How An Abdominal Aortic Aneurysm?

E. List the arteries that branch from the descending abdominal aorta to the femoral artery.

The upper GI series shows a dilated first and second portion of the duodenum with an abrupt and vertical obstruction of the third portion. (Gebhart 2015). CT scan may also be used to provide images of both the abdominal tissue in cross-sections allowing to see the superior mesenteric artery and the abdominal aorta including the internal organs (Brunner's and Suddarths 2014). An Endoscopic ultrasound can also be used to visualize the aorta mesenteric angle with accuracy. The use of an upper endoscopy can rule out other pathologies such as mechanical lumen structure and duodenal ulcerations that can mimic superior mesenteric artery syndrome (Gebhart

Aortic stenosis—aortic valve stenosis— is caused by a narrowing of the heart’s aortic valve which leads to obstruction of the outflow of the left ventricle. Aortic stenosis is uncommon in patients under the age of 50. The most common cause in adults within industrialized countries is due to aortic valve calcification. Compared to any other cardiac diseases stenosis of the aortic valve is associated with increased morbidity and mortality. Some of the more uncommon causes of this deadly disease include congenital heart disease, rheumatic fever, and radiation. Patients with increased age, increased low density lipoprotein (LDL), increased lipoprotein A, hypertension, and smoking history have a higher risk of developing a stenotic valve. Patients suffering from aortic stenosis often are asymptotic until more of the advanced symptoms develop. At this time, they may present the “classic triad symptoms,” which include (1) angina, (2) syncope, and (3) heart failure. There are also some broad symptoms such as fatigue, dyspnea (usually with exertion), swollen ankles, difficulty exercising, or palpitations. Physical examination provides valuable insight into the diagnosis of aortic stenosis. Findings on physical examination can be confirmed through non-invasive two-dimensional Doppler echocardiogram, which is the gold standard for aortic stenosis. However, other imaging modalities for diagnosis and treatment options of this debilitating disease will be discussed in this

There were no early or late postoperative deaths and we achieved 100% follow-up for included patients. No patient had aortic valve replacement after one year. Preoperatively the mean ejection fraction in group S was 62.33±4.39% while in group R was 59.53±6.10%, the width of the regurgitant jet in group S was 34.67±2.72 % and in the group, R was 35.73±1.87 % ( p-value non-significant). Postoperatively after 1 year follow up the width of the regurgitant jet in group S increased significantly to 37.27±4.67% ( p > 0.5) while in group S almost remained unchanged 34.73±4.13% ( p <

Sometimes people who have problems with the valve that blood flows through as it leaves your heart have an increased risk of thoracic aortic aneurysm.

All of the factors contribute to the patient’s risk, but only hypertension can potentially be modified to decrease the patient’s risk for further expansion of the aneurysm.

A common treatment for aortic stenosis is an aortic valve replacement. There are currently two methods of performing an aortic valve replacement. The first is a surgical aortic valve replacement (SAVR), where the surgeon removes the diseased valve and replaces it with either a mechanical or a tissue valve. This requires a large incision, breaking of the breastbone and ribs and going on heart-lung-bypass. The recovery time for a SAVR is typically 6-8 weeks to heal and a few months to return to normal activity [4]. The second method of replacing the

Approximately 22.18% of the 11,381 aneurysms studied in this quantitative meta-analysis have recurred over the span of 2009 to 2017. By analyzing the two proportions of aneurysm recurrence between two treatment methods, stent assisted coiling and unassisted coiling; I was able to determine a statistical significance in the recurrence rates.

Since Arteriosclerosis is caused by high blood pressure, smoking, or high cholesterol, one in three Americans find themselves at risk for developing the disease. (WebMD, 2017)

Diseases of native aortic valves have been linked to mechanical factors, for example, the sclerotic lesions that occur in calcific aortic valve disease occur preferentially in the subendothelial space on the aortic side of the leaflet, a region that is exposed to low shear stress and disturbed

Occurrence of venous aneurysms remains much less common when compared to arterial ones. However with extensive use of cross-sectional imaging, the incidence portal vein aneurysms have increased. Over 210 cases have been described since the first case report in 1956 by Barzilai and Kleckner [6].They represent about 3% [6]of all venous aneurysms and a reported prevalence of 0.43%[7]. There is no correlation between incidence of portal aneurysm and age or sex of the patient [7].

Ascending aorta - rises up from the heart. The coronary arteries branches out from ascending aorta to supply blood to the heart. The coronary artery is responsible for supplying the heart with blood because the heart needs plentiful of supply of blood and oxygen. The heart been a pump made out of living tissue, the blood circulating from the heart cannot meet up with its demand for oxygen, therefore the coronary arteries supplies the heart muscle with blood and oxygen.

Younger individuals with with a mobile and pliable valve, commissurotomy or valve repair is adviced thus relieving obstruction to outflow to a greater extent.

The followingkeyword combinations were used for both databasesand the results combined: “iliac artery occlusion”, “endovascular”, “endoluminal”, “aorto-iliac artery occlusion”, “TASC C”, and “TASC D”. Additional “relatedarticles” suggested by MEDLINE or EMBASE, referencelists of retrieved articles, and reviews on the subject werealso evaluated to identify any additional relevant publishedstudies.

Diseases of the aorta. In: Goldman L, Schafer AI, eds. Goldman's Cecil Medicine. 24th ed. Philadelphia, PA: Elsevier Saunders; 2011:chap 78.