How To Muscle Respiration

The effect of exercise/physical activity on pulse, blood pressure, and respiration rate was tested on biology students. The results of the experiment showed that each of these parameters is affected after engaging in 2 minutes of physical activity. We found that average pulse, mean arterial pressure, and respiratory rate were all higher in males than in females. But, it was also found that women have a longer time to recovery for all these parameters. The reasons for higher pulse, MAP and blood pressure, and respiratory rate could be

In addition a small rise in breathing rate and this is called anticipatory rise, this happens when exercising. The average reading for breaths per minute during exercise is 23-30. This shows that with more blood pumping through the body more oxygen is needed to keep the body at a sustainable rate to help our body create more energy. Our breathing rate will keep increasing until

During exercise there is an increase in cardiac output, which corresponds to an increase in maximal oxygen consumption. With the increase in oxygen consumption, a greater increase in blood flow occurs. This means there is more oxygen circulating in the blood for the tissues to take up. Due to the increase in blood flow, vasoconstriction of arterioles occurs to maintain mean arterial pressure (Bassett & Edward, 1997). This limits oxygen consumption because some of the blood flow is directed to the brain and skin. It is further pointed out that the heart is another limiting factor because it determines how much blood and oxygen are supplied to the muscles especially when blood flow exceeds maximal cardiac output (Bassett & Edward,

In these experiments we talked about the Resting Metabolic Rate and Progressive Submaximal Exercise Testing to determine a subjects’ carbon dioxide production to oxygen consumption. This was done by having examples provided for us to calculate the RMR by using a formula, (X ml/kg/min x weight in kg) / 1000 mL= # L/min, to determine the number of calories burned at rest, as well having subjects perform the standardized treadmill test for 12-15 minutes for the Progressive Submaximal Exercise Testing.

The rate of cellular respiration (and hence oxygen consumption and carbon dioxide production) varies with level of activity. Vigorous exercise can increase by 20-25 times the demand of the tissues for oxygen. Getting excited has that similar effect and also increase the tissues desire for oxygen. This is met by increasing the rate and depth of breathing. It is a rising concentration of carbon dioxide not a

The results of the experiment generally provided evidence in support of the hypothesis. The data collected for the heart rates of the subjects provided strong evidence in support of the hypothesis since Figure 1 illustrates an increase in the heart rates of all subjects during the exercise phase of the procedure. This increase is demonstrated by a general positive slope for the three lines during the exercise phase. The data for the carbon dioxide clearance and oxygen consumption also provided strong evidence in favor of the hypothesis. Figure 2 has a positive slope for all subjects throughout the exercise phase. Figure 3, however, has an initial positive slope for all subjects near the beginning of the exercise, but later levels off midway through the exercise. A potential explanation for this observation is that the body may have a limit on the volume of oxygen that can be inhaled per unit of time, even during physical stress. Another unusual occurrence in Figure 3 is the drastic increase in oxygen consumption for Subject 2 during the recovery phase.

During this lab, various lung volumes and capacities for one subject were calculated for different conditions. These conditions included at rest and during 3 bouts of exercise varying in resistance on a cycle ergometer. Two subjects were supposed to participate in this specific lab, but there were technical difficulties with the computer set up, which only allowed for one individual to complete the entire procedure. The experiment began by having the subject sit down and breathe normally for 3 minutes into the transducer. Then, the participant performed 5 complete breathing cycles and at the end of the last cycle the individual was directed to inhale as deeply as possible followed by an exaggerated exhale. From there, the volumes were measured, including tidal volume (TV), inspiratory reserve volume (IRV), expiration reserve volume (ERV) and residual volume (RV). Next, the 3 exercise bouts at 60 repetitions per minute (RPM) occurred. The first exercise bout was at 0 kg followed by another at 2% and one at 5% the subject’s body weight, respectively. Similar to the resting condition, the different volumes were calculated for the 3 exercise bouts. After gathering all of the different values for the volumes, the capacities were calculated by using various equations.

Using the data from Table 1, calculations were performed to determine how much CO2 was expelled. This data is listed in Table 2. According to the data in Table 2, it is confirmed that more CO2 was expelled after exercising. While exercising, more oxygen is needed in the body for energy. Since more oxygen is inhaled, it makes sense that more CO2 is exhaled.

At rest our body’s energy expenditure results from our basal metabolic rate (sleeping metabolic rate) and our arousal (wakefulness). At the onset of exercise, VO2 cannot initially support or new metabolic demands at sub-maximal level, so an oxygen deficit is created and must be compensated by our anaerobic metabolic systems. As exercise continues, for about 3-5 minutes, our aerobic system syncs in and supplements VO2, and a steady state is achieved. Heart rate increases as exercise increases in order to support an increase in VO2 and the metabolic demands required to pump more blood; this represents and linear relationship. Heart rate and VO2 are also linearly related, as the maintenance of a steady state during exercise is largely dependent on the heart’s ability to stimulate the metabolic activities that result in increased ventilation and a subsequent increase in VO2.

The purpose of this experiment was to determine the subjects’ ventilatory response during incremental exercise. The equipment we used in this experiment was the cycle ergometer, and in addition we had to test the subjects’ ventilatory response during rest and recovery. In this experiment, we have two subjects with different fitness levels, which fortunately make the subject pool in this experiment very diverse. In addition, the purpose of this experiment is to determine the relationship between ventilatory response, ratings of perceived exertion, heart rate, and blood pressure in rest, exercise and recovery and the intensity and the duration of the exercise in this experiment for each subject are equal.

During running, there is an increase in the respiratory rate which is the rate at which one breathes. This is due to the increased metabolism in the muscles brought about by running. Thereby increasing the amount of oxygen that is needed and the amount of carbon dioxide given off. This increases the demand put on the respiratory system which then compensates by breathing faster. At rest, the respiratory rate is at 12 to 20 breaths per min but can increase up to 32 when running. This also explain the concept of hyperventilation during running. Gas exchange in the alveolar-capillary membrane also increased because of increased muscle metabolism. There is an increase in the VO2max, which is “the measure of the body to use oxygen” (Kisner & Colby, 2012) . This means that the amount of oxygen that is extracted from a certain volume of blood is increased. During running, muscle metabolism increases, oxygen is a requirement for muscle

The effects of exercise on blood pressure, heart rate, respiration rate and electrical activity of the heart were assessed. The measurements of respiration rate, pulse rate and blood pressures were noted as described in Harris-Haller (2016). Data was first taken from subjects in a relaxed position and then followed by sets of reading after exercising based on one minute intervals. The data also noted sitting ECG traces from Harris-Haller (2016). The respiratory rate, pulse, blood pressure, P wave, QRS complex and T wave were defined for each subject. The class average was calculated for males and females and graphed to illustrate the results by gender for each cardiopulmonary factor.

Consistent with preceding literature (Jones & McConnell, 1999; Carter et al., 2000; Basset & Boulay, 2000), both men and women showed a significant increase in estimated maximal oxygen uptake on the treadmill ergometer, with a mean estimated VO2max of 50.5±9.4 mL/Kg/min compared to the cycle ergometer averaging a VO2max of 45.5±9.8 mL/Kg/min. The commonly accepted reasoning to account for this difference is that the treadmill running engages a greater muscle mass (the swinging motion of the arms and the stabilization of the core muscles) compared to cycling which predominately uses leg muscles and therefore leads to a greater demand for oxygen supply to skeletal muscle tissue resulting in a greater amount of inspired air and a greater maximal oxygen uptake value (Jones & McConnell, 1999; Carter et al., 2000).

The purpose of this study is to show how exercise allows higher heart rates and more oxygen intake. To accomplish this, two people were chosen to run up and down stairs, and take each other’s resting heart rate after the exercise. They ran up and down the stairs for 1 minute. This was repeated two additional times to have more data to compare. As a result, running up and down stairs did actually increase their heart rates. This means that more oxygen was received because the heart had to work harder to give oxygenated blood to the rest of the body.

As the intensity of exercise increased, so did the rates of the heart and breathing. After a small period of rest, the heart rate and breathing rate both decreased to a point close to their resting rate. This proved the stated hypothesis. First, the hearts average resting rate was recorded to be 76 bpm. The heart is therefore transporting oxygen and removing carbon dioxide at a reasonably steady rate via the blood. During the low intensity exercise (Slow 20) the heart rate increases to 107 bpm, which further increases to 130bpm at a higher intensity level (Fast 20). The heart therefore needs to beat faster to increase the speed at which oxygen is carried to the cells and the rate at which carbon dioxide is taken away by the blood.