Hypoxia: Procedures Don Oxygen

Hypovolemic shock is an urgent condition of rapid reduction of circulatory volume in the body, which can be created due to blood or plasma or body fluids loss (Kettley & Marsh, 2016, p. 31; Perner & Backer, 2014, p. 613). Blood loss can be induced by internal or external injuries, excessive perspiration or diuretics (Craft & et al, 2015, p. 852). Maureen Hardy’s hypovolemia has been precipitated by hematemesis.

It results when one or both of these gas-exchanging functions are inadequate . It is not a disease but a symptom of an underlying pathology affecting lung tissue function, 02 delivery, cardiac output, or the baseline metabolic state. It is a condition that occurs because of one or more diseases involving the lungs or other body systems. Symptoms of this is hypoexia and hypercapnia.

A low partial pressure of oxygen (PaO2) suggests that a person is not getting enough oxygen; Metabolic acidosis->Kidney failure, shock, diabetic ketoacidosis

Anemia is a disorder of the blood. It occurs when your body does not produce enough erythrocytes or red blood cells (RBCs). Without the erythrocytes oxygen can not be adequately delivered to the tissues and organs throughout the body. This will cause you to become weak and tired. A person may also experience headaches, skin pallor, and faintness. Your body may attempt to compensate for these symptoms by speeding up the heart rate and respiratory rate. This is the body’s attempt to return oxygen levels to normal(Thibodeau and Patton, 2005).

2. What might put desert-dwellers in danger of developing hyponatremia? How can they avoid this danger?

Both rapid, shallow breathing patterns and hypoventilation effect gas exchange. Arterial blood gases will be monitored and changes discussed with provider. Alteration in PaCO2 and PaO2 levels are signs of respiratory failure. Patient’s body position will be properly aligned for optimum respiratory excursion, this promotes lung expansion and improved air exchange. Patient will be suctioned as needed to clear secretions and maintain patent airways. The expected outcome is that the patient’s airway and gas exchange will be maintained as evidence by normal arterial blood gases (Herdman,

Cari’s states, “I feel like I’m panting like a dog, and my chest hurts” Her shortness of breath, fatigue and headache are all results of the lack of oxygen exchange to her tissues. The pH of her blood was rising due to the hyperventilation also.

Hypoventilation is breathing at an abnormally slow rate, resulting in an increased amount of carbon dioxide in the blood.

The clinical symptoms that will be seen with these patients are hyperthermia, tachycardia, tachypnea, hypertension, aggression, and agitation. Sometimes, these patients have been in this state for so long they may be acidotic or entering the early stages of rhabdomyolysis. These patients will have “super human” strength, appear as if nothing hurts them and they feel no pain, extremely diaphoretic (and often times naked due to hyperthermia), be extremely combative, and have what appears to be a complete disconnect from the outside world. Being in contact with these patients is extremely dangerous for EMS providers as they are so altered, they have no idea what they are doing and it is reported that they often times have paranoia and think that they are fighting for their lives.

The decrease in her PCO2 and pH will cause her central nervous system to slow down causing her breathing to slow down to try to give her body more carbon dioxide to level out the amount of oxygen/carbon dioxide ratio.

The altitude that this stage could happen in is between 5000 to 11400 ft.(3) "The body generally has the ability to stave off further effects of hypoxia by increasing the rate and depth of ventilation and cardiac output ".(1) The respiration rate, blood pressure, and the heart rate can rise up in this stage.(2) the arterial oxygen saturations in this stage is between 80 and 90 percent.(1)

Hypoxaemia can result when there is inequality in alveolar ventilation and pulmonary perfusion (V/Q mismatch). V/Q mismatch is the most common cause of hypoxia in critically ill patients. It is caused by intrapulmonary shunting of blood resulting from airspace filling or collapse. Findings include dyspnea and tachypnea. Diagnosis is by ABGs and chest x-ray. Treatment usually requires mechanical ventilation.

After being reminded by the instructor, I was aware of my mistakes and noticed that I failed to maintain patient’s safety. An oxygen below 90% can be very dangerous for the patient, especially for a post-op day #1 patient, because prolonged hypoxemia can cause fatigue, headache, acute respiratory failure, cardiac problems (increased heart rate,

55). According to Wen-Chih et al. (2010, p.11), ageing patients are susceptible to blood loss since they have limited physiological replacement. Thus, considering the age and the surgical procedure the patient is at high risk of hypovolemia. If the loss of blood progresses and the body is unable to compensate, the patient will be at risk for hypovolemic shock (D’Angelo, M., Dutton, R., 2009, p. 279). Hypovolemic shock means deficit in flowing blood causing to ineffectively filing the intravascular space. Mamaril, M., Child, S., & Sortman, S., 2007, p. 191. Kolecki, P., & Menckhoff, C. (2014) emphasizes that there are four (4) classification of hypovolemic shock. Class 1 is when there is 0-15% of blood loss with only slight change in heart rate. If the patient heart rate becomes more than 100 beats per minute and they experience tachypnoea and has cool clammy skin, the patient is in the class 2 of hypovolemic shock. Class 3 and 4 is when the patient will manifest the severe sympathetic response of the body due to the inability of compensate with the blood loss. Thus, considering the age and the surgical procedure the patient is at high risk of hypovolemia or even the more severe

In most tissues of the body, ATP production primarily occurs through mitochondrial oxidative phosphorylation of reduced intermediates, which are in turn derived from substrates such as glucose and fatty acids. In order to maintain ATP homeostasis, and cellular function, the mitochondria requires a constant supply of fuels and oxygen. In many individuals at altitude, tissue oxygen levels fall and the cell must meet this hypoxic challenge to maintain energetic and limit oxidative stress. Varying on protocols, the body can adapt to the lack of oxygen which can be increasing the mass of red blood cells and haemoglobin or changes in muscle metabolism. Depending very much on the protocols used, the body may adapt to the relative lack of oxygen in one