Explain The Conclusions Gained From The Investigation

Introduction: In this experiment, cardiovascular fitness is being determined by measuring how long it takes for the test subjects' to return to their resting heart rate. Cardiovascular fitness is the ability to "transport and use oxygen while exercising" (Dale 2015). Cardiovascular fitness utilizes the "heart, lungs, muscles, and blood working together" while exercising (Dale 2015). It is also how well your body can last during moderate to high intensity cardio for long periods of time (Waehner 2016). The hypothesis is that people who exercise for three or more days will return to their resting heart rate much faster than people who only exercise for less than three days.

Two intervals did show a significant shorting post exercise. Besides the RR interval, the other recorded interval which contained a marked shortening after exercise was the TP interval. To understand why the TP interval is shortened when the heart rate increases and lengthened when the heart rate slows, one must first understand what the TP interval is. The TP interval, which is the the interval between the end of the

Heart rate anticipatory response – this is where the heart rate starts to automatically increase before you start to exercise. The heart rate is able to increase automatically by chemical hormones, the hormones are adrenaline and noradrenaline. These hormones are found inside the brain. The reason the heart rate increase before exercise is because it prepares the muscles for exercise, the reason it prepares the muscles for exercise is because by the heart rate increase the more oxygen is getting to the muscles there fore they will not be needing a such a large oxygen supply all at once. It doesn’t only supply oxygen it supply’s nutrients, the supply of nutrients also provides energy and helps to repair the muscles after exercise. By the heart rate starting to increase gives the heart a head to start pumping hard this enables the heart to not have as much stress on it.

This experiment was carried out as noted about in Procedure 1. The resting heart rate was established and used as a baseline value from which to compare all future deviations. While data could

Exercise increases heart rate by a process of sympathetic autonomic stimulation. Sympathetic (adrenergic) nerves increase the excitability of the sino-atrial node and reduce the P-R interval .As exercise continues, the physiological changes in the body are continuously monitored by a number of physiological systems and the balance of activity of the sympathetic system (speeding up) and the parasympathetic system (slowing down) is constantly adjusted. When exercise is over, the heart rate does not drop immediately as the body has to undergo a period of re adaption to return to the resting state.

The authors then share their analysis on the changes of heart rate and GSR at the many different points throughout their study.

In the first experiment I was subject 2. For the most part my heart rate did not oscillate too much. My heart rate was a little slower than the other subjects as well. Compared to Activity 2, his oscillations while exercising were a little more dramatic. The reason for this could be that your heart is trying to adjust to the new physically taxing activity that you are enduring. Your body is trying to balance, but if it is thrown into a demanding activity it will take a little bit more adjusting than if you are just resting.

The effects of heart rate on differing durations of exercise were studied in this experiment. For people, heart rate tends to increase as they perform physical exercises. The amount of beats per minute gradually increases as people perform physical activities. Heart rates taken before exercise are relatively low, and heart rates taken one minute after exercise increase significantly. Heart rates slowly begin to decrease after they are taken two minutes and three minutes after performing the step test, which is to be expected. The rates of intensity throughout exercise relates with changes in heart rate throughout the step test performed in the experiment (Karvonen 2012). The age of the participants affected the experiment, since the heart rate during physical exercise, in this case the step test, is affected by age (Tulppo 1998).

The independent variable of my experience meant is what I’m going to be changing which is the substance, coffee. The dependent variable of my experiment is the Daphnia’s heartbeat per minute, which will also increase immensely. The units I will be using to collect the data is one minute per trial.

The controlled variable included the exercise bike and heart rate monitor. There are several limitations, systematic and random errors that should be considered when interpreting these results. (4) The controlled variables were not tested before this experiment to see if they were working and reliable. Figure 2 heart rate was quite inconsistent and did not follow the pattern of the other results, which maybe suggest a random error with the heat rate monitor. A systematic error could include the fitness of the participants. One of the test subjects is an endurance athlete and the other does not compete in any sport. This would affect the results because for the endurance-trained athlete, from their training they increase their cardiac output results from a substantial increase in maximal stroke volume. In untrained persons, cardiac output increases in response to exercise primarily by an increase in heart rate. The endurance-trained athlete does so mainly by an increase in stroke volume. Simply meaning that although both participants are doing the same cadence and length the endurance athletes skewers the results by already having an increased rate in stroke volume. Another systematic error may include the rate of perceived effort. For the most accurate results, the measured maximum heart rate would be necessary to give an accurate cadence to ride at.

Aims - This study is to ascertain, if there is an effect on heart rate after exercise. This is being done to see, if there is a difference between resting heart rate and heart rate after performing exercise.

The effects of exercise on blood pressure, heart rate, respiration rate and electrical activity of the heart were assessed. The measurements of respiration rate, pulse rate and blood pressures were noted as described in Harris-Haller (2016). Data was first taken from subjects in a relaxed position and then followed by sets of reading after exercising based on one minute intervals. The data also noted sitting ECG traces from Harris-Haller (2016). The respiratory rate, pulse, blood pressure, P wave, QRS complex and T wave were defined for each subject. The class average was calculated for males and females and graphed to illustrate the results by gender for each cardiopulmonary factor.

As the intensity of exercise increased, so did the rates of the heart and breathing. After a small period of rest, the heart rate and breathing rate both decreased to a point close to their resting rate. This proved the stated hypothesis. First, the hearts average resting rate was recorded to be 76 bpm. The heart is therefore transporting oxygen and removing carbon dioxide at a reasonably steady rate via the blood. During the low intensity exercise (Slow 20) the heart rate increases to 107 bpm, which further increases to 130bpm at a higher intensity level (Fast 20). The heart therefore needs to beat faster to increase the speed at which oxygen is carried to the cells and the rate at which carbon dioxide is taken away by the blood.

The literature on the effects of exercise of cardiac output maintains the idea that exercise should affect cardiac output- pulse rate, systolic blood pressure, diastolic blood pressure, QRS-pulse lag, P-T and T-P intervals, because of increased heart rate. For our experiment, we tested this theory by measuring our cardiac output before and after some rigorous exercise. We measured the individual cardiac output and then combined the data to compose a class-wide data average. We compared the results of the experiment to what we expected, which was that exercise does affect our heart. Our data from this experiment supported the notion that exercise does, in fact, change cardiac output.

I predict that during exercise the heart and respiratory rate (RR) will increase depending on the intensity of exercise and the resting rates will be restored soon after exercise has stopped. I believe that the changes are caused by the increased need for oxygen and energy in muscles as they have to contract faster during exercise. When the exercise is finished the heart and ventilation rates will gradually decrease back to the resting rates as the muscles’ need for oxygen and energy will be smaller than during exercise.