Hashimoto Thyroiditis Essay

My grandmother suffered with thyroid and use to always ask me if I had thyroids. I remember a time when she told me to get a quarter and some iodine to put on my stomach above my navel. She told me if it changed colors that I had thyroids. Also, because I felt so drained most of the time and would swell in my body often, she had me to consult with my physician about the matter. My grandmother was a very intelligent woman that was very health conscience about herself and her family.

The exact mechanism of the autoimmune disease is unknown. Some of the medications that increase the risk of Hashimoto’s disease are called interferon, lithium, and amiodaron. The environmental radiations that one would be exposed to can increase the risk of getting hypothyroidism as well as having the Hashimoto’s Disease. The symptoms of the disease can develop after a pregnancy and a viral illness. Symptoms of Hashimoto’s disease tend to develop slowly over a period of several years. There are several different symptoms a couple of the visual symptoms are the enlargement of the thyroid and growth of the nodular. Other symptoms for the disease are that of being fatigue, having an intolerance for the cold, the menstrual cycle changes as well as infertility, can have an unexplained weight gain can happen and many more symptoms. There are some experiences of the patient having tenderness and prolonged pain, but pain is not a common symptom of the

Hashimoto’s Thyroiditis, also called Hashimoto’s disease is an autoimmune disease in which the body’s immune system attacks the thyroid gland. It was named for the Japanese surgeon who discovered it in 1912 The thyroid is a small butterfly shaped gland in the front of the neck. The thyroid makes hormones called T3 and T4. These hormones regulate metabolism. The thyroid is controlled by hormones of the pituitary gland, which is also called the “master gland.” It is a pea-sized gland located in the base of the brain which, among others, makes thyroid stimulating hormone, or TSH. TSH stimulates the thyroid to make thyroid hormone. With Hashimoto’s disease, the thyroid cells are damaged resulting in the inability to make enough thyroid

Laboratory Report/ Miranda Tefft/ Homeostatic Imbalances of Thyroid Function/ Aline Potvin/ 11.18.2014/ Page [2] of [3]

2. The subject who was diagnosed with secondary hypothyroidism was given levothyroxine (synthetic Thyroxine). After 6 weeks of

Hashimoto thyroiditis is a common disease caused by hypothyroidism. The immune system cells attack the thyroid gland, causing inflammation and eventually destruction of the gland. This reduces the thyroids ability to make hormones. These hormones are known as thyroxine (T4) and tri-iodothyronine (T3) (Better health channel 2015).

Week 3 – LOM Assignment The thyroid is responsible for taking iodine and converting it into thyroid hormones which are released into the blood stream and transported throughout the body where they control metabolism.1 The thyroid is a vital part of the human body as every cell in the body depends on it for metabolic regulation. Too much or too little thyroid hormone secretion can result in hyperthyroidism or hypothyroidism. Hyperthyroidism is caused when the thyroid secretes too many thyroid hormones. Hyperthyroidism causes a rapid heart rate, elevated blood pressure and tremors. Other symptoms include increased nervousness, inability to concentrate, weakness, restlessness, difficulty sleeping, frequent bowel movements, weight loss and irregular

Hashimoto’s Thyroiditis is considered an autoimmune disorder of the thyroid, first discovered in 1912, by a Japanese doctor, Hakaru Hashimoto. Hashimoto’s Thyroiditis disease is currently considered a genetically predisposed disorder caused by both environmental and endogenous conditions. Genetically predisposed Hashimoto’s Thyroiditis, occurs more often in Caucasians over other ethnicities. Environmental factors that produce the autoimmune disorder are infections, disproportionate iodine intake, drugs, as well as chemical and radiation exposure, according to Syrenicz, Anhelli (2013) This paper will discuss the importance of Hashimoto’s Thyroiditis disease, symptoms, Metabolic and Physiological effects, treatment options, and side effects associated with treatments.

Background: An 83 year old female is taking her levothyroxine medication to help treat her thyroid disorder induced by Hashimoto. She has been taking this medication for thirty years. While always being adherent to taking her medication, she has never really understood what Hashimoto thyroiditis actually is, or why levothyroxine is the best choice of therapy for treating her hypothyroidism caused by the disease. She is also positive for hypertension, GERD, and insomnia.

Hashimoto’s thyroiditis is an autoimmune disease in which the body’s normal defense mechanisms attack the thyroid gland. This causes inflammation in the thyroid, which at first causes uncontrolled

The thyroid gland is the gland that makes and stores hormones that help regulate the heart rate, blood pressure, body temperature, and metabolism. Thyroid hormones are essential for the function of every cell in the body. They help regulate growth and the rate of chemical reactions in the body. Thyroid hormones also help children grow and develop. The thyroid gland is located in the lower part of the neck, below the Adam's apple, wrapped around the trachea. It has the shape of a butterfly with two lobes attached to one another by a middle part called the isthmus. The thyroid uses iodine, a mineral found in some foods and in iodized salt, to make its hormones. The two most important thyroid hormones are thyroxine (T4) and triiodothyronine

c.i. Fatigue, weight gain, cold intolerance, depression, and slowed heart rate are just a few of the many symptoms

The main cause of hyperthyroidism Graves’ disease which affects quite a few people in the general population. This disease is characterized as an autoimmune disorder. This means that the body makes antibodies known as thyroid stimulating immunoglobulin that bind and over activate the production of thyroid hormones [3]. So in essence the antibody mimics the thyroid stimulating hormone and when the negative feedback loop is in affect the antibody still binds to the thyroid and T3 and T4 are over produced [2].

Hyperthyroidism is caused by an excess of thyroxine (T4) or triiodothyronine (T3), or both. This excess is a result of a malfunction of the thyroid gland as indicated by the picture below. There are many different symptoms of hyperthyroidism, some of the most common being major weight loss despite having a insatiable appetite, anxiety, heat intolerance, fatigue, weakness, hyperactivity, irritability, apathy, depression, and sweating. In addition, many experience shortness of breath, loss of balance, nausea, vomiting, osteoporosis, and diarrhea. However, those who undergo hyperthyroidism later in life may not experience most of these symptoms at all and instead only feel constantly fatigued and considerable weight loss. Nonetheless, neurological symptoms may occur in addition to the physical manifestations. These neurological symptoms include tremor, chorea (ticks), myopathy (muscle weakness), and temporary but periodic paralysis. There are a variety of ways to treat hyperthyroidism, depending on the age of the patient, the size of their thyroid gland, and their general health. The most common treatments include beta-blockers, which do not actually cure hyperthyroidism but mask some of the symptoms such as tremors, anxiety and so on. This, however, is not advised for those with heart or lung problems. Another possible treatment for this disease is radioactive iodine, or RAI. RAI therapy is the most common treatment for hyperthyroidism

Chronic diseases such as hyperthyroidism can be debilitating if not managed well. Hyperthyroidism is an overactive thyroid gland that secrets too much hormones throughout the body causing many different clinical manifestations within all systems. The thyroid gland is located right underneath the larynx also known as the voice box, and straddling the windpipe.