Myocardial Infarction: Echocardiographic Study

Carbon Monoxide (CO) is a colorless, odorless, tasteless, and poisonous gas. CNESST reported that 4 workers died at work of CO poisoning in the last 5 years. (25) CO could be fatal at certain concentration. Its effects can occur well before being aware enough of its presence. In Canada, more than 50 person lose their life each year from CO poisoning, of which an average of 11 deaths in Ontario. Every year significant number are suffering from CO poisoning, many of whom are permanently disabled. In Canada between 2000 and 2009, 380 accidental deaths were documented by CO. (27) Headache, nausea, burning eyes, fainting, confusion, and drowsiness are such alarm that you are exposed to CO. (28) Exposure to CO may be particularly dangerous for elderly,

The most common physiological process leading to a myocardial infarction is the occlusion of coronary arteries by a process known as atherosclerosis. In fact, atherosclerosis is present in more than 90% of persons with coronary heart disease.1

1. What is the common name for Mr. Thomas’ condition? Look this condition up in a reference

The first phase occurs within 24 hours. It is caused by thermal damage and inflammation to the upper airway. Intubation should be considered early because of swelling and possible loss of airway. When mechanical ventilation is initiated it is important that burn victims have a higher than normal minute ventilation because they require a higher metabolic rate. The tidal volume should be at 6-8 kilograms per ideal body weight and respiratory rate should be 14 -16 breathes per minute. In the early phase, we also worry about carbon monoxide poisoning. Normal carbon monoxide levels in a healthy, non-smoking adult is usually around 5%, When CO levels are higher symptoms of headaches, dizziness, dyspnea weakness, impaired judgement, convulsions, coma, tachypnea, tachycardia, and hypotension may be present. This is caused by CO taking the site of oxygen on the red blood cell. This shifts the O2- HB curve to the left. To diagnose carbon monoxide poisoning a coximeter is needed. Treatment for CO poisoning is 100% FIO2 until CO level return to 5-10%. (There was not a carboxyhemoglobin reading for this patient all I could find was a pending reading. This would have been an easy noninvasive test that could have been perform quickly in the

In recent times, women have been increasingly more prone to heart related diseases. Even with campaigns that are run across internationally, there is a lack of realization by many people that the leading causes of deaths in women today are heart diseases. Often women are misdiagnosed by the healthcare providers and their symptoms are not recognized when they are having an acute cardiac arrest. According to statistics, every 30 seconds in the United States of America, a woman suffers from a heart attack (Health, 2013). Out of many types of heart diseases, the myocardial infarction is one of the leading causes of death in women.

There are many races, hurdles, and obstacles to contend with throughout the course of life such as: life itself, family and health. In the realm of Certified Nursing, I have become more intent on knowing more about the lifestyles of the people, and the good and bad results of daily tasks that ensure their happiness. Safety, and the many diseases/disorders that have been encountered by others, can only be counter-acted by better knowledge, resources, and means of assurance in providing ways of increasing the (ADLs). While reading, I came across the disorder of Myocardial Infarctions, and found the information to be remarkably interesting. First of all, I learned that infarctions are commonly known for being obstructions of the blood supply to the organs and tissues; whether by the thrombus or embolous. This obstruction causes tissues to die. ” (Medicine Net, 2015).There’s different kinds of infarctions, such as: Myocardial, Acute, Cerebellar, and Right Ventricular. Therefore, myocardial infarctions are “irreversible necrosis of heart muscle, which are secondary to prolonged ischemia” (emedicine, 2015). Therefore, myocardial infarctions are “irreversible necrosis of heart muscle, which are secondary to prolonged ischemia” (emedicine, 2015).

to a chronic Carbon Monoxide (CO) poisoning at lower concentrations from airplane’s fuel emissions. The amount of CO she inhaled on a daily basis was equivalent of smoking 100-800 cigarettes. Plaintiff suffered from migraine-like headaches, chest pain, blurring of vision and irregular heartbeat, she was tired and disoriented all the time. All symptoms indicate that CO presence in her blood was close to 50% and very close to a deadly level (Exhibit 2)

Coronary heart disease (CHD), also known as ischemic heart disease is the most prevalent form of cardiovascular disease in Australia (Australian Institute of Health and Welfare, 2014). While over 20,000 of deaths in 2011 were attributed to CHD. There were estimated 590,000 Australians 18 years old and above diagnosed with CHD in 2011-2012 (AIHW, 2014; Craft, 2014, pg. 596). Myocardial ischaemia is a common form of CHD. A sufficient coronary artery blood flow is essential to supply oxygen for normal cardiac activities. Myocardial ischaemia develops when there is an insufficient supply of blood and oxygen to support the function of myocardial cells (Craft, 2014, pg. 599). A decrease in blood supply can led to the formation of atherosclerotic plaques by narrowing or occluding the arteries. Other conditions such as hypotension, coronary spasm, dysthymias, hypoxemia and anaemia can also decrease the blood and oxygen supply to the myocardial cells (McCance & Huether, 2014, pg. 1153)

Binding of CO to hemoglobin is greatly affected to anemic and smoking individuals. Hemoglobin plays a major role in our body to transport oxygen. Since hemoglobin has a higher affinity for oxygen, it binds to an oxygen molecule and increases its oxygen concentration. Thus, when the blood cells are at a different part of the body where the oxygen concentrations are low, the oxygen will leave the hemoglobin and diffuse into the cells. However, carbon monoxide has a 250 fold greater affinity than oxygen. So, when CO enters the blood from the lungs, CO would bind with hemoglobin instead of oxygen, and block the blood’s ability to carry oxygen to the cells throughout the body. In a normal healthy individual, the total carboxyhemoglobin (COHb) complex is 1% or less, whereas, smoking increases COHb to 3-8% and further to 15% for chain smokers. When smoking a cigarette, CO binds to hemoglobin and displaces to

Carbon monoxide is harmful when it is inhaled, because it displaces oxygen in the blood and deprives the heart, brain, and vital organs of oxgyen causing

Carbon monoxide is a by-product of natural gas, with potentially deadly effects. When inhaled, this toxic gas can cause unconsciousness, permanent brain damage, and even death. Furthermore, carbon monoxide is completely odorless, tasteless, and colorless, making it almost impossible to detect without specialized equipment or

The aim of the study was to explore the connection between the neutrophil/lymphocyte ratio and the functional capacity in patients diagnosed with idiopathic dilated cardiomyopathy. 37 patients with idiopathic dilated cardiomyopathy participated in the treadmill test to evaluate their functional capacity. The mean age of the patients that participated in the study was 46.7 to 11.7 years, with 81% of the participants being male. Echocardiographic variables and a baseline clinical were attained from the patients. Also, the hematological and biochemical limitations were measured using customary methods. The patients were then separated into low and high functional capacity groups based on the functional rank that they received in the exercise test

Some clinical studies reported electrophysiological changes by RNS in patients with OP poisoning and attributed these changes to IMS (Avasthi and Singh, 2000). Findings of other experimental and clinical studies documented that IMS is associated with rhabdomyolysis which is followed by a proportionate rise in CPK and LDH levels (Bhattacharya et al., 2011).

A heart attack, also known as a myocardial infarction, usually occurs when a blood clot forms inside a coronary artery at the site of an atherosclerotic plaque. The blood clot severely limits or completely cuts off blood flow to part of the heart. In a small percentage of cases, blood flow is cut off when the muscles in the artery wall contract suddenly, constricting the artery. This constriction, called vasospasm, can occur in an artery that is only slightly narrowed by atherosclerosis or even in a healthy artery. Regardless of the cause of a heart attack, the oxygen deprivation is so severe and prolonged that heart muscle cells begin to die for lack of oxygen. About 1.1 million people in the United States have a heart attack every year;

Since the Industrial Revolution, there has been an increased presence of pollutants and carcinogens in the environment, specif-ically in urban areas.1 The presence of airborne pollutants can lead to an increase in allergic reactions and asthma rates. A common airborne pollutant is carbon monoxide (CO), which is commonly found in diesel exhaust. It is an odorless gas produced from me-thane and non-methane hydrocarbon oxidation.2 The toxic gas eliminates and takes the place of oxygen in the form of carboxy-hemoglobin when it binds to hemoglobin in red blood cells. It also affects the binding sites of heme groups, which complicates the transfer of oxygen to tissues.3 The majority of deaths from carbon monoxide poisoning is brought on from car exhaust.4 Between the years 1979 and 1988, 57% of deaths from carbon monoxide poi-soning were brought from car exhaust. There tend to be higher rates of carbon monoxide poisoning from car exhaust in northern regions, where temperatures are relatively low (Figure 1). 5