HPV Life Cycle Model

These possible increases of HPV throughout the United States and the rest of the world can be attributed to many

Summary of article: In “Bridging the HPV Knowledge Gap,” Christina Demopoulos and two others talk about the importance of knowledge on HPV and how most patients are unaware of this virus and that is it on the oral health professionals to educate them. This virus is transmitted easily through a small cut or tear in the oral cavity and is associated with a variety of oral lesion, cances, and oropahygnel cancers. The article included a statistic that about 12,000 people between ages 15 and 24 are affected everyday by HPV and its only increasing. HPV causes more than 30,000 cancer cases a year which the average age diagnosis is 62, but ther is arise of cases in the adolescents. With that being said, it is put on oral health professional to discuss the ipmo ratio of HPV with patients, and they should become more familiar with HPV because there is an association between HPV and oropharyngeal cancers. One way to bridge the HPV knowledge gap is to do what NEvada is doing. Nevada has a Immunization program which promotes for better health and they hold a lot of information conferences. Overall,

Human Papilloma Virus, more commonly known as HPV, is a sexually transmitted virus. It is spread from skin-to-skin contact with an infected person, typically during sexual encounters. It is the most common sexually transmitted disease in the United States. According to the Immunize Action Coalition, “most sexually active American men and woman will contract at least one type of HPV virus during their lifetime” (A Parent 's Guide to Preteen, 2013, p. 1). To put the numbers in perspective, it infects an estimated seventy-nine million Americans today with approximately fourteen million more people being affected additionally each year (Human

Human papillomaviruses (HPV) are a group of more than 40 types of viruses that infect the genital areas, throats (recurrent respiratory papillomatosis), and mouths of males and females and is the number one most common sexually transmitted disease (Center for Disease Control and Prevention [CDC], 2009). It is spread by genital contact. It is most easily spread by vaginal and anal sex, but also through oral sex and even just genital-to-genital (skin-to-skin). In 90% of people that contract HPV, their own immune system

HPV stands for the human palliloma virus. It is estimated that there are around 100 different types of HPV. The HPV virus can cause lesions to develop on the soft palate, tonsils and tongue. It can also increase the risk of certain forms of cancer, including oral cancer and cervical cancer.

Human Papillomavirus(HPV) is the most common sexually transmitted infection in the United States, according to Centers for Disease Control and Prevention(CDC) around 20 million people are infected with HPV and additional 6.2 million people are newly infected every year. According to National Health and Nutrition Examination Survey in 2003-2004 among sexually active women (57% of 14 to 19 years and 97% of 20 to 59 years) HPV was highest prevalent in youngest age group (40% of 14-19 years and 50% of 20-24 years). Infection rates decrease after age of 24 years. There are more than 100 types of HPV viruses out of which 40 cause genital infections. Out of

In their 2015 study Alves Ribeiro et al. found that women who began having sexual intercourse prior to age of 16 were more than four times as likely to have a HPV infection (OR 4.41; 95%CI:1.20-19.33), and nearly five times as likely for that infection to involve serotypes 16 and 18 (OR 4.68; 95% CI: 1.20-21.32). Moreover, women with a sexual debut prior to age 16 were twice as likely to have a diagnosis of cervical intraepithelial neoplasia (CIN)1 or greater (OR 2.2; 95% CI: 0.94-5.08 CITE 2. The same study also demonstrated a statistically significant association between CIN 1 and HPV infection, especially infection with HPV serotypes 16 and 18 (OR 2.76; 95% CI: 1.05-7.19) and (OR 3.53; 95% CI: 1.17-10.67) respectively (Alves Riberio et al., 2015). Over 90% of females in the United States are sexually active by age 24; correspondingly, the prevalence of high-risk HPV peaks at over 43% in the 20-24 age group (Vegunta, Kransdorf, & Mayer, 2014). By age 30, the prevalence of high-risk HPV decreases to 30.4%, and continues to decrease as age increases. It is thought that this age related decrease in prevalence can be attributed to a combination of factors, including, decreased exposure, resistance to reinfections, and HPV’s self-limiting nature (Alves Ribeiro et al., 2015; Vegunta et al., 2014).

Did you know there is more of a link between cervical cancer and HPV than smoking and lung cancer? Cervical cancer is currently the fourth most common cancer in women and the second leading cause of death from cancer in women. Along with those statistics, human papillomavirus is the most common sexually transmitted viral disease amongst men and women worldwide. The prevalence of women having a strain of HPV that ultimately leads to or increases the chance of cervical cancer is highly relatable. What is pathophysiology, signs, symptoms, prevention, and treatments of this ailment? You might ask will be covered in the next few pages of this paper.

Human Papillomavirus (HPV) is a double -stranded deoxyribonucleic acid (DNA) virus that only infects humans with an attraction to both cutaneous and mucosal surfaces such as the cervix, anus, tonsil, and oropharynx (Clark, 2013). HPV is a type of oncogenic virus that goes into the cells and can cause several diseases. Over the years, research has surfaced connecting genital HPV to several types of cancer. There are over a hundred strains of HPV but the most high risk strains, 16 and 18, have been shown to cause vulvar, vaginal, anal, and the most concerning, cervical cancer (Chan, Ng, & Wong, 2012). Genital HPV

Human papillomavirus (HPV) is made up of double stranded DNA with a capsid. Their viral genome is consisting of such as early (E) gene like E6 and E7encodes for oncoproteins and late (L) gene such as L1 and L2 encodes for structural proteins like capids. There are more than 100 different types of HPV identified and they generally infect around cutaneous or mucosal areas. HPV viral genome can be categorized into low risk (LR) and high risk (HR). High risk types such as HPV-16 and -18 are ones that can develop into cancer and other tumors mostly in genital area or a head and neck squamous cell. Low risk types such as HPV-6 and -11 are more frequently found I larynx or genital area (Klozar et al., 2010). Therefore, careful evaluation such as risk factors, or possibly a new vaccination to prevent against HPV related to oropharyngeal SCC should be questioned in public health

Human Papilloma Virus (HPV) accounts for the third most prevalent cancer in females worldwide,15 as it is the most common sexually transmitted infection (STI).2 There are more than 100 subtypes of HPV; 16 of those subtypes are known to be high-risk.15 HPV is a crucial precursor to cervical cancer in 99.8% of those affected,2 with subtypes 16 and 18 being the most common types.15

My initial instinct was to continue looking for another topic; however, after a few moments it occurred to me that I didn’t know as much about the topic as I had thought. All I truly knew was the fact that it is a STI, and one can receive a series of vaccines to prevent it. After conducting my research on HPV, I am happy I choose this topic due to my need for increased knowledge not only for myself but for my patients’ health as well.

The human papillomavirus is a type of virus that is spread through skin to skin contact with an infected person. It is very common. In fact, it is estimated that 20 million people in America have been infected with the HPV virus. This virus may not produce any symptoms.

HPV types 6 and 11 account for 90 percent of all genital warts cases (Dinh, et al., 2008).

The male sexual is the vector of the HPV infection. Personal interviews were conducted and demographics showed smoking, genital hygiene, circumcision use, and history of STDS. HIV was not tested. The HPV was detected by swabbing the external genital area with cotton swabs, one over the sulcus coronaries of the penis, and the other over the shaft of the penis, the scrotum, and the perianal region. The swabs were immediately placed in the same plastic tube containing 3 ml phosphate buffered saline and 0.005% thiomersal and kept 4 degrees Celsius until mailed for analysis. Next, after centrifugation for 10 minutes at 2755 g, the pellet was resuspended in 100