Partial Anomalous Pulmonary Venus Return

Atrioventricular Canal Defect is an abnormality that causes the mixing of blood. There is a hole in center of heart where the wall between the upper and lower chambers meet. The tricuspid and mitral valves aren’t formed properly and one large valve crosses the defect. The defect lets oxygen rich blood pass to the heart’s right side and mix with deoxygenated blood, then go back to the lungs. Another abnormality is Atrial Septal Defect (ASD), where the walls of the upper chambers of the heart don’t close completely, causing a left to right movement of blood due to the higher pressure. The mixing of oxygenated and deoxygenated blood may cause the right atrium and ventricle to enlarge due to the higher volume of blood.

Congenital heart disease is a cardiovascular condition resulting from an abnormality in the structure of the heart. The exact time this defect forms is unknown but it is during the fetal development in the womb. Researchers think the defect could be case partially by genetics and medical conditions of the fetus and the parents. If the baby’s mother had rubella, while pregnant or other conditions such as diabetes could have caused the defect. Some medications that were taken by a pregnant mother could also have caused birth defects. An error on chromosome 22 could also have caused the heart defect. Some symptoms that a person may have a congenital heart may not show up until later in life but many children also have the symptoms. Symptoms of a congenital heart defect are abnormal heartbeat, bluish tint to skin, shortness of breath, dizziness or fainting, and swelling of body tissues and organs.

AS can occur in the congenital period or later in life, but is more common in adults due to lack of exercise, and nutrition causing calcium buildup. If it is congenital it will occur within the first eight weeks of pregnancy, which the cause is unknown. In a child with AS, the pressure is much higher than normal in the left chamber pumping. Blood that has leaked back into the left ventricle through the valve in between heartbeats, causing the ventricle to be enlarged. There are three stages of AS. Stage A occurs in patients with bicuspid aortic valve or aortic sclerosis who are symptom free but are at risk for AS. In stage B people have progressive AS with mild or moderate calcify valve leaflets, leaflets mobility, or mild or

Some diagnosing can be made at birth or close to birth because of heart problems, an unusually wide neck or swelling of the hands and the feet.

A ventricular septal defect or VSD is a heart defect. It’s congenital, this means that a baby is born with it. It is an opening or hole in the dividing wall called the Septum, between the two lower chambers of the heart, right and left ventricles. Ventricular septal defects are the most common type of congenital heart defects. A VSD allows oxygen-rich blood to pass from the left ventricle through the opening in the septum. Then it mixes with oxygen-poor blood in the right ventricle. There are different types of ventricular septal defects, the type your child has depends on which part of the wall between the ventricles is involved. Also the size of the opening or hole varies. A ventricular septal defect may occur more often in some families, this is because of gene defects. But most of the time, the cause is unknown. Similarly to ventricular septal defect, an Atrial septal defect is a birth defect of the heart in which there is also a hole in the wall in the septum, but instead of dividing the lower chambers it divides the upper chambers of the heart (atria), here a hole can vary in size and may close on its own or may require surgery as well. An atrial septal defect is one type of congenital heart defect, in which also the baby is born with it.

4) A chest X-ray reveals a cardiac silhouette that is normal in diameter. Does this rule out a possible problem with Martha's heart? Explain. No, a normal diameter of a cardiac silhouette does not rule out a problem with Martha’s heart. The heart adapts and will compensate for damage in order to still function optimally. The right ventricle, in this case, will become stronger in order to push the same amount of blood (stroke volume) through the narrowed pulmonary semi-lunar valve. This thickening doesn’t necessarily change the inner diameter.

Critical Congenital Heart Defect (CCHD) is a subgroup of congenital heart defects that normally necessitates surgical intervention or catheterization procedure during the neonatal period (Good, Canale, Goodman, & Yeager, 2015). According to Mahle et al. (2009), although there are measures taken to detect CCHD like prenatal ultrasound and comprehensive newborn physical examination during newborn’s birth hospitalization, there is still a significant percentage of newborns with CCHD that is left undetected and discharged home without being diagnosed. Delay in diagnosis of CCHD increases morbidity and mortality (Mahle et al., 2009). In response to this, Riede et al. (2010) mentioned that the diagnostic gap in CCHD could be bridged by screening newborns using pulse oximetry

During the process of amniocentesis, usually done between 15 and 20 weeks of pregnancy, a needle is inserted through the abdomen and into the uterus in order to retrieve a sample of amniotic fluid for testing. As for CVS, throughout the cervix a thin tube is inserted in order to extract a small sample of the placenta for testing. This is usually done between 10 and 12 weeks of pregnancy. If pregnant or considering it is recommended you and your partner be tested to see if you are carriers for the gene. When one or both partners are carriers of the disease genetic testing is typically done. If both partners carry the gene for the disease their child has a 25% chance of developing the disease (Elsevier, 2007). In order to inherit the disease the child must receive 2 copies of the defective gene, one from each parent. A blood test can be performed after birth in order to see if your child has the disease. The most common form of the disease is classic infantile. Between 3 and 6 months of age signs will begin to show in this case. Progression is fairly quick and children with the disease usually will not live past the age of 4. The disease also causes severe mental and physical side effects. Initial symptoms can include seizures, listlessness, decreased eye contact, delayed mental and social skills, slow body growth, increasing irritability, increased startle reaction, and the infant may stop smiling, crawling or rolling over, and lose the ability to grasp and reach out (Weiss, 2010). Symptoms that occur later on may include deafness, blindness, a loss of intellectual skills, difficulties eating, a loss of motor skills, and abnormal body tone (Weiss, 2010). In juvenile Tay-Sachs symptoms typically appear between the ages of 2 and 5. The disease progresses more slowly, but death usually occurs by the time the child

parent due to sexual desire over the opposite-gendered parent (incestuous desire). My interpretation of this is that Venus was a boy (shown by masculine head) who was trapped in incestuous sexual desire (covered eyes) who masturbated to the image of his mother (enlarged hand), so his father claimed his castrated penis (penis is the color of the man).

During the Upper Paleolithic era artists created a wide range of small sculptures. These sculptures were made from various materials, including ivory, bone, clay, and even stone. They represented humans, as well as animals; they even combined them at times. Most of the sculptures from this time show a high level of skill. From this time, there were two very influential sculptures, The Venus of Willendorf and the Venus of Laussel. This paper will discuss both sculptures, in detail, as well as, compare and contrast them.

From March 6 to April 15, 2017, Venus will go retrograde and this could mean a new type of love based relationship in your life. However, from a cosmic perspective, this could potentially turn into other problems that you have not expected. In other words, this not a time for you to change the status of relationships or transition into new ones.

Echocardiography is the initial diagnostic modality for a patient with suspected congenital heart disease. In some patients, however, use of this modality is encumbered by its limited ability to delineate great arteries and intra cardiac anomalies, pulmonary veins, and coronary arteries. (7)

Atrial Septal Defect(ASD) is a very large problem concerning the heart in its overall function. When the heart, being the core of the cardiovascular system, has issues; it effects the rest of the body as a result. The core of the problem resides in the atrial septum. Normally the heart is divided into four separate chambers. But a person with atrial septal defect has an atrial septum that allows the blood from the left side of the heart back into the right side. This results in increased pulmonary blood flow and diastolic overload of the right ventricle. By having this constant left-to-right shunt, it can alter the pulmonary vascular resistance leading to hypertension or even the reversal of the shunt itself.

Atrial Septal Defect is a common and interesting disorder. Atrial Septal Defect is mostly know as “a hole in the heart.” This disorder is birth defect. It can happen to male, female, adult, and kids. This disorder mostly happens to kids and infant. Atrial Septal Defect is when there is a hole in your heart that doesn’t allow oxygen to flow through. This can causes difficulty breathing, heart murmur, and e.t.c... There three different types of level. These level are called ostium secundum, ostium primum, and sinus venosus. The most severe level is ostium secundum which is the true atrial septal. Then is ostium primum, and last sinus venosus. This genetic disorder is typically diagnosed by noticing .symptoms.

Patent ductus arteriosus (PDA), one of the more common cardiac defects at birth, is the persistence of an opening between the pulmonary artery and the descending thoracic aorta. This opening is as a result of failure of the physiological fetus ductus arteriosus to close, which normally occurs soon after birth. This hole allows for oxygenated blood from the aorta to mix with oxygen-depleted blood from the pulmonary artery. As a consequence, significant strain is placed on the heart and pressure within the lungs’ arteries is dramatically increased.