Good Agreement Between Echocardiography And MRI For The Estimation Of Segmentation

At this point, we do not know exactly what are the settings, we have previous requested pacemaker operative report from Regions. Unfortunately, from what he is telling today, his date of the birth was not correct as such dose report were not same. He noted that he thinks he came back from the hospital, he has not had similar complaint or concern or report of chest pain. It should be noted that hospital records, with a troponin that was negative, 12-lead EKG was similar to the one that was obtained here, essentially identifying sinus bradycardia with first-degree AV block, left ventricular hypertrophy with repolarization abnormality. QT was prolonged, similar EKG obtained at facility also identified pretty much the same abnormality pattern. Troponin was negative. Other workup included chest x-ray in the hospital were all unremarkable. Today he is not reporting any chest pain, no shortness of breath, no nausea or emesis. He has got healed ____ scar to the left chest from pacemaker implantation which is completely healed but slightly

4. left ventricle 5. superior vena cava 6. inferior vena cava 7. ascending aorta 8. aortic arch 9. brachiocephalic artery 10. left common carotid artery 11. left subclavian artery 12. pulmonary trunk 13. right pulmonary artery 14. left pulmonary artery 15. ligamentum arteriosum 16. right pulmonary veins 17. left pulmonary veins 18. right coronary artery 19. anterior cardiac vein t s w x v 20. left coronary artery 21. circumflex artery 22. anterior

Cardiovascular Assessment: No visible pulsations, no heaves or lifts. Apical pulse present in the fifth intercostal space at the left midclavicular line. Auscultation of apical rate 62 beats per minute, normal rhythm regular S1 - S2 heart sounds present. Pulsations present when supine and disappear at a 45 degree angle position. Extremities are brown color without redness, cyanosis, lesions or varicosities bilaterally. Temperature warm bilaterally, Allen test was negative. Homan’s sign negative. Carotids: +2 and present bilaterally. Right Radial +2, left radial +1 , Right Brachial: +2

Cardiac Echogram- This is to identify structural abnormalities in cardiac function, cardiac output, abnormal valves and MI (Buttaro, Trybulski, Polgar-Bailey, & Sandburg-Cook, 2017).

The patient has been diagnosed with atrial defibrillation and congestive heart failure. There is no visible jugular vein distention or pulsations on either side of the neck. The patient was not comfortable with removing her sweater, therefore, pulsations, lifts, or heaves were not seen if present. Palpated and auscultated the carotid arteries for vascular sounds, no bruits heard. Heart sounds were auscultated with the bell and diaphragm of the stethoscope. S1 82/min, even and regular. S2 85/min, even and regular. S3 82/min, even and regular. S4 83/even and regular. No murmurs were heard. The apical pulse rate was 92 bmp, regular and was accessed with patient in sitting position and between the 4th and 5th intercostal space. Patient stated

Denies family member with risk factors, morbidity, mortality related to cardiovascular system, hypertension, dyslipidemia, diabetes, heart disease, thrombosis, peripheral vascular disease, or abdominal aortic aneurysms

A heart murmur are harmless, healthy and don’t need a treatment. During a physical exam, doctors know that every child has a heart murmur and can hear the whooshing sound. There are some things that can be abnormal when it can be very damaged by the heart valve which 1/3 of the populations are born with valve problems. This condition is making the heart go faster than it normally would and it can be forced to have the blood goes faster. The causes are anemia, high blood pressure, thyroid, and a fever. Those causes can make the blood flow a lot faster, which can be dangerous if the problems are by the birth defect of the child. The valves open and close by four chambers, two on top and two on bottom. Stenosis, mitral prolapse, congenital, and

Mr. Heart had coronary artery bypass to four vessels three days ago. His transcutaneous epicardial pacing wires were discontinued in the morning along with the single chest tube he had. He is out of bed in a chair and ambulated 200 feet. His blood pressure is 120/72, heart rate 88 regular in rhythm and without ectopy, respiratory rate 18 and unlabored.

Sinus bradycardia is defined as a heart rate of less than 60bpm in conjunction with normal sinus rhythm. A bradyarrhythmia, on the other hand has a disturbance in cardiac conduction with rate of less then 60bpm (Swift, 2013). Intrinsic and extrinsic etiologies contribute to bradycardia. Intrinsic causes relate to abnormalities in the conduction system, where extrinsic causes include: increase vagal tone, electrolyte imbalance, hypoxia, hypothermia and hypothyroidism (Swift, 2013).

Supraventricular tachycardia is increase in heart rate over 150 bpm due to do the over firing or redirected firing of the SA Node conduction above the ventricles. With supraventricular tachycardia the patient can have an abrupt onset and termination of rhythm, flattened or retrograde conduction P waves and narrow QRS waves specifically less than 0.08 second (Kyle, 2012).

Atrial septal defects (ASDs) are a deficiency in the wall between the right and left atrium of the heart (5). ASDs account for 10% of all congenital heart defects(19) and 30% of those diagnosed in adulthood (20). Definitive diagnosis is by transthoracic echocardiography, though signs of an ASD can be found on physical examination, electrocardiography and chest radiography (21).

In contractile tissues, its function is directly related to contractile fibers [4]. However, in the heart, its function depends on contractile, conducting and endocrinal fibers. The contractile fibers (CF) are responsible for the pumping function [5]. While the cardiac conduction system (CCS) is responsible for the initiation, coordination and conduction of the heartbeat [6]. Many researchers have paid considerable attention to discriminative techniques between CF and CCS as they can be used to describe their normal function and predict dysfunction with pathology or malformation [7]. The traditional methods of gross dissection are Periodic Acid-Schiff (PAS)

The cardio-trachea ration is appropriate with the heart being no wider than half the width of the chest. The heart is located mostly on the center line and left side. There is not much heart volume on the right side. The left hila and right hila differ in size. They seem to be the same density though. The left hila is sharper than the right hila. There seems to be a loss of definition on the right border of the heart. The gastric bubble on the patients left side under the diaphragm is evident. The left costophrenic angle appears more sharp. The right costophrenic angle seems blunted. However, the costophrenic angle are pointed downward. There is no evidence of air under the diaphragm. When looking at the bones and soft tissue, I notice a bright white speck on the 8th left posterior rib. The ribs appear smooth with no abrupt edges. In the area lateral to the right side of the heart, there is minute blotches that are bright and white compared to the other vascular

Echocardiography is the initial diagnostic modality for a patient with suspected congenital heart disease. In some patients, however, use of this modality is encumbered by its limited ability to delineate great arteries and intra cardiac anomalies, pulmonary veins, and coronary arteries. (7)

A two-dimensional transthoracic echocardiogram is a primary diagnostic procedure used to identify cases of restrictive cardiomyopathy. Bi-atrial enlargement is most commonly seen during a transthoracic echocardiogram. The left ventricle is often normal in size, but wall and septal thickness may be increased if infiltrative diseases are present. Ventricular walls are seen to be inflexible or stiff causing diastolic dysfunction. Valvar thickness may also be seen along with mitral and tricuspid regurgitation. The walls of the heart will have a ground glass appearance, better seen in the parasternal long axis during a transthoracic echocardiogram. Systolic function is generally well persevered while diastolic function is decreased. Ejection fraction