Medical Term Shock Essay

How is Toxic Shock caused? Well its is caused by many ways. The first way could be cause is by a bacterial infection. The other way it could be caused would be your body might produce a little much of Staphylococcus Aureus bacterium. Another way could be the use of Tampons for women. The Tampon can scratch the vagina creating an opening for bacteria to get into your bloodstream. The last thing could be women who are menstruating or just had a baby.

Hypovolemic shock is an urgent condition of rapid reduction of circulatory volume in the body, which can be created due to blood or plasma or body fluids loss (Kettley & Marsh, 2016, p. 31; Perner & Backer, 2014, p. 613). Blood loss can be induced by internal or external injuries, excessive perspiration or diuretics (Craft & et al, 2015, p. 852). Maureen Hardy’s hypovolemia has been precipitated by hematemesis.

The purpose of this study was to determine the type of learning acquisition in dogs that were subjected to three different styles of electric shock. They wanted to determine what method of learning worked the best to avoid a shock for an extended period of time. Each of the three groups of dogs learned escape/avoidance training, however the "escape" group and the "yoked" group gained more training than the normal control group. The "escape" group was taught during their training that touching the side panels during the shock would terminate it. This was repeated 64 times in the harness and the same training was done 10 more times in the shuttle box, 24 hours later. The "yoked"

Cardiogenic shock is a frequently fatal complication that occurs when the heart cannot pump an adequate amount of blood in order to perfuse tissues. This hypoperfusion causes multiple organ dysfunction and damage which classifies cardiogenic shock a medical emergency. In the past, cardiogenic shock had a poor prognosis. However, currently approximately half of the people that go into cardiogenic shock survive (National Heart, Lung, and Blood Institute [NHLBI], 2011).

Captain Edwards documented a cause of Shell Shock in Private F. Private F was deaf, and mute when he entered the hospital, he also reported having a headache upon entry. Captain Edwards described Private F as being depressed, irritable, and talking to himself while experiencing Shell Shock. Captain Edward’s patient’s symptoms are associated with the emotional distress that he experienced during his time in war. Shell Shock’s symptoms are associated with the mental state of a soldier and they inhibit their emotional state.

This is the stage of the injury and will become the start of the rehabilitation process. When an injury occurs your body reacts immediately to heal it. This is often referred as the primary response mechanism. The main symptoms of primary damaged response are pain, bleeding and also inflammation.

Charging to 200....Stand clear…Shocking! CPR....EMS providers experience the adrenaline and rush of a patient in cardiac arrest. Trying to bring dead back to life is not a simple task by far, especially with the limitations and resources of the field. But, what happens after the patient makes it to definitive care? Annually, around 300,000 adults in the United States experience out-of-hospital cardiac arrests (AHA), and EMS providers only see the results of the short term survival of the patient, but rarely the actual patient care and recovery after an arrest. Patients undergo intense, aggressive treatment and recovery measures in the hospital post-code. These patients have a variety of treatment regimens

According to information published by the Mayo Clinic, sepsis has three stages: sepsis, severe sepsis and septic shock. The mortality rate for septic shock is nearly 50 per cent, and an episode of severe sepsis increases the risk of future infections. Severe sepsis causes blood flow to the vital organs, such as the brain, heart and kidneys, to become impaired. Sepsis can also cause blood clots to form in organs and extremities such as arms, legs, fingers and toes, which can lead to organ failure and tissue death (gangrene).

An anaphylactic reaction (anaphylaxis) is a sudden, severe allergic reaction that affects multiple areas of the body. Affected areas of the body may include the skin, mouth, lungs, heart, or gut (digestive system). Anaphylaxis can be life-threatening. This condition requires immediate medical attention, and sometimes hospitalization.

Dugdale, D. (2012, June 22). Cardiogenic Shock. U.S National Library of Medicine. Retrieved November 23, 2013, from http://www.nlm.nih.gov/medlineplus/ency/article/000185.htm

As sepsis progresses, tissues become less perfused and acidotic, compensation begins to fail, and the patient begins to show organ dysfunction. The cardiovascular system also begins to fail, the blood pressure does not respond to fluid resuscitation and vasoactive agents, and signs of end-organ damage are evident (e.g., renal failure, pulmonary failure, hepatic failure). As sepsis progresses to septic shock, the blood pressure drops, and the skin becomes cool, pale, and mottled. Temperature may be normal or below normal. Heart and respiratory rates remain rapid. Urine production ceases, and multiple organ dysfunction progressing to death occurs. Adventitious lung sounds occur throughout the lung fields, not just in the upper fields of the lungs.

This paper will be going over a scenario involving a real patient and what things could have been different with EMS care. It will also be covering what exactly cardiac arrest is and what rhythms produce it. And for every cardiac rhythm in cardiac arrest, there is a specific treatment plan paramedics can follow.

Septic shock is a subset of severe sepsis and was defined as “sepsis-induced hypotension persisting despite adequate fluid resusci- tation” (see Fig. 12.1). While the quantity of fluid that qualifies as “adequate fluid resuscitation” is controversial, we believe septic shock is best defined as a “mean arterial pressure (MAP) less than 65 mmHg after a fluid challenge of 20 mL/Kg body weight (given 30–60 minutes) in patients with sepsis and in the absence of other causes for hypotension” (also see Chap. 14). According to the ACCP/SCCM defini- tions, three stages in the hierarchy of the host’s response to infection were recognized, namely, sepsis, severe sepsis and septic shock, with sepsis having the best prognosis and septic shock the worst. While the use of the SIRS criteria to define sepsis is some- what controversial [3–6], many consider sepsis to be best defined as the “systemic response to infection with the presence of some degree of organ dysfunction”

Shock is a collapse of Circulatory function caused by severe injury, blood loss, or disease, and characterized by pallor sweating, weak pulse, and very low blood pressure .There are three basic types of shock. Cardiogenic Shock [includes tension pneumothorax, cardiac tamponede and pulmonary embolism] which is caused by the heart failing to pump as designed. Distributive shock [includes septic shock, neurogenic shock, anaphylactic shock and psychogenic shock] which is caused by poor vessel function. Lastly we have hypovolemic shock [includes hemorrhagic shock and non hemorrhagic shock]. Cardiogenic shock is basically shock that is cardiac in nature. It is also the end off of the road for all other causes of shock. Cardiogenic shock

55). According to Wen-Chih et al. (2010, p.11), ageing patients are susceptible to blood loss since they have limited physiological replacement. Thus, considering the age and the surgical procedure the patient is at high risk of hypovolemia. If the loss of blood progresses and the body is unable to compensate, the patient will be at risk for hypovolemic shock (D’Angelo, M., Dutton, R., 2009, p. 279). Hypovolemic shock means deficit in flowing blood causing to ineffectively filing the intravascular space. Mamaril, M., Child, S., & Sortman, S., 2007, p. 191. Kolecki, P., & Menckhoff, C. (2014) emphasizes that there are four (4) classification of hypovolemic shock. Class 1 is when there is 0-15% of blood loss with only slight change in heart rate. If the patient heart rate becomes more than 100 beats per minute and they experience tachypnoea and has cool clammy skin, the patient is in the class 2 of hypovolemic shock. Class 3 and 4 is when the patient will manifest the severe sympathetic response of the body due to the inability of compensate with the blood loss. Thus, considering the age and the surgical procedure the patient is at high risk of hypovolemia or even the more severe