Extreme Pulmonary Fibrosis: A Case Study

D.Z., a 65-year-old man, is admitted to a medical floor for exacerbation of his chronic obstructive pulmonary disease (COPD; emphysema). He has a past medical history of hypertension, which has been well controlled by Enalapril (Vasotec) for the past 6 years. He has had pneumonia yearly for the past 3 years, and has been a 2-pack-a-day smoker for 38 years. He appears as a cachectic man who is experiencing difficulty breathing at rest. He reports cough productive of thick yellow-green sputum. D.Z. seems irritable and anxious; he complains of sleeping poorly and states that lately feels tired most of the time. His vital signs (VS) are 162/84, 124, 36, 102 F, SaO2 88%. His admitting diagnosis is an acute

The diagnosis is hemoptysis due to lesion on the lung. This is coded as 239.1

As a result of emphysema there is a significant loss of alveolar attachments, which contributes to peripheral airway collapse. There are two major types of emphysema according to the distribution within the acinus and they are; (i) centrolobular emphysema which involves dilatation and destruction of the respiratory bronchioles; and (ii) panlobular emphysema which involves destruction of the whole of the acinus. According to theory, centrolobular is the most common type of emphysema in COPD and is more prominent in the upper zones, while panlobular predominates in patients with alpha-1 antitrypsin deficiency and is more prominent in the lower zones. In relation to patients D.Z. with emphysema, the walls between the tiny air sacs in the lungs are damaged due to long-term cigarette smoking effect on his lungs as evidenced by patient c/o difficulty breathing at rest and productive cough with thick yellow-green sputum r/t a

A female patient 83 years of age has been diagnosed with Emphysema and has had this disease for 10 years. She has been in good shape her whole life and has been big into health and fitness. However, the critical etiological risk factor was that she participated in tobacco smoking for a brief period of her 20’s. She was informed of the disease by visiting a general practioner and therefore getting referred to a respiratory specialist. The laboratory tests used to establish her diagnosis included a lung function test, X ray and sleep apnoea test in hospital overnight. Her most common symptoms include shortness of

R.W. appears with progressive difficulty getting his breath while doing simple tasks, and also having difficulty doing any manual work, complains of a cough, fatigue, and weight loss, and has been treated for three respiratory infections a year for the past 3 years. On physical examination, CNP notice clubbing of his fingers, use accessory muscles for respiration, wheezing in the lungs, and hyperresonance on percussion of the lungs, and also pulmonary function studies show an FEV1 of 58%. These all symptoms and history represented here most strongly indicate the probability of chronic obstructive pulmonary disease (COPD). COPD is a respiratory disease categorized by chronic airway inflammation, a decrease in lung function over time, and gradual damage in quality of life (Booker, 2014).

Emphysema is the most common cause of death from respiratory disease in the United States and is generally caused by several years of heavy cigarette smoking (Olendorf, 2000). When a person smokes, the body’s immune system tries to fight off the invading smoke by using certain substances. These substances can also attack the cells of the lungs, but normally the body is able to release other substances to prevent this. In the case of people who are smokers, this doesn’t happen and the original substances that were released to fight off the smoke also end up injuring the cells of the lungs as well. Eventually, the lungs will not be able to supply enough oxygen to the blood and a host of problems can occur with this. Risk factors that have been identified for emphysema include exposure to tobacco smoke either through active or passive smoking (2nd hand smoke), occupational exposure such as dust or chemicals, ambient air pollution, or genetic abnormalities, including a deficiency of alpha-antitrypsin, an enzyme inhibitor that normally counteracts the destruction of lung tissue by certain other enzymes (Smeltzer, 2010). The symptoms of emphysema develop gradually over many years. It is generally characterized by three primary symptoms: chronic cough, sputum production, and dyspnea on exertion. Other signs and symptoms include weight loss and the development of a

Data: Pulmonary function testing dated 2010 showed moderately severe obstruction with positive bronchodilators response. Normal lung volumes. Evidence of air trapping. Severely reduced diffusing capacity for carbon monoxide.

Based on the examination of the patient, a secondary pathology resulting from primary pathology is evident. The primary pathology is Lung Cancer and the secondary pathology is Superior Vena Cava Syndrome.

his neurologic intensive care unit (NICU) stay, Y.W. was intubated and placed on mechanical ventilation, had a feeding tube inserted and was placed on tube feedings, had a Foley catheter to down drain (DD), and had multiple IVs inserted. He developed pneumonia 1 month after admission.

Emphysema is a condition of the lungs that involves the over-inflation of the air sacs, otherwise known as the alveoli. Normal functioning of the alveoli helps breathing in normal respiration. Unlike other lung diseases this one can be inherited. This is a disease that can affect people of all ages and genders. In 1994, the number of males with emphysema outnumbered the females by fifty four percent. However, in the two years that followed the difference between males and females decreased by ten percent.

Collard, H. R., Ward, A. J., Lanes, S., Hayflinger, D. C., Rosenberg, D. M., & Hunsche E., Burden of illness in idiopathic pulmonary fibrosis, (2012).

TREATMENT FOR EMPHYSEMA Doctors can help persons with emphysema live more comfortably with their disease. The goal of treatment is to provide relief of symptoms and prevent progression of the disease with a minimum of side effects. The doctor's

Eventually, you’d welcome death… but an emphysema sufferer can go on living like this for years.

When a non-smoker gets pulmonary fibrosis, it’s generally considered idiopathic pulmonary fibrosis, a form with no known cause. Pulmonary fibrosis is a progressive disease that varies in the rate of degeneration from person to person. Scarring of the lungs is irreversible.

The simple act of breathing is often taken for granted. As an automated function sustaining life, most of us do not have to think about the act of breathing. However, for many others, respiratory diseases make this simple act thought consuming. Emphysema is one such disease taking away the ease, but instead inflicting labored breathing and a hope for a cure.