Myocardial Infarction Research

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LeMone, Burke, & Bostick (2012) states that an acute myocardial infarction (AMI) is when blood flow to a portion of cardiac muscle is obstructed, which causes prolonged tissue ischaemia and irreversible hypoxaemic cell necrosis and tissue damage. It is the necrosis of myocardial cells caused by a coronary occlusion which is usually due to the rupture of a complicated atherosclerotic lesion (LeMone, Burke, & Bostick, 2012).
Farrell & Dempsey (2014) suggests that the main cause of myocardial infarction (MI) is the underlying coronary artery disease (CAD), such as atherosclerosis. Atherosclerosis, is the abnormal accumulation of lipids, fatty substances or fibrous tissue in the lining of arterial blood vessels. These deposits, called atheromas, cause a narrowing or blockage of the arterial lumen which reduces blood flow to the myocardium. If the atheroma becomes too large it may rupture or haemorrhage into a plaque. The ruptured plaque becomes a site for thrombus formation, and this thrombus may then obstruct coronary blood flow resulting in an AMI
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For both men and women, IHD resulted in more Australian death than any other single cause (Nichols et al., 2015). From 2011-2012, 22% of Australian adults had CVD, while in 2013-2014 11% of all hospitalisations where attributed to CVD. In 2012, CVD was the underlying cause of 30% of all deaths (Australian Institute of Health and Welfare, 2015). Nichols et al. (2015) stated that IHD was the leading cause of death for aboriginal and torres strait islander (ATSI) people, with a death rate from AMI 2.5 times higher than the general population. IHD is among the top four leading causes of death for people 35 years and older, accounting for 6% of deaths aged 35-44 years old, and 19% of deaths 95 years and older (Nichols et al.,
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