroxine (T4) and triiodothyronine (T3), which control body metabolism. Controlling metabolism is important for regulating mood, weight, and mental and physical energy levels. When the body makes too much thyroid hormone, the condition is called hyperthyroidism. Graves disease is the most common cause of hyperthyroidism and is most common in women over age 20. But the disorder can occur at any age and can affect men as well. Some of the symptoms of Graves diseases are

There are many signs and symptoms of Graves’ disease. Symptoms of Graves’ disease include anxiety,

3. As hypothyroidism progresses, symptoms will start to occur including fatigue, weight gain, cold intolerance, peripheral neuropathy, memory loss, sleep apnea, and daytime drowsiness.

Graves’ disease is autoimmune that effects the thyroid in front of the neck. This thyroid creates T3 and T4 which helps regulate how the body uses energy. We have a small gland called pituitary in the brain that helps the thyroid regulate the hormone level properly. With grave’s disease the immune system creates antibodies that act like TSH (thyroid stimulating hormone) which than makes the thyroid to create more hormones than what the body needs to have. This is called hyperthyroidism. Graves’ disease causes the body to speed up such as the heart and the metabolism. The symptoms for graves’ disease are goiter (enlarged gland) Anxiety and irritability, A fine tremor of your hands or fingers, Heat sensitivity and an increase in perspiration

Eye trouble -- usually in the form of inflamed and swollen eye muscles and tissues that can cause the eyeballs to protrude from their sockets -- is a distinguishing complication of Graves' disease. However, only a small percentage of all Graves' patients will experience

A radioactive scan may be done to see whether the entire thyroid gland is overactive. In this scan, radioactive iodine is administered to the patient which then may demonstrate diffuse enlargement and increased uptake of radioiodine. The extent of this uptake helps determine the dose of radioactive iodine necessary for treatment. Ultrasound may also be utilized to help determine the size and location of the affected thyroid gland, which can then be paired with the radioactive

The first (1st) is by prescribing antithyroid medications which help the thyroid from producing too much of the thyroid hormone. The second (2nd) treatment that is recommended is radioactive iodine. This treatment is in pill form that damages the thyroid by giving it radiation. Even in this case, you will most likely end up having to take thyroid medications for the rest of your life. The third (3rd) and last treatment is surgery. This is when they completely remove all of your thyroid which cures the overactive thyroid but in addition from having it removed, you have to take a thyroid hormone to replace what has been removed. Aside from these treatment plans, your doctor may prescribe you a drug called beta-blocker which slows down your heart rate and reduces symptoms of shaking and nervousness. This drug works quickly and helps make you feel better while it takes

Primary causes include iodine deficiency and the autoimmune disease named after Hashimoto (NIDDK, 2013). As stated earlier iodine deficiency occurs in countries that do not have iodine in their water supply. Hashimoto’s disease occurs when there is a problem with the immune system that causes white blood cells to accumulate on the thyroid gland that results in loss of thyroid function (NIDDK, 2013). Other causes of primary hypothyroidism include: thyroidectomy, thyroid ablation, inflammation of thyroid gland, not enough medication and some medications (NIDDK, 2013). Causes of secondary hypothyroidism include: pituitary cancer, Sheehan syndrome, injuries of the brain, brain cancer, radiation, administration of growth hormones, and brain hemorrhages (NIDDK, 2013). The numerous symptoms of hypothyroidism include: weight gain, decreased activity, fatigue, constipation, intolerance of cold conditions, pain in the muscles and joints, dry, course hair with or without hair loss, depression, problems with memory, bradycardia, problems in women with menstruation (NIDDK, 2013). People may experience any of these symptoms. People that have hypothyroidism for prolonged period of time have symptoms that may include: loss of taste, hoarse voice, edema, slow speech, thin eyebrows and thick skin (NIDDK, 2013). Diagnosis of this disease is not always easy because these

The reason I pick this disease is this is what my grandmother pass away from and is not in a better place. Graves disease which was first described by Robert Graves in the 19th century, this disease if one of the most common thyroid problems.It is also the leading cause of hyperthyroidism, a condition in which the thyroid gland produces excessive hormones. Once this disorder is diagnosed it is east to treat, in some cases the disease has gone into remission or disappeared completely after some months or years. if left untreated this disease can lead to many serious complications

According to the American Thyroid Association hyperthyroidism is an condition in which there is excessive amount of thyroid hormone in the body. It is also stated that another term for this problem is called Grave’s disease or thyrotoxicosis, and thyrotoxicosis is when there is high levels of thyroid hormones in the blood stream. The thyroid is a small shaped butter-fly gland located in the front of the neck and that is apart of the immune system. The authors of this article talks about who is the most affected, diagnoses, and treatment of this disease.

There are many causes of hyperthyroidism, but the symptoms of hyperthyroidism are the same no matter what causes the over expression of thyroid hormones. The main symptoms of hyperthyroidism are fatigue, shortness of breath, weak muscles, anxiety, difficulty sleeping and heart palpitations. Interestingly, even with an increase in appetite the patient will have weight loss.

When it goes untreated, there are physical signs and symptoms that may develop such as an enlarged thyroid gland, mild to severe eye problems, and even skin lesions with a thickening of the skin of the tibia. Some of the early symptoms of Graves’ disease are weight loss, anxiety, chest pain, muscle weakness, shortness of breath, and irregular menstrual periods. During the early stages, not everyone will experience symptoms; while others will experience many of them.

Graves’ disease has a wide range of symptoms and signs that includes weight loss, but an increase in appetite, thirst, frequent bowel movement even diarrhea. According to the book I read other symptoms are progressive nervousness, tremors, palpitations, fatigue, heat intolerance, difficulty concentrating, and muscle weakness even sudden paralysis. Patients have also shown increases in blood pressure, systolic pressure increase higher than the

Hyperthyroidism , part of the endocrine system, is a the gland that makes hormones that controls the way every cell in the body uses energy. Your thyroid is located in the front of your neck where the collarbones meet. Having this disease can affect some vital signs. Having hyperthyroidism can cause heart palpitations and high blood pressure. Therefore, because of having this disease, blood pressure and pulse need to be checked and documented every visit. It is important to collect vital signs and note them because it could help diagnose a patient.

Grave’s disease (GD) is rare in childhood (1); however, it is the most common cause of hyperthyroidism in children and adolescents (2). Graves’s orbitopathy (GO) is the most common extrathyroid manifestation of GD but it can be present in 5 to 10% of euthyroid patients (3). Usually, both eyes are equally affected, although 9% of patients may present an asymmetric form (4). It can develop before, during or after the onset of hyperthyroidism (5), but presents a wide range of atypical presentations that reveal a pathogenesis still enigmatic. The evidence indicates that anti-TSHR antibodies are probably responsible for the genesis of the GO, and there are some polymorphisms that show a high enough risk of GO suggesting a complex genetic basis (6).