Reflection on Mentorship

1831 Words Oct 29th, 2015 8 Pages
Nursing Management of a Patient with Raised Intracranial Pressure after Traumatic Brain Injury and Dealing with Family Anxiety.

Introduction

The aim of this assignment is to explore, analyse and evaluate the nursing management of raised intracranial pressure (ICP) and family anxiety after head injury. In order to obtain a wider knowledge of the care of patients with raised ICP, a literature review was carried out. From this information I hope to improve the standard of care and ultimately patient outcome. The anxiety felt by family members are large and therefore I have decided to discuss this in my assignment. Consent has been sought from the next of kin. Confidentiality will be maintained in accordance with Nursing and
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The brain has an ability to tolerate increase intracranial volume without increasing the intracranial pressure, known as compliance (Barker 2002). Some intracranial contents (blood and cerebro spinal fluid) can be displaced into spinal cord(space) (Woodrow,2000). This displacement creates initial compliance, but once compensation is exhausted, intracranial pressure rises sharply (Chitnavis & Polkey 1998),squeezing brain tissue causing herniation and likely death.

Aldi’s ICP was monitored by passing a catheter into his lateral ventricle connected to a transducer and monitor known as ICP monitor. A normal ICP is 0-10mmhg ( Hickey 2002).ICP that is persistent >15mmhg is termed as “intracranial hypertension and will impair cerebral perfusion and cerebral blood flow (Garner,2007).Therefore early identification of increases in ICP using ICP monitoring enables prompt treatment which could decrease morbidity and mortality rates (Mestecky,2007). Aldi’s ICP was 30 mmhg, ICP guided therapy commenced which would be discuss. Due to word restrictions only 4 main strategies will be discuss in depth.

As there was an increased in Aldi’s ICP concern would be the ability of his brain to be perfuse. Normally, autoregulation which defines by Drummond (1990,p.118)) as “the intrinsic ability of the cerebral blood vessel to dilate or constrict in response to

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