Exercise causes you to breathe harder and your body produces more CO2. Before exercise, partner A took 9.5 seconds for the indicator (B.T.B) to turn from blue to yellow, after exercise, the indicator took 9.06 seconds to turn yellow, a difference of.44 seconds. Partner B’s data contradicts what we expected to happen because it took longer after after exercise by 1.3 seconds. When we ran our test, our data showed that when we exercise, that we would make the indicator turn yellow faster, meaning there was more carbon dioxide. When we exercise our heart starts beating faster and we produce more CO2. Partner A’s data showed that when they exercised, the indicator changed faster, because there was a larger amount of CO2. When you exercise you use
Using the lab activity, observe and record the physiologic changes that occur during exercising using the following chart:
After the exercise, we then took the measurements of our pulse, breathing rate and temperature too to see the change. Once we had completed this the first time, we then did it 2 other times, so in total 3 time so that the data was reliable and trustworthy. (Stretch, B., & Whitehouse, M. (2007).
Exercise increases the use of energy by your muscles, which activates a series of reactions to create new energy to keep exercising and maintain homeostasis. The first reaction that occurs is an increase in your breathing rate. Energy creation requires significant oxygen. The only way to provide the necessary oxygen is to increase the speed at which your respiratory system is introducing it into your bloodstream. The harder you exercise, the more energy is used, resulting in your body increasing your breathing rate even more to maintain adequate energy levels for balance.
In addition a small rise in breathing rate and this is called anticipatory rise, this happens when exercising. The average reading for breaths per minute during exercise is 23-30. This shows that with more blood pumping through the body more oxygen is needed to keep the body at a sustainable rate to help our body create more energy. Our breathing rate will keep increasing until
In this assignment I will be reviewing the different effects of exercise on the body system including the acute and long term using the pre-exercise, exercise and post-exercise physiological data which I collected based on interval and continuous training method. I will also be including the advantages and disadvantages of these, also the participants’ strengths and areas where they can improve on.
1. Explain the change in ERV with exercise. the ERV decrease with exercise asssuming that the volume of air was exhaled more than being enhaled at the time. 2. Explain the change in IRV with exercise. IRV decreased as well because the amount of air that was supposed to be inhale was very little inhalation during the time of exercising. 3. Explain the change in IC with exercise. IC increase with exercise beacause the subjects were able to allow their lungs to breath. 4. Explain the change in FRC with exercise. The FRC decrease just a little with exercise. 5. Explain why RV does not change with exercise. the ERV decrease with exercise asssuming that the volume of air was exhaled more than being enhaled at the time. 6. Explain why VC does not change with exercise. the vital capacity remain the same because it accumulated the tidal
Four interval times (PR, RT, TP and RR) measured in seconds were recorded both with the subject at rest and after the subject had exercised. The PR and RT intervals remained virtually unchanged with the PR intervals remaining the same both before and after exercise with an interval time of 0.15 seconds, and the RT interval increase by 0.01 seconds from 0.37 at rest to 0.38 seconds after exercise. More substantial changes were noted in TP and RR intervals. The TP interval decreasing from 0.32 seconds at rest to just 0.08 seconds after exercise, a decrease of 0.24 seconds (just 25% of the resting 0.32 seconds). The RR interval decreased from 0.84 seconds at rest to 0.61 seconds seconds after exercise, a decrease of 0.23 seconds
Hypothesis: If a person practices an aerobic sport on a daily basis (athlete), then he will have a higher recovery rate due to the fact that their hearts are more accustomed to intense physical exercises than someone who does not (non-athlete).
As the intensity of exercise increased, so did the rates of the heart and breathing. After a small period of rest, the heart rate and breathing rate both decreased to a point close to their resting rate. This proved the stated hypothesis. First, the hearts average resting rate was recorded to be 76 bpm. The heart is therefore transporting oxygen and removing carbon dioxide at a reasonably steady rate via the blood. During the low intensity exercise (Slow 20) the heart rate increases to 107 bpm, which further increases to 130bpm at a higher intensity level (Fast 20). The heart therefore needs to beat faster to increase the speed at which oxygen is carried to the cells and the rate at which carbon dioxide is taken away by the blood.
I predict that during exercise the heart and respiratory rate (RR) will increase depending on the intensity of exercise and the resting rates will be restored soon after exercise has stopped. I believe that the changes are caused by the increased need for oxygen and energy in muscles as they have to contract faster during exercise. When the exercise is finished the heart and ventilation rates will gradually decrease back to the resting rates as the muscles’ need for oxygen and energy will be smaller than during exercise.
The literature on the effects of exercise of cardiac output maintains the idea that exercise should affect cardiac output- pulse rate, systolic blood pressure, diastolic blood pressure, QRS-pulse lag, P-T and T-P intervals, because of increased heart rate. For our experiment, we tested this theory by measuring our cardiac output before and after some rigorous exercise. We measured the individual cardiac output and then combined the data to compose a class-wide data average. We compared the results of the experiment to what we expected, which was that exercise does affect our heart. Our data from this experiment supported the notion that exercise does, in fact, change cardiac output.
Introduction: In this experiment, cardiovascular fitness is being determined by measuring how long it takes for the test subjects' to return to their resting heart rate. Cardiovascular fitness is the ability to "transport and use oxygen while exercising" (Dale 2015). Cardiovascular fitness utilizes the "heart, lungs, muscles, and blood working together" while exercising (Dale 2015). It is also how well your body can last during moderate to high intensity cardio for long periods of time (Waehner 2016). The hypothesis is that people who exercise for three or more days will return to their resting heart rate much faster than people who only exercise for less than three days.
The effects of exercise on blood pressure, heart rate, respiration rate and electrical activity of the heart were assessed. The measurements of respiration rate, pulse rate and blood pressures were noted as described in Harris-Haller (2016). Data was first taken from subjects in a relaxed position and then followed by sets of reading after exercising based on one minute intervals. The data also noted sitting ECG traces from Harris-Haller (2016). The respiratory rate, pulse, blood pressure, P wave, QRS complex and T wave were defined for each subject. The class average was calculated for males and females and graphed to illustrate the results by gender for each cardiopulmonary factor.
The controlled variable included the exercise bike and heart rate monitor. There are several limitations, systematic and random errors that should be considered when interpreting these results. (4) The controlled variables were not tested before this experiment to see if they were working and reliable. Figure 2 heart rate was quite inconsistent and did not follow the pattern of the other results, which maybe suggest a random error with the heat rate monitor. A systematic error could include the fitness of the participants. One of the test subjects is an endurance athlete and the other does not compete in any sport. This would affect the results because for the endurance-trained athlete, from their training they increase their cardiac output results from a substantial increase in maximal stroke volume. In untrained persons, cardiac output increases in response to exercise primarily by an increase in heart rate. The endurance-trained athlete does so mainly by an increase in stroke volume. Simply meaning that although both participants are doing the same cadence and length the endurance athletes skewers the results by already having an increased rate in stroke volume. Another systematic error may include the rate of perceived effort. For the most accurate results, the measured maximum heart rate would be necessary to give an accurate cadence to ride at.
The effects of heart rate on differing durations of exercise were studied in this experiment. For people, heart rate tends to increase as they perform physical exercises. The amount of beats per minute gradually increases as people perform physical activities. Heart rates taken before exercise are relatively low, and heart rates taken one minute after exercise increase significantly. Heart rates slowly begin to decrease after they are taken two minutes and three minutes after performing the step test, which is to be expected. The rates of intensity throughout exercise relates with changes in heart rate throughout the step test performed in the experiment (Karvonen 2012). The age of the participants affected the experiment, since the heart rate during physical exercise, in this case the step test, is affected by age (Tulppo 1998).