MMI Scm Personal Statement

If selected for admission, I plan to take advantage of IMSA’s unique learning opportunities by working diligently in all of my classes, joining clubs and teams to learn from my peers, and participating in Student Inquiry and Research to satisfy my curiosity and allow for more independent work. I come from a small high school with approximately one hundred students per class. Funding is a constant problem and learning opportunities are thus very limited. I’ve attempted to take as many science and math classes as possible, as well as world history, foreign language, and an advanced English course, but I still do not feel challenged. My school has done what it can to enhance my learning experience, but my math and science classes aren’t quite

Experiencing hardships and my resilience to overcome them is the main reason I chose to pursue a Master’s of Social Work (MSW) degree. My familiarity with asking for resources, as a former foster youth, a number of my interactions involved social workers. I utilized resources as a transition aged youth; and I believe it was those same resources that kept me focused on my future and school. I became homeless at eighteen years of age and it was transitional housing, through The Department of Children and Family Services (DCFS) that gave me hope and direction.

I would love to be a part of Central Michigan's athletic training program. I have a deep love and passion for sports. After High School I would still want to be in the sports field and I also like caring for people which would make Athletic training the field for me. I am currently a captain on my basketball and track team. I make others around me better and take on the role of being a leader. My goal is to graduate and get my degree. After that I envision myself being a strength and conditioning coach or an athletic trainer. I want to be a good athletic trainer at a college or pro level.

I discovered Loma Linda University School of Medicine through a friend who urged me to apply knowing how much of a devout Christian I am. I am aware that LLUSM educates its students in the teachings of Christ, and teaches them to heal patients and make man whole as Christ did. This in itself requires an understanding of Christianity and its application to medicine as a career. As a future medical student, I recognize that I will be required to act according to these Christian ideals, a privilege that I look forward to. I admire LLUSM due to its ability to spread love to its patients and inform them of Christ’s ministry. I wholeheartedly agree with the philosophy; healing a patient is much more than treating the symptoms.

My aim is to pursue a job in the quality improvement department. I realize that when I am in this position, I should ensure the institution is in compliance with the recommended quality standards of the accrediting bodies. This is a team effort that needs the participation of all the employees. Hence, I would encourage the teamwork of the staff to achieve this goal. I would provide the clear idea about the goals and the timeframe to meet it. The team activity would let the staff in the team assume the responsibility to achieve the goals with a unified spirit (Axner, 2015). A good team needs the appropriate team members. They should have a diverse background, expertise and experience so that the input of one person can supplement that of the

The Mississippi School for Mathematics and Science has been called the most diverse square mile in Mississippi. Before coming to MSMS I had never seen other children my age who were Asian, Indian, or any other race except for Caucasian and Mexican. I didn't have a chance to talk to other children about their cultures or beliefs until I came to MSMS. Being at MSMS gives me the opportunity to speak to one of the most diverse student bodies in Mississippi. I am able to listen to the different languages spoken by the children here such as Vietnamese, Korean, Hindi, and Spanish. I also get the chance to join cultural clubs established by the student body. Currently, I am a member of the Asian Student Association. The ASA meets monthly to discuss

To seek admission into the MFA–Acting program at Yale School of Drama to transform myself into a lifetime actor by working on my physical and vocal instrument at the world-class academy, learning acting techniques, scene study, voice, text, speech, dialects, singing, particularly Alexander technique, in a collaborative, creative and diverse environment.

There is nothing quite like being from a former Soviet Union member state to instill a healthy respect for the need for democratic law with a solid foundation of juris prudence. Understanding the struggles my ancestors had gone through, in stories by word of mouth among the harsh rule of communist leaders, had solidified an interest in the methods America took to evolve into a “land of opportunity” for all. The majority of my family immigrated to America, all expressing a strong interest in its cosmopolitan life-style, made true by the democratic government instilled in the political system. Therefore, my curiosity in the political system stems from my ancestors wish to leave home and start new lives in America.

Through the study they found that the rate of aging may be set very early in life before reproduction ends. They found that by finding that genetic mutations and environmental factors early in life affected the day-3 mitoflash frequency, which they found to predict lifespan. The mutations from the mother in the mitochondrial DNA were also found to have a correlation with litter sizes, and a decrease in total number of liters. The authors also found that a relatively low amount of maternally transmitted mitochondrial DNA mutations can induce the premature aging phenotypes. The mutations in the mitochondrial DNA resulted in many things such as, alopecia, kyphosis, reduced body size, lower body weight, and decreased spontaneous rearing. To try and help prove their work the authors talked about the literature that was already on the topic of DNA damage in time exceeds what the body can repair, leading to manifestations of aging in many

My interest to pursue post-baccalaureate study is driven by my passion to become a future leader in my greater community, and I believe San Francisco State University’s Master of Public Administration (MPA) program will enhance my experience in leadership skills, such as, critical thinking, productive collaboration, effective completion of tasks, and powerful communication. The fiery passion that is engrained in me sparked early in my high school career when I volunteered for the late San Francisco Mayor Edwin Lee with his mayoral campaign in 2011. By walking the precinct and hanging signs around the Richmond neighborhood, this elementary form of public service began to pique my interest as a potential career path. Having experienced different

We have worked together not for a long time and I may miss some positive qualities of Ms.Ago. However, I am pretty sure that she has a quality of carrying out the academic and life goal of her own.

Despite the heterogeneity between different diseases, it has been widely shown that a common inner mechanism of ageing lies on mitochondrial dysfunction for it plays an important common role in the defect of respiratory chain dysfunction and the onset of age-related diseases

Aging is a multifactorial process in the cell that cell survival is decreased and normal physiologic activities of tissues and organs may reduce. In this process, cells are prone to various diseases which may eventually lead to cell death.1 Aging is predisposed by various factors, including genetic damage, increasing in DNA methylation, abnormal changes in proteins, increased ROS, dysregulation of immune diseases and increasing in inflammatory cytokines.2, 3 Despite of scientific progress, cell aging is a controversial topic and the exact cellular and molecular mechanisms of aging and age-associated disease such as cardiovascular diseases (CVD), cancer, arthritis, dementia, cataract, osteoporosis, Alzheimer’s disease, diabetes and hypertension

Saccharomyces cerevisiae more commonly known as “brewer’s yeast” is simple yet elegant model organism. Thanks to modern science the yeast genome in it’s entirely has been mapped out and because it is a eukaryotic organism knowledge gained from their lifespan can be easily applied to larger and more complexed organisms. Because their genome is known and because they can be easily manipulated, is exactly why Kaeberlein lab uses them for their scientific research. Kaeberlein’s lab focuses on age related disease and potential remedies for fixing them. My work within Kaeberlein lab is primarily focuses on studying the replicative life spans of yeast.

Age is a universal time-dependent deterioration factor that affects all organisms and their cellular functions, thus, determining their lifespan. Actin organization and function declines with age in many cells, tissues and organs. For example, age-associated declines in myosin and actomyosin ATPase activities, changes in myosin structural state, and oxidative damage to actomyosin occur and may contribute to sarcopenia [1]. Similarly, the age-associated deficit in the motility of fibroblasts, which contributes to impaired wound healing, has been linked to a progressive decline in actin organization [2]. There is an age-associated decline in immunological synapse formation between CD4+ T cells and the antigen presenting cells (APC) in which actin cytoskeletal structure and dynamics are critical for this formation and activation of T cells (6 and 14 – CS). Defects in T cell activation are shown to be a result of defects in recruiting actin binding protein talin, which is critical for reorganization of the actin cytoskeleton, and development of lamellipodia, which is responsible for increasing the communication surface area between CD4+ T cell and APC at the immunological synapses (6 and 14 – CS). Finally, actin cytoskeleton integrity declines in Alzheimer’s patients affecting the integrity of dendritic spines by disrupting its morphology and impinging memory and learning (20n21 - CS). In Alzheimer’s patients there is a disproportionate expression of cofilin,