Pathophysiology Of Postoprative Hypovolaemia And The Body 's Compensation Mechanisms Essay
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1. Pathophysiology of Postoprative Hypovolaemia and the body’s compensation mechanisms:
Presented with the post-operative assessments of Mr. George Jensen who suffered from an open fracture of his right tibia and fibula and underwent an Open Reduction and External Fixation procedure, a nurse can tell that he is experiencing post-operative Hypovolaemia.
Hypovolaemia pertains to an abnormal loss of blood volume and body fluid. It is a life-threatening condition brought on by low blood venous return to the heart that results to decreased Cardiac Output (CO), leading to a lack of adequate circulatory filling, reduced organ blood flow and organ damage (Brown & Edwards, 2013; Craft, Gordon, Huether, Tiziani, McCance, & Brashers, 2013; Plenderleith, 2007).
There are numerous factors responsible for the onset of hypovolaemia. But among the most common causes and relevant to Mr. Jensen are:
o Haemorrhage from trauma, or surgery. It is the common cause of body fluid loss, and it may be internal or external. In the limbs, a considerable amount of blood can be lost to a fractured tibia (Page & McKinney, 2012, p.157).
A patient who undergoes surgery such as an open reduction external fixation fracture under general anaesthesia can futher have an estimated blood loss in volume of 200cc, as suggested by Mendelson, Kates, Pacos, Clark, & Wu (2011, p. 3).
o Dehydration that can be a result not just of poor fluid intake but also of badly managed Type 2 Diabetes Mellitus. Blood