Gastroscopy: A Case Study Of Peptic Ulcers

ulcer causes the patient to suffer with pain, disfigurement, and prevents them from their daily

During a colonoscopy, your doctor will be looking for the telltale colon ulcerations inside of your colon that signal a case of ulcerative colitis. There are none of these ulcerations in IBS sufferers, and in Crohn's disease, many other parts of the digestive tract can be affected. This means that if your doctor finds ulcerations in only your colon, then a diagnosis of ulcerative colitis may be made.

With regards to the peptic ulcer disease, the most common cause is H. pylori infection, thus, testing for it is essential in all patients with peptic ulcers (Anand, 2015). Upper GI endoscopy is the preferred diagnostic test in evaluating patients with symptoms of peptic ulcer disease. Non-invasive methods of detecting H.pylori infection includes fecal antigen testing, which identifies active H. pylori antigens in the stool, and urea breath tests.

Peptic ulcer causes sores to the stomach and the beginning of the small intestines due off acid (hyperacidity) or bacteria. Also certain pain medications can be a trigger. Symptoms may include pain in the upper abdomen, gas, indigestion, heartburn, and vomiting. Treatment for a peptic ulcer includes certain medication, antibiotic for bacteria, and change of diet.

Though Ulcerative Colitis is not a disease that can cause certain death, but it can lead to the creation of many other which have high mortality rates. Such as, strokes and colon cancer. We know that there is bleeding when Ulcerative Colitis occurs. So, during blood clotting, increased amounts of blood clots also form in the brain causes strokes. Again, the breaking of the mucus layer in the colon can give rise to cancerous lesions, which develops to colon cancer later

The most symptoms of ulcerative colitis are abdominal pain and bloody diarrhea mixed with pus, blood and mucus. Individuals may become very sick and weak, they can suffer form anemia, weight loss, loss of appetite, rectal bleeding, loss of body fluids and nutrients, skin lesion, joint pain, and growth failure specially in children according to the extent of the disease. Just how risk factors have a different affects on everyone symptoms are also different. A quarter of the patients only have the disease only have mild symptoms while others suffer from fevers, bloody diarrhea, nausea and severe abdominal cramps. Complications may also occur outside the colon such as arthritis, inflammation of the

Eric, I think your presentation is really clear and informative. I like the choices you made on the font type and color, it popped with the images that you used. I think that your points are well stated and easy to read and understand. The only thing that I can see that you might want to consider is adding more frames so that you could include even more information on the topic. I think that you did a nice job with the Prezi presentation, I was very worried about using Prezi myself, as it is completely new to

PUD is not as common in pediatric patients when compared to adult incidences. The most common cause of PUD in pediatrics is due to Helicobacter pylori (H.pylori) infection (Guariso & Gasparetto, 2012). Primitive ulcers, due to H. pylori, cause changes in the gastric or duodenal mucosa and alter gastric function such as overproduction of HCl and pepsin, resulting in ulcer-forming lesions (Guariso & Gasparetto, 2012). NSAIDs, stress and other extra-gastric pathogenic events cause secondary ulcers within the stomach. The underlying pathology of PUD is due to the reduction of prostaglandins that serve as a protective mechanism on the gastric mucosa. Patients most commonly present with dull stomach ache or pain usually after a meal. Gas, bloating, nausea and vomiting are some less common symptoms. PUD, although rare when compared to adults, is reported worldwide in children and most commonly identified as initiated by H. pylori infection and the use of NSAIDs occurring in the second decade of life. Other events such as stress, shock, or major trauma can trigger acute ulcers in children but there is a hereditary family history component that increases acid output and accelerates gastric emptying in more than 20% of patients (Guariso & Gasparetto, 2012). Although H. pylori infection is the more common cause of PUD, the use of NSAIDs increases the risk of acquiring PUD (Guariso & Gasparetto, 2012).

A pressure ulcer is localized in some part of the skin that break down when it stays in a same position because is pressing the skin or rubbing with something for a long time. Pressure ulcers have symptoms but it depends on the category, first category looks redness at the skin and the skin is not yet broken, second starts to look pink, like a blister and break the skin, the third may have some parts badly damaged that look yellowish, and fourth in the ulcers can have dead skin can be dark color that can expand to the bone and requires surgery to move the damaged parts. A patient that suffer pressure ulcer describe the color of the ulcer, how feel around the area and if they have a little bit of pain. Some patients say that ulcer look horrible,

Pressure ulcers (decubiti) continue to be problematic in all health care settings. Most pressure ulcers can be prevented, and deterioration at Stage I can be halted". The use of clinical practice guidelines can effectively identify residents and define early intervention for prevention of pressure ulcers (2015).

Stage one of these ulcers is that the skin is discolored. The skin may appear to be red. If you have a darker complexion, the skin will appear to be blue or purple. Stage two is when the skin is now open and shows signs of tissue death. A blister may appear and be filled fluid. Stage three, the ulcer is deeper in the skin. The ulcer affects the fat layer and may have the appearance of a crater. Stage four, many layers have been infected. This includes the muscle and bone layer. “Eschar”, which is a dark dead tissue, may be inside the

The vessels inside the lining also break down causing the patient to bleed. Depending where the ulcer is will the patient present with Hematemesis or Hematochezia. Another complication could be perforation of the stomach. This allows gastric contents to release into the sterile portion of the abdominal cavity. This could cause a severe infection and often requires surgical repair. The third major complication could be obstruction. Often if a ulcer presents in the narrow part of the distal stomach, an obstruction can occur. That is because scar tissue grows back often occluding the path of gastric contents. Surgical repair would also be necessary in this

In conclusion this case demonstrates the potential opportunities for the better management of patients with upper gastrointestinal bleed, including adhering to guideline-directed therapy, improving transition of care and proper documentation, which can result in better patient outcomes, lower readmissions due to rebleeding and higher

Stage two:The skin breaks open, wears away, or forms an ulcer, which is usually tender and painful.

The symptoms of the peptic ulcer will depend on the location of it. A gastric ulcer located in the stomach will result in pain of the upper abdomen approximately 15 to 30 minutes after consumption of food. Because of the pain, one may be afraid