The study by (Zhang et al. 2012) uses online open-ended questionnaires (for textual analysis) and subgroups or focus groups (in this case there were non-time variant groups such as race, sex, BMI and time variant groups such as purine intake, use of alcohol, treatment with diuretics, allopurinol and colchicine use). Also in the conclusion it mentioned that cherry intake reduces the risk of gout attacks “Should our findings be confirmed in randomized clinical trials…”. These features all point towards it being a qualitative study.
The objective for the study by (Zhang et al. 2012) was to research the connection between the intake of cherries and the risk of recurrent gout attacks in patients with gout.
This qualitative case-crossover study
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Also when cherries were consumed with allopurinol (gout treatment) it showed a 75% lowering in the risk of recurrent gout attack compared to no exposure to either with an OR of 0.55, which was within the 95% confidence interval (0.30 – 0.98). Across the subgroups of sex and BMI the relationship trend of cherry intake and lower gout attacks persisted as being within the confidence interval (95%). Also, the subgroups with higher alcohol consumption and purine intake whilst using diuretics were associated with higher gout attacks than those that use allopurinol and colchicine. Also, cherry intake effects seemed to give lower attack risks when associated with an abstinence from alcohol or higher purine intake and when diuretics were not used. Important to note though is that none of these factors showed large interaction (P value for interaction was > 0.17) with the effect of cherry intake (e.g. cherries still lowered the risk across all subgroups. From these results it is clearly apparent that cherries have a relation to lowering the risk of recurrent gouty arthritis
Raspberries are in fact the exposures associated with the illness. The high odds ratio and p-value indicates the strong association.
Gout is a painful form of arthritis caused by the buildup of uric acid in the joints. In addition to medications, treatment for gout involves limiting the intake of foods high in purines, according to MayoClinic.com. These foods include liver, herring, mackerel and anchovies. Red meat, fatty fish and other seafood can also exacerbate gout. MayoClinic.com says the diet for gout involves high intake of plant-based proteins, low-fat dairy products and complex carbohydrates.
Being that different health authorities and nutritionists class sugar in a different category than let's say fructose, as found in fruit, or sucrose, as found in desserts, some health experts recommend that people get to know their fruit well as to their glycemic index count.
Using a double blind placebo clinical trial the research team hoped to find accurate results to support their hypothesis. All these factors would normally yield accurate results, however with only 22 individuals being given the cinnamon pills, the experimental group is too small to be an accurate representation of how this treatment would effect people worldwide. Another flaw in this grouping is that the participants in this study were limited to people who went to Tehran University of Medical Sciences' Endocrinology and Metabolism center, which was likely to have left the group of participants without the genetic diversity to be representative of the world's population of type 2 diabetes patients. Given these flaws it can be drawn that errors could have occurred in the measurements, or that the cinnamon could have influenced some other factor of the patients medical status that was not taken into account for this study. The inclusion of these issues into the result of this study would have also left them flawed.
Fourteen million American adults needlessly suffer from macular degeneration. Kiwifruit is rich in phytochemicalsóxanthophylls and especially a subcomponent, lutein. Lutein is known to accumulate in the retina of the eye. Recent USDA studies have linked these important compounds to the prevention of macular degenerationóthe leading cause of irreversible blindness in the U.S.
A diet high in sugar, seafood and meat increases uric acid levels. Alcohol and being overweight can also raise your risk of developing gout. Kidney disease and high blood pressure can also cause gout. The condition can be hereditary. Gout occurs
Along with the development of modern society and lifestyle activities, gout is formed by the impact of the environment and daily activities, is becoming many people's fears, so the understanding of the disease to early preventive measures, effective treatments play an extremely important role. Gout is a metabolic disease uric acid, leading to deposition of monosodium crystals in the organization (synovium and organizations around the joints, kidneys and kidney tissue tube), the contributory factors can find in some certain common medication, alcohol, and dietary foods (Gout 2016). The frequency of occurrence of gout increased significantly with age, and correlated with the increase of serum uric acid concentrations. The pathogenesis
This passage addresses some risk factors of gouty arthritis and discussed dietary recommendations for different groups to prevent developing or worsening of gout. Overall, hyperuricemia and gout patients should adhere to the treatment regimen provided by medical professionals and follow the dietary recommendations suggested, aiming to reduce sUA by medications and lifestyle changes so as to prevent gout attack. High risk group, such as man and postmenopausal women, should take actions to reduce modifiable risk factors of
A recent study by the American Journal of Clinical nutrition (2011) found that over a 15 years period, men and women who consumed nuts had a 51 percent lower risk of dying from an inflammatory disease like rheumatoid arthritis compared with those who ate the fewest nuts while another study found that subjects with lower levels of vitamin B6 found in most nuts had higher levels of inflammatory markers while there is no definitive arthritis diet. Vitamin B6 functions in the metabolism of amino acids, it is essential for formation of red blood cells and metabolism
Dietary sugar consumption has gained prominence as a highly controversial subject at present in regards to public health due to the significantly high level of added sugar intake in the western world among both adults and children (Wittekind and Walton 2014). The dietary sugar fructose in particular has become an area of great interest in the field of nutrition and health (Feinman and Fine 2013). Excessive consumption of any macronutrient or micronutrient has the potential to influence health outcomes, however fructose has been implemented as a significant contributor to the western world’s current health epidemic of cardiorenal disease which is characterised by obesity, hypertension, metabolic syndrome, type II diabetes, kidney disease and vascular disease (Collino 2011). The incidence of these diseases have grown to pandemic proportions and are continuously rising in the western world. Among the United States population, more than one-third of adults (34.9 %) and one-sixth of children are affected by obesity, 24.5 % of adults are affected by hypertension (CDC 2014), 8.5 % of adults are affected by diabetes, 22.9 % adults are affected by metabolic syndrome, 1.8 % adults are affected by kidney disease and 10.9 % of adults are affected by cardiovascular disease (http://ftp.cdc.gov/pub/Health_Statistics/NCHS/NHIS/SHS/2014_SHS_Table_A-4.pdf). Not only are these diseases a huge medical burden they also are an economic burden and because of this, the metabolic effects of dietary fructose are of immense interest.
A prospective study was conducted by the Nurses’ Health Study, which tested vitamin A intake in the risk of women. The results proved no support for dietary carotenoids such as alpha-carotene or beta-carotene in lowering the risk of MS in women (7). Even though this was one of the few large-scale studies-- comprising of 81, 683 women inspected throughout 12 years (7) -- the scientific data proved no association to a beneficial role of vitamin A intake to MS risk in women.
Mean intake of total fructose as a percentage oftotal energy increased from 8.1% in 1978 to 9.1% in2004, and thisincrease was due to increases in fructose from sugarsand sweeteners and not from naturally occurringfructose in fruit which rather decreased in all age groups.7 The major source of fructose in thediet comes from fructose-containing sugars(sucrose and HFCS) largely in the form ofadded sugar.Since the glucose intake and fructose intake co-varies , epidemiological studiescannot completely differentiate between the
The authors, Raatz, Johnson and Picklo, based out of the University of Minnesota, performed an experiment to test three popular nutrient sugars on glucose tolerant and intolerant individuals: honey, sucrose, and HFCS. HFCS is said to be the worse sweetener for health on the market followed by sucrose. On the other hand honey is thought to be the ‘natural’ sweetener based on its supposed ability to provide the body with
Why? Because the fructose in these drinks can increase the levels of calcium, oxalate, and excretion of uric acid, which ultimately contributed to the formation of kidney stones.
In general, the three foods/beverages that place patients at risk for gout are red meat, beer, and fructose, a sugar commonly found in sugar sweetened soft drinks. Finally, patients with gout should decrease their use of hard liquor and beer with a substitution of wine if