Poop Pills: Article Analysis

Clostridium Difficile (C-Diff) is considered one of the most common infections a patient can acquire within their hospital stay. It is estimated that C-Diff is responsible for 337,000 infections and 14,000 deaths a year (Centers for Disease Control and Prevention, 2012). Working in the emergency department (ED), I have witness first hand how debilitating this gastrointestinal infection can be. Patients are admitted to the ED for having severe watery diarrhea, abdominal pain, and fever. Elderly patients are at increase risk for sepsis and dehydration related to recurrent infections. Appropriate management and education of C-Diff is optimal for patient survival and decrease contamination across lifespan.

Clostridium difficile also known as C-diff is a pathogen. According to Doctor Michelle Wright, It lives harmlessly in the gut of many people. About three in hundred healthy adults and as many as seven in ten healthy babies have a number of c difficile bacteria living in their gut (2015). Healthy people normally have a certain number of c-diff bacterium living in their gut, but this bacterium is usually kept in check by all the other harmless bacteria that lives in our gastrointestinal tract. Clostridium difficile

Clostridium difficile was discovered and isolated from neonates in 1935. It was initially considered a component of the fecal flora of newborns and not thought to be pathogenic (Keessen, Gaastra, & Lipman, 2010). The history of C. diff and other antibiotic resistant pathogens are closely related with the history of antibiotics. The first antibiotic discovered was penicillin by Alexander Fleming while working with Staphylococcus. With this discovery, a surge of natural and synthetic drugs was discovered to treat bacterial infections. During the 1970s, clindamycin and cephalosporins were highly used as an effective antibiotic against bacterial infection but at the same time disrupted the normal, healthy bowel flora, allowing C.

Clostridium difficile is a spore forming, anaerobic, toxin-producing, gram-positive bacillus that is the most common cause of nosocomial, antibiotic-associated diarrhea (15-25%).1,2,3 The pathogenesis of C. difficile-associated diarrhea (CDAD) is the result of broad spectrum antibiotics, such as clindamycin, flouroquinolones or ceftriaxone, which reduces the population of normal bowel flora and allowing for an overgrowth of C. difficile.1,2 The toxins synthesized by C. difficile, A and B, lead to the inflammation and damage of the intestinal mucosa creating the symptoms of C. difficile infection (CDI). These symptoms can range from asymptomatic carriers, to mild diarrhea to sudden and occasionally deadly colitis. The clinical practice guidelines for the treatment of CDAD recommends the use of metronidazole (MET) and vancomycin (VAN) that is dependent upon the severity of the CDI.1,2,3

These single celled tube-like organism finds a home in the gut of every human during their lifetime. If the person affected does not have the proper bacterial flora to balance Clostridium difficle, the bacteria will manifest itself. The bacteria produces a toxin that breaks down the outer and inner linings of the small and large intestine causing the condition, Clostridium difficle Colitis. The impaired intestine will cause the body to have up to 20 bowel movements a day. The bowel matter will eventually turn to water and blood, causing extreme malnourishment and dehydration. The bacterial infection, if not intervened will cause morbidity. According to Roos (2015), the CDC reported there are half- million cases and thirty-thousand deaths each year in the United States. Clostridium difficle is one of the world’s leading causes of deaths in assisted living facilities (Roos,

Clostridium difficile infection is a suprainfection cause by prolong use of antibiotics. Board spectrum antibiotic such as Penicillins, clindamycin, and cephalosporins are the antimicrobial drugs most commonly associated with C difficile colitis. According to Owens, in his research, C. difficile is primarily acquired in hospitals. Spread by spores, it can colonize a patient’s gut after helpful gut bacteria are killed by antibiotics. Its toxins can cause severe diarrhea and colitis, and it can be fatal (Owens 2013). On the other hand Kim in his research agreed clostridium difficile infection has been considered a hospital-acquired infection. However, a recent population-based study found 41% of CDIs were actually community acquired. It is becoming apparent that community acquired CDI affects populations previously thought to be at low risk; younger patients and patients who had no exposure to antibiotics in the 12 weeks before the infection. Thus, it is necessary to advocate and teach patient about

Clostridium difficile is a gram-positive spore-forming bacteria; first isolated and published in 1935 by Hall and O’Toole. This was accomplished from the stool of a healthy infant. However, it was not until 1978 that C. difficile was identified as being associated with many cases of antibiotic-resistant diarrhea.1 C. difficile has since become one of the leading causes of nosocomial infections in the United States. A study conducted in 2015 has estimated that C. difficile infection has an incident of 453,000 (95% confidence interval) in the United States.2 This has placed a tremendous burden on the US healthcare system. According to data from 2008, C. difficile infections have accumulated an excess cost of $4.8 billion to the US healthcare system.3

Clostridium Difficile, or C. diff, is a common occurrence in hospitals. But how does one know that they are in fact dealing with a patient with C. diff and not just diarrhea? It all lies in the symptoms. C. diff diarrhea has a distinct odor. The patient also feels a constant urgency, a need to always go to the restroom. The stool contains mucous. Beyond that, most patients will present with a low grade fever, mild leukocytosis, hyperactive bowel sounds, and mild abdominal tenderness. (Headly, 461)

Would you rather take poop pills or eat cockroach brains? It may seem nasty, but it’s actually good. I’m not saying that it do or don't taste good, I’m just saying that it’s good for your body. Medical professionals should be able to use questionable cures like poop pills and roaches because they can help your body in many ways, they can fight infectious diseases or they can restore the good bacteria that you need.

I looked up the laxative, Senna, because I was unaware of this type of laxative. It’s actually a laxative you can purchase without a prescription. Moreover, one of the surprising things I learned about Senna, was using it while taking birth control pills can decrease the effect of the birth control pills. Therefore, you may become pregnant if your not using any other methods of being safe.

Scientist are starting to make pills that can cure C-Differ and its curing millions of people that have the infection. The infection causes many problems with the human body.you will become nauseated. Have severe diarrhea and a lot of cramping. Doctors have been working for years on some type of antibiotic to cure C-Differ and they finally found the perfect pill. There are pills made from feces that come from other human beings. If you go on youtube you can find do it yourself videos and you could have a huge discount from the normal because treatments cost a fortune.The treatments also hurt a

The medicine that is created has positive aspects to them also. They help cure bacteria that could make you really sick. In “Cockroaches could be good medicine”, it say, “Ground-up tissue from cockroach and locust brains and locust nerves, however, killed almost all the harmful bacteria”. This shows that the cockroach can cure the diseases. That still don mean it will work for everybody. Another example is in, “Frozen Poop Pills Fight Life-Threatening Infections”, it say, “Doctors have recently discovered that a fecal transplant will restore good gut bacteria that banishes the C. diff.”. This shows that the poop get rid of that infection.

The article about how poop pills cured serious gut infections was about how the ingredients in the pills worked together to kill the infection in the gut. Doctors work together to find out why poop pills can cure gut infections and how this works. The dangerous infection kills up to 14,000 people per year and the poop pill kills bacteria in the gut and fights of the serious infection.

Most of the public have heard of broad-spectrum drugs, especially in terms of antibiotic resistance, because they fight a wide range of bacteria but also kills normal flora in the gut (Haddox, 2013). The loss of this gut flora can lead to an abnormal growth of harmful bacteria such as clostridium difficile (C-Diff). The four “C” antibiotics that have a high risk for patient to develop C-diff are clindamycin, cephalosporins, coamoxiclav, and ciprofloxacin (Haddox, 2013). These antibiotics have the highest risk of leading to C-diff development, however all antibiotics increase a patient’s likelihood of a C-diff infection. This effect can last up to 12 weeks post antibiotic administration (Haddox, 2013).