Whilst studying the current research done on Kawasaki Disease, what was never emphasized was the cause of the disease. The research is heavily focused on the two most common cures along with the diagnostics. Although this is quite helpful, the focus now should be directed towards preventive studies instead of different treatments. Kawasaki disease (KD), also known as Kawasaki syndrome, is an acute febrile illness of unknown etiology that primarily affects children younger than 5 years of age. The disease was first described in Japan by Tomisaku Kawasaki in 1967, and the first cases outside of Japan were reported in Hawaii in 1976. It is a disease of unknown cause, occurring primarily in young children and giving rise to a rash, glandular swelling, …show more content…
A rash on the mid-section of the body and in the genital area. Red, dry, cracked lips and a red, swollen tongue. Red, swollen palms of the hands and soles of the feet. And redness of the eyes. The testing needed to properly diagnose this is to get an echocardiography, a painless test uses sound waves to create pictures of the heart and coronary arteries. Other tests include, an EKG, blood work, and a chest x ray. These tests, the procedure, will take a short amount of time; but to analyze the blood work may take a day or two. Those involved with the examination would be Pediatricians often are the first to suspect a child has Kawasaki disease. Pediatricians specifically. If the disease has affected your child's coronary (heart) arteries, a pediatric cardiologist will confirm the diagnosis and give ongoing treatment. Pediatric cardiologists treat children who have heart problems. Other specialists also may be involved in treating children who have Kawasaki disease. Although, there may not be a sudden conclusive diagnosis because the symptoms overlap with Rocky Mountain spotted fever, scarlet fever, and juvenile rheumatoid arthritis. Therefore, the patient must have all (or most) of the beginning symptoms as mentioned …show more content…
According to Medscape, “Aspirin has traditionally been part of the standard treatment of this disorder. Although some authors have suggested that aspirin is no longer needed, most experts use high-dose aspirin for a variable period, followed by lower-dose aspirin for its antiplatelet effects. Aspirin is used in patients with small coronary artery aneurysms (CAAs). Dipyridamole is indicated in patients with larger CAAs,” so aspirin is the more common, usual approach, but in some more severe cases, they use an IV. “The appropriate treatment of patients who fail to respond to IVIG remains unclear. A severe Kawasaki disease that is resistant to IVIG may benefit from intravenous pulse corticosteroid therapy or infliximab infusion,” as Medscape further explains. It’s not as common, but it does treat it with the more severe cases. Treatment will typically start with either the right diagnosis or right as the fever breaks. Less people are involved with the treatment than with the diagnosis. Now, it’s just the pediatrician and family. With the IV fluids, it may still remain in the child’s system for many many years. Thus causing some blood testing to come back “wonky” such as being tested for mono. It may not show up as positive or negative because of what was in the IV bag. The medications won’t affect the patient after the treatment, the only lasting effects may be
As listed in Wong the “cardinal symptoms of cancer in children are unusual mass or swelling, unexplained paleness and loss of energy, sudden tendency to bruise, persistent, localized pain or limping, prolonged, unexplained fever or illness, frequent headaches often with vomiting, sudden eye or vision changes and excessive, rapid weight loss.” When completing an assessment of the child the healthcare work should be able to incorporate these signs to help them complete the necessary physical assessment and also lab or diagnostic testing.
Diagnosing the disease can be relatively simple, but this depends on accurate patient history and ones ability to recognize varied clinical signs and then perform diagnostic procedures. These diagnostic procedures may include antigen testing, x-rays, ultrasonography, angiography, a complete blood count, the Knott’s or Filter test, and in the worst case necropsy.
A 32-year old nurse who has rheumatic fever as a child noticed a persistent tachycardia and light-headedness. Upon examination, chest x rays showed an enlarged left atrium and left ventricle. ECG analysis showed atrial fibrillation. There was also mild pulmonary congestion. Cardiac evaluation resulted in the following information:
The treatment of patients with mild disease consists of acid suppression (esomeprazole) and a short course of steroids (prednisolone). Those patients who have moderate to severe disease should be treated aggressively in the following way: acid suppression (esomeprazole), a longer course of steroids with prednisolone, and consideration of immunosuppressive therapy with azathioprine. Infliximab should be considered in refractory patients in order to prevent the complications of stricturing and fistula formation. Treatment with balloon dilatation of the stricture followed by injection of a long-acting steroid such as triamcinolone can also help to alleviate symptoms. Surgery may be required for severe, refractory symptoms, but it has a high morbidity
The paper attempts to find any correlation/connections between Hirschsprung Disease and Congenital heart disease. This was done by identifying children with Hirschsprung disease, which is a disease that causes the colon of the affected person to infllamme. Hirschsprung is Congenital,present at birth, and becomes an issues as the child mature, since passage of stool becomes difficult. This issue can be corrected but issues can arise when dealing with other diseases such as congenital heart disease. Some patients cases seem to imply that in attempting to solve the issues the patient is then affected long term. The study took place in a pediatric surgery center, in which 53 out of 61 children were chosen to take place of the study. All of these
Meningitis can be destructive without proper understanding of what it is caused by. There are three types of meningitis- bacterial, fungal, and viral. Contrasting factors tend to arise during a comparison of them. One of the most notable areas that viral, fungal and bacterial meningitis differ in are their treatment ability. However, they have the same general affects on the human body. In any case, there are tests that doctors can utilize in order to discover if the meningitis is bacterial, fungal, or viral.
When looking into treatment for Kawasaki disease, I found that most of the information was relatively the same. The initial treatment calls for “high dose” aspirin, which acts as an anti-inflammatory and the IVIG is an immunoglobulin that assists your immune system. The anti-inflammatory effects of aspirin are used to also to prevent the formation of a clot.
In mild cases, treatment is usually not needed. In severe cases, your child may need to be given red blood cells through an intravenous (IV) line (packed red blood cell transfusion).
Guillain-Barre Syndrome (GBS) is a disorder where your own immune system starts to attack part of the nervous system, mainly the peripheral nervous system. The peripheral nervous system is a very important system in the body it is what contains all the nerves that are not directly connected to the brain and spinal cord. The main function of the peripheral nervous system is to connect the central nervous system to the rest of the body, such as the organs and limbs. Beginning signs that you may have Guillain-Barre Syndrome is that of noticing varying degrees of tingling and weakness in the legs, most people throw this sensation off for restless leg syndrome so they never go to get diagnosed for the disease. Guillain-Barre syndrome can affect
The patient will need to be hospitalized for immediate treatment to prevent coronary disease, as the disease can cause heart disease (Starkebaum, 2015). Treatments will include intravenous gamma globulin along with aspirin. Most children will recover from the disease. However, some children will die from heart disease secondary to the Kawasaki disease; therefore the child should have an echocardiogram every 1 to 2 years to assess for coronary artery disease (Starkebaum, 2015). The parents will be advised to call their health care provider if the child experiences recurrent symptoms of swelling and redness in the palms, reddened eyes, and high fever that does not resolve with
“Rasmussen’s Encephalitis is a rare chronic inflammatory neurological disease that usually affects only one hemisphere of the brain.” (What is Rasmussen’s Encephalitis: http://www.ninds.nih.gov/disorders/rasmussen/rasmussen.htm). It is more commonly found in children 10 years of age or younger, and is described by seizures, loss of speech and motor skills, hemiparesis, inflammation of the brain, and mental deterioration. Patients with Rasmussen’s Encephalitis will have progressive brain damage in the affected hemisphere, and is accompanied by seizures. Rasmussen’s Encephalitis will have permanent, but stable neurological deficits by the first 8-12 months of diagnosis. There are currently minimal treatments, but the progression of the research
29. What is the text mainly about? A. Vaccines for children B. Fake news about vaccines C. The fear of being vaccine D. The importance of measles-rubella vaccines E. Attempts to apply measles-rubella vaccines 30.
Your child's health care provider can diagnose this condition based on your child’s health history and symptoms. The health care provider will collect reports from parents and teachers. Your child may also have tests to confirm the diagnosis or to rule out other conditions. These tests may include:
Encephalitis is a condition caused by viruses which cause the brain to become inflamed. There are two types of encephalitis one is called primary because the viruses affect the brain itself. Secondary is the viruses travel from some other part of the body that has been affect to the brain. When the virus reaches the brain it begins to multiply causing inflammation. The brain’s white matter can be destroyed. This destruction causes cell death, hemorrhage and edema. The edema begins to compress the blood vessels this causes intracranial pressure (Mayo Clinic, 2011).