How To Write A Case Study Rodenticide

“It profoundly affects the neurological system,” Dr. Jeff Goodall, a veterinarian who runs the Sunnyview Animal Care Center in Nova Scotia, told CBC News. “It can progress to tremors and seizures, and they can go into a coma.”

A variety of mammals have been tested for pathogenicity and other toxic effects of B.t.k. using several exposure routes. No evidence of pathogenicity was found in these experimental animals. However, viable B.t.k. has been recovered in humans up to several months after exposure. A few inconsistent studies were also reported in rats exposed to high B.t.k. levels. Symptoms included lethargy, frequent urination, hair loss and piloerection (hair stands up on end).

Physicians can review the details of the poisonous substances, animal and human exposure and use this information to provide improved patient care services. The website includes all pediatric exposure and this information can educate parents to protect from different poisonous exposure (June 2013 GPC). The GPC maintains a database to provide better understanding of the poison related issues by educating GA residents and healthcare professionals. Healthcare providers can be aware of any drug that probably has caused poisonous effect of consumers by referring to the GPC website. The GPC had received an average of more than six thousand poison related calls each month during the 2014. The GPC website and toll free number for anyone who wants to have more improved knowledge about poison related issues (June 2013

Symptoms of pesticide poisoning are often deceptively misdiagnosed as flu or allergies. Other problems associated with the use of chemicals include headaches, nausea, fever, breathing difficulties, high blood pressure, and temporary paralysis. The National Academy of Sciences reports that at least one out of seven people are significantly harmed by pesticide exposure each year (American Defender Network, 1989). Unfortunately, due to the high costs of inspection, tests for pesticide poisoning are rarely performed.

Before that the animal can be seen drooling or frothing at the mouth says The Merck Veterinary Manual. A quickening heart that becomes weaker, the muscles will have tremors and spasms, the last spasm is characterized by an arched back with the head tipped back and heels bent backward (in humans it is the stereotypical movie position for exorcisms, the scientific name is opisthotonos). Like any animal they try and fight going down till the very end because it means the struggle for life is almost over, so they can be seen struggling and staggering trying not to fall, but eventually lose the battle because they have little oxygen left in their tissues to keep the tissue living and supporting their weight. For the livestock that have been poisoned it is not a quick and painless death because it can take up to 45 minutes to suffocate pretty much. In my first three sources each one said that the mucous membranes (“a thin, wet layer of skin that is inside some parts of the body (such as the nose and throat) and that produces mucus”-Merriam-Webster), would become bright cherry red which is a signal that cyanide is present. The heart

After eating it the early symptoms include headache, dizziness, fast heart rate, shortness of breath, and vomiting. This may then be followed by seizures, slow heart rate, low blood pressure, loss of consciousness, and cardiac arrest. Onset of symptoms is usually within a few minutes. If a person survives, there may be long-term neurological problems. Like most cyanides, it is one of the fastest working toxins in the world. An oral dose as small as 200-300 mg can kill you.

Symptoms include: excessive perspiration, constriction of the pupils, tear production, salivation, abdominal cramps, diarrhea, nausea, vomiting, chest tightness, difficulty breathing, muscle weakness, muscle cramping, ataxia, and paralysis and may cause blood pressure changes with either decreased or increased heart rate. Effects on the central nervous system can also cause symptoms such as: headaches, confusion, decreased respiration, convulsions, and coma. These symptoms have been studied but symptoms have occurred only after extremely high or lethal doses of malathion were administered. In studies done with lower doses of malathion, no effect level was observed. The effect of malathion on human health and the environment depend on how much is present at the time of exposure as well as other environmental factors. When administered properly at low levels, no effect was

The patient that was presented in the ER had a severe headache, unusual degree of sweating (diaphoresis), inability to produce or control body movement (motor dysfunction), abnormal sensation with no apparent physical cause (paresthesia), feeling of sickness with the urge to vomit (nausea) , and an ascending paralysis that spread to the upper body, arms, face, and head along with a bluish discoloration to the skin (cyanotic), inadequate ventilation (hypo-ventilating) and also developed a slow heart rate (bradycardia) with a BP of 90/50. The diagnosis was still unclear at this time with an un-known medical history but atropine was administered to treat for the bradycardia. There was a diagnosis of tetrodotoxin poisoning based on the current symptoms therefor a hydration IV, a stomach pump (gastric lavage), and activated charcoal were administered to treat the symptoms. Within a few hours the patient’s condition improved greatly and he was on his way to a full recovery from the effects of the poisoning with a BP of 125/79, HR 78bpm, and oxygen saturation of 97% on room air, “the fraction of the hemoglobin molecules in a blood sample that are saturated with oxygen at a given partial pressure of oxygen. Normal saturation is 95%-100 %”( medical dictionary, 2009).

Nine mice had parts of their tail clipped of and the drug was put on the tail. Seven out of the nine mice stopped bleeding within ten minutes. The drug was tested on a more severe injury by surgically punching a hole in the femoral arteries of pigs. With no treatment all the pigs bled out and died in less than three hours. All pigs treated with the drugs survived.

5. (You really shouldn't, but if you have to at least) Choose a first-generation rodenticide over a second-generation one because second-generation rodenticides "kill slowly, so rodents keep eating them long after they've ingested a lethal dose," says

Nerve agents are the deadliest chemical warfare agent that needs immediate medical attention. All patients who experience more than miosis should be triage “immediately” category and decontamination “immediately.” The application of antidotes, airway control and other supportive measures” should be applied. If the patient has more than two systems such as “miosis and wheezing, the patient should receive treatment immediately. Quick response result into a successful resuscitation for these individuals (Keyes,

Sufferers will first feel nausea, then dizziness, confusion, loss of co-ordination, rapid deep breathing and gasping, followed by muscle spasms and loss of consciousness; provided they are exposed to high enough levels(Which is not that high).

Medical effects of soman are typical of nerve agents, and likewise varies depending on the extent of exposure. Specifically, GD is more volatile than VX, but less volatile than sarin (CDC 2013). While the severity and timeliness of symptoms depend on the exposure, the symptoms include: change in blood pressure, blurred vision, chest tightness, drowsiness and confusion, cough, diarrhea, excessive drool and sweating, eye pain, headache, accelerated breathing patterns, nasal and optical discharge, and weakness (amongst others) (CDC 2013). Exposure requires immediate medical treatment. Lower to mild levels of exposure are generally treatable, whereas severe exposure levels are likely to induce death. Exposure can be the result of inhalation, cutaneous contact, and ingestion.

Humans weren't the only ones which were effected by this syndrome. What were once battle fields for the Gulf War, were later used for agricultural purposes. Sheep particularly were raised in these areas. Many sheep died mysteriously, and other sheep became very sick. To prevent more sick sheep, agricultural organophosphates are carefully established on the known degree of toxicity of each compound. The organophosphorous used is Bayer 21/99, which is a popular compound that inhibits treated animals from the skin and digestive tract. Sheep are also more susceptible to poisoning then cattle and most other emaciated animals. Pre-treatment consisted of weighing, ear tagging, and the collection of blood samples for a baseline red cell count. It is shown that animals which have been treated have a much higher tolerance to poisons and a less likely to have more ailments. Symptoms include: Irregular gait with generalized muscular weakness and severe diarrhea. Slight increase in salivation and isolated diarrhea. Irregular gait with generalized muscular weakness and severe diarrhea.

In this test the chemical being tested if force fed to rats or mice in small amounts until the dosage is in excess. This is continued until at least 50% of the test subjects die. Although the test rodent may not die right away, it may experience seizures and internal damage. This test is considered to be the most cruel of the tests and can only determine how much of a chemical substance is needed to kill a small animal not a human being.