A person’s genetic make up has a significant influence on whether the person will become obese or not. If both parents are obese, the likelihood that their children may end up being obese too is higher compared to a situation where neither of the parents is obese or where only one of the parents is obese. This is particularly so because genetics influence the way the body stores energy and how energy is used. This can be seen in the differences that have recorded in the basal metabolic rates (BMR) among groups of people who differ by age, gender and the make up of their bodies. People who have a low metabolic rate have a higher risk of becoming overweight. The genetic similarities shared by members of one family can explain why people who come form certain families end up being overweight (DeBruyne, Pinna
There have been many theories brought by research to best explain the cause of obesity and one major possible causes is genetics. The chances of a person being obese are much greater if both parents are overweight or obese. Genes can influence how much body fat we have, where we tend to store our fat and even the types of tastes we prefer. Family members not only share genes but also diet and lifestyle habits that may contribute to obesity. Research points to heredity as a strong determining factor of obesity. In one study of adults who were adopted as children, researchers discovered that the weight of the adults was closer to their biological parent’s weight than their adoptive parents. The environment provided by the adoptive family had less influence on the development of obesity than the person's genetic makeup. A clear pattern of inherited obesity within a family is caused by a specific variant of a single gene, called monogenic obesity. Although inactive lifestyles contribute to the approximately 10-pound average weight gain of Americans compared to the recent years, some people are heavily affected by this lifestyle than others. The reason is that they have inherited genes that increase their predisposition for gaining body
Obesity is measured using the Body Mass Index formula, this calculates the body fat by dividing body weight in kilograms to determine if an individual is a healthy weight range for their height (Australian Institute of Health and Welfare [AIHW], 2004). Childhood obesity can occur from eating large amounts of unhealthy and high energy dense food and drink or a lack of physical activity or a combination of both, is then stored as fat as they are not burning enough energy, other contributing factors to weight gain include a child’s race, ethnicity, socio-economic barriers, environmental influences, the lack of parental health knowledge and guidance and genetics (Berkowitz and Borchard, 2009). Obesity has a strong genetic
Child obesity is alarmingly prevalent in the United States today; approximately 18 percent of adolescents are considered obese. Obesity is defined as “one of the most pervasive chronic diseases...that affects more than one-third of the population” (Source 2). While obesity may be a major challenge facing adults in the United States, adolescents are facing a similar struggle. More than 33 percents of all children and teens suffer from obesity. But why is child obesity such a major problem in the United States? Though obesity has been shown to have some genetic inheritance between parents and offspring. Adolescents are regularly eating fatty and unhealthy foods, and are not getting the
Genes control almost every aspect of human life, and when it comes to weight there is no exception. Little is known, however, how much of an individual’s genes actually control the weight of certain individuals. Body fat can vary from person to person, yet some people have always carried more weight than others. Often times, when one person is overweight in the family, most of the other individuals in the same family are overweight as well. However, more than just genetics can go into being overweight, and a person’s genes are not the end all be all of obesity. Many times the environment surrounding people who are obese contributes to the overall weight of the individual, and when obesity promoting genes are mixed
Childhood onset overweight and obesity and its’ associated health consequences are quickly becoming major significant public health issues facing America today. Centers for Disease Control and Prevention (CDC) define overweight as a body mass index (BMI) between the 85th and 95th percentile while obese is defined as BMI above the 95th percentile for children of the same age and sex . The prevalence of overweight children, defined based on 2009 CDC’s National Center for Health Statistics data, has more than tripled in the past 30 years. Between 1980 and 2006, the incidence of overweight among children aged 6 to 11 years increased from 6.5% to 17.0% while overweight levels for adolescents aged 12 to 19 years increased from 5.0% to 17.6% .
The Physiological Theory of Obesity draws a distinctive link between overweight parents, and the possibility of their children being overweight. It has been recorded that if one parent is obsess, then the child of that parent has a forty percent chance of being obese. If both parents are obese, the change of the child being obese jumps to 80 percent. The clear connection between a parents’ weight and a child’s chance of becoming overweight is alarming. While one cannot infer causation from correlation, the statistics may point to the
Childhood obesity can lead to a life long struggle with obesity. The National Institutes of Health discovered that “The percentage of overweight Children has increased, by almost 50% in the last two decades of the 20th century… It has also been observed that about 40 percent of overweight children will continue to have increased weight during adolescence and 75-80% of obese adolescents will become obese adults” (National Institutes of Health). This proves that the increase in childhood obesity has led to an escalation in obesity in adults.
The researchers recruited two thousand three hundred preschoolers for their study and acquired the student’s height and weight in order to solve their body mass index (BMI). The BMI of a person is the estimate of excess fat on their body, but doesn’t take in to account their muscle mass. At the same time the children’s BMI were being taken, the parents were also asked for their height and weight in order to attain their BMI. The researchers found that children with one obese parent are twice as likely to be obese and children with two obese parents are two point four times more likely to be obese (Overweight Parents More Likely to Have Overweight Children). This study directly proves that parents have a huge impact on their children when it comes to their
Obesity in America is a developing issue, and not simply in grown-ups. Today, around one in three American children and teens are overweight or fat. Young obesity has rapidly turned out to be a standout amongst the most genuine wellbeing difficulties of the 21st century. Frequently, obesity is the aftereffect of an imperfect way of life. Despite the fact that hereditary qualities
Childhood obesity is a major problem in the United States. There are multiple reasons for this problem including the child’s school system, the access to technology in young children and the child’s genes. It is sometimes inevitable for a child to be obese due to their genes. There are some precautions being made to help this problem. Some might work but others might fail. Childhood obesity is a major problem and needs to be fixed because it causes major health issues for the child in the future.
In the United States, Healthy People 2020 tracks children aged 2 to 19 that carry a BMI greater than 95% per the Centers for Disease (CDC) pediatric growth chart. The goal is a target rate of 14.5%, with a current improvement of 10% over the baseline. There are disparities related to gender, ethnicity and race, therefore this is very important public health problem and for the future of our country and health care of our citizens. Obesity is of epidemic proportions so there was a plethora of information found at South University’s library site of which will be reviewed in this paper.
Introduction: In 1998 the World Health Organization announced that obesity had become an epidemic in both adults and children. This epidemic is a major concern in public health yet many countries are failing to address the issue. The purpose of this paper is to discuss the causes contributing to this increasing epidemic of childhood obesity. There are many causes that factor into childhood obesity but I will focus specifically on genetics, overeating, and the lack of exercise.
Childhood obesity is becoming more of a medical concern everyday. With obesity rates up almost 10% since the 1970’s and it being the second leading cause of death in the United States it is becoming a problem that has lifelong consequence (Johnson 1 ). The cause of childhood obesity is more than just genetics, it is what influences the child from family and friends to the social aspect such as television. Although this is becoming an epidemic in the United States, there is still hope for a better future. Childhood obesity can be influenced by social factors and lead to severe medical issues but can be aided with the help of a lifestyle change.
Everybody knows the child that can eat any type of food all day and never seem to gain weight. This occurrence has led researchers to investigate the role that genetics plays in childhood obesity. Not all children who are inactive or who eat poorly are obese, much in the same way that some obese children eat fairly healthy, and exercise moderately. Heredity has recently been shown to influence body fat percentage, regional fat storage, and the body?s response to overeating (Rush, 1). Children who have obese parents are 80% more likely to be obese than their lean parented counterparts (Buffington, 16). This familial correlation is contributed to genetics as well as the parents eating habits. Children with obese parents typically aren?t taught the correct way to choose when and what food to eat, leading to poor eating habits and eventually obesity. Many genetic defects can have a significant effect on obesity such as variable thyroid activity and pituitary defects. Abnormalities in any one of these regulators could be responsible for appetite abnormalities and weight gain. Furthermore, obesity leads to defects in appetite regulation, hormone production, and metabolic events (Oklahoma Cooperative, 4) that are responsible for further weight gain,