I was born in 1998 with Patent Ductus Arteriosus (PDA). The ductus arteriosus is only open when a child is in the womb and should close after birth to prevent the oxygenated blood from mixing with the deoxygenated blood in the heart. PDA put a tremendous strain on my heart and lungs and not too long after birth; the doctors put me on a ventilator. A surgery was suggested, called PDA ligation, which involved closing the PDA with a small clip. The doctors had explained to my parents that the surgery may not be successful and if it was, there could be certain lasting effects such as being blind, deaf or mental retardation.
Patent ductus arteriosus (PDA), one of the more common cardiac defects at birth, is the persistence of an opening between the pulmonary artery and the descending thoracic aorta. This opening is as a result of failure of the physiological fetus ductus arteriosus to close, which normally occurs soon after birth. This hole allows for oxygenated blood from the aorta to mix with oxygen-depleted blood from the pulmonary artery. As a consequence, significant strain is placed on the heart and pressure within the lungs’ arteries is dramatically increased.
The heart leaked a small VSD doesn't require surgery to close the hole. After birth, your baby's doctor will observe and treat the symptoms while waiting to see if the heart of the leak can be shut down by itself.
The incidence of PDA is approximately 0.02 to 0.04 percent in term infants and 20 to 60 percent in preterm neonates. Patent ductus arteriosus accounts for six to eleven percent of all congenital heart defects. PDA is found twice as often in females than in males (Shinde, Basantwani, & Tendolkar, 2016). The incidence of PDA is increased in children who are born prematurely, children with a history of perinatal asphyxia, and, possibly, children born at high altitude. Up to 30 percent low birth infants develop PDA (Kim, 2016).
G4 P3003 (4 Gestations, 3 Full Term, 0 Preterm, 0 Miscarriages, 3 Currently Living); 3 Spontaneous Vaginal Deliveries; Last birth was 7 years ago by SVD, weighed 4000 grams; No previous obstetrical complications or morbidity; No past medical history; No past surgical history; No prior antenatal care
She is without complaints. She has not noted any increase in preterm labor. No signs/symptoms or change in pelvic pressure. She is compliant with bedrest and has help taking care of her son. She is otherwise aware that she should discontinue Motrin next week and is aware of the signs/symptoms that we are monitoring. The placental cord insertion does appear marginal as noted on prior ultrasound and we are following monthly growth. She is aware that after surveillance of cervical length which will the last one we would anticipate would be next week at 32 weeks and after that we would still recommend monthly evaluation of fetal growth. Preterm labor precautions were reviewed. She is scheduled to return in one
This particular case study involves a 29 year old obstetric patient who presented to the labor and delivery unit at 33 weeks gestation with complaints of abdominal pain for the past three days that had become more severe and absence of fetal movement noted since the previous evening. Her obstetric history revealed she has one living child and has had one previous miscarriage at ten weeks
Patient is a G1P0, 23-year-old Caucasian female patient, LMP is January 10, 2013, EDC is October 17, 2006 and gestation of 384/7 weeks confirmed by an ultrasound per chart. Pre-pregnancy weight was 110 lbs and pregnancy weight is 145 lbs for a total gain of 35 pounds. Patient stated she eats a normal diet, does not drink alcohol and she has never smoked cigarettes nor taken any kind of recreational drugs. Was admitted 0430 hrs on October 5, 2014 with intact membranes with 2cm dilation and 50% effaced. Birthing plan shows she plans on a natural birth, patient states “I plan on following my birthing plan to have no drugs during my labor, but am open to hearing my choices.” Patient plans on breastfeeding her infant for at least 1 year. Supportive husband was at bedside throughout the labor, delivery and postpartum.
On ultrasound there is a live fetus in cephalic presentation. Fetal biometry is consistent with dates. A detailed anatomic survey was fairly unremarkable although slightly suboptimal due to late gestational age. There were no gross abnormalities seen. The placenta is anterior. Amniotic fluid was 8.3 cm. Umbilical artery Doppler was within normal limits.