Helicobacter Pylori Research Paper

If you were prescribed an antibiotic medicine, use it as told by your health care provider. Do not stop using the antibiotic even if your condition improves.

Helicobacter pylori (H. pylori) is a human pathogen that is responsible for ulcers and stomach inflammation. The organism had been researched by several scientists around the world since 1875; however none of them was successfully culture it. Until 1982, Dr. Barry Marshall and Dr. Robin Warren succeeded in observing the colonies, and thus became the first scientists to discover this bacterium. By self-testing, Dr. Marshall drank a liquid that contained H. pylori culture. A few days later, he got sick with many symptoms such as vomiting and nausea. After ten days, by undergoing endoscopy, he was able to confirm the role of H. pylori in causing gastritis. Then he and Dr. Warren also demonstrated the effect of

In the world of microbiology it is vitally important to be able to discern the identities of microorganisms. Not only is it important in a lab setting but as well as in healthcare in general. Properly identify what strain of bacteria a person has will aid in the proper medicine and dose given. Throughout the semester we have learned about different types of bacteria and certain test that can clearly identify them. The purpose of this lab report is to identify a Gram-positive or Gram-negative bacterium. Using all the knowledge of procedures and lab techniques identify the unknown and discuss all the tests you performed.

patient tested positive for H. pylori and wakes up at night due to cramping pain with bloating, so it is assumed to be peptic ulcer and the status of his condition is uncontrolled. Her symptoms were not improved with over the counter medication, Tums. Therefore, it is not GRED. However, Patient does not need a referral for endoscopy this time around because she does not hat the symptoms vomiting, nausea or dizziness.

With the antibiotics if H Pylori is confirmed, what would you educate the patient about these medications?

H. pylori thrives in the acidic environment of the stomach and the finding that this bacteria contributed to the condition was remarkable news and led to the standard practice of treating peptic ulcers with antibiotics. As a result, the rate of H. pylori-induced ulcers has dropped by more than 50 percent. (Ackerman, 2012)

H. pylori is a narrow, curved, Gram-negative rod with polar flagella. It grows slowly in a microaerophilic atmosphere in a manner similar to Campylobacter. The bacteria have only recently be identified as the causative agent to gastric ulcers, a condition long believed to be the result of stress and lifestyle habits.

In our world we are surrounded by microorganisms, some are harmless, many are beneficial; they are a part of our normal flora on our skin and in our intestines, providing a barrier against microorganisms that may not be so harmless. It is important, however, to distinguish which microorganisms are potentially pathogenic from those that are not, and when someone does get ill to determine what the organism is so appropriate treatment can be determined.

Helicobacter pylori weaken the gastric layer, predispose the stomach to infection which can lead to achlorhydria. “The classic symptoms of GERD are heartburn, acid regurgitation, dysphagia, and belching”( over time cause narrowing and strictures. A person can experience reflux one hour after eating high fatty undigested meals, smoking (nicotine) and alcohol drinking, sore throat and asthma. Pressure on the diaphragm from pregnancy and obesity in male and female increases intra-abdominal pressure exposing the individual to reflux and heartburn. GERD can occur in infants, elderly, and at any age in between. Patient can take antacid (Tums) to neutralize the acidity, H2 blocker (cimetidine, famotidine, or ranitidine) decrease acid production in the stomach and Proton pump inhibitors (PPI) inhibit acid production from gastric parietal cells. Others treatment, Endoscopy of esophagus or stomach, to obtain a biopsies for (precancerous cells), pH monitoring rule out H. pylori infection, looking for edema, erosion, and bleeding. If blood loss is low, replace with

Antibiotics like ciprofloxacin and doxycycline kill the bacteria or inhibit growth of the bacteria that causes eventual death.

One of the important aspects to consider is the high resistance levels of H. pylori to antimicrobials agents commonly used for treatment of gastric mucosal infections, such as ampicillin, clarithromycin, tetracycline and metronidazole

If you were prescribed an antibiotic medicine, take it as told by your health care provider. Do not stop taking the antibiotic even if you start to feel

Since then, H. pylori has been almost completely eradicated in western countries, being placed onto the endangered species list. Unfortunately in developing countries, gastric and duodenal ulcers remain huge problems. The lack of access to antibiotics and the emergence of antibacterial resistant strains have greatly hindered efforts to cure the diseases. The controversy is not over, however, as 80% of people infected with H. pylori never develop any gastrointestinal diseases. We need to thoroughly research the bacterium and exactly how it works in order to fully understand the role of H. pylori in stomach diseases. With that information, we will have the means to combat the diseases caused by the bacterium and the lack of the

The ways you can prevent from getting bacterial vaginosis is abstaining from having sexual contact with someone, limiting your number of sex partners to a minimum of people and not a huge amount, and by not douching.

The bacteria have an ability to fight the normal bodily process of eliminating germs. Recently, the mechanism used by the bacteria to avoid detection has been identified. The typhoid bacteria use a protein to kill macrophages, which are our first defense against infection. Without these macrophages, infection goes unnoticed into the body. (sciencedaily.com) Killing these macrophages is the key to survival for the typhoid bacteria. Researchers identified in mouse cells a protein kinase called PKR that causes the death of this macrophage, or large white blood cells that act as the body's first defense against