Respiratory Failure Essay

The new versus classic BPD features have changed over the years. The approaches to care, including surfactant administration, permissive hypercapnia, and noninvasive ventilation have changed. All these has increased the survival of low birth weight infants as before with classic BPD. The classic BPD was before surfactant and more management techniques, and inflammation and alveolar septal fibrosis. All these changes were associated with oxygen toxicity, infection, and barotrauma.

Premature babies sometimes have apnea. It may happen together with a slow heart rate. Respiratory distress syndrome or RDS is a breathing problem most common in babies born before 34 weeks of pregnancy.” Babies with RDS don’t have a protein called surfactant that keeps small air sacs in the lungs from collapsing. “Intraventricular hemorrhage or IVH is bleeding in the brain. It usually happens near the ventricles in the center of the brain. A ventricles is a space in the brain that’s filled with fluid. Patent ductus ateriosus or PDA is a heart problem that happens in the connection between two major blood vessels near the heart. If the ductus do not close properly after birth, a baby can have breathing problems or heart failure. Heart failure is when enough blood can’t get pumped into the heart causing it to shut down. Necrotizing enter colitis (NEC) is a problem with a baby’s intestines. It causes feeding problems, a swollen belly and diarrhea. It sometimes happens 2 to 3 weeks after a premature bay has been born. Retinopathy of prematurity (ROP) is an abnormal growth of blood vessels in the eye. ROP can lead to vision loss. Jaundice is when a baby's eyes and skin look yellow. A baby has jaundice when his liver isn't fully developed or isn't working well. Anemia is when a baby doesn’t have enough healthy red blood cells to carry oxygen to the rest of the body. Bronchopulmonary dysplasia (BPD) is a lung condition that can develop in premature babies as well as babies who have treatment with a breathing machine. Babies with BPD sometimes develop fluid in the lungs, scarring and lung damage. Premature babies often have trouble fighting off germs because their immune systems are not fully formed. Infections that may affect a premature baby include pneumonia, a lung infection; sepsis, a blood infection; and meningitis, an infection in the fluid around the brain and

The list of proposed chronic abnormalities is lengthy. To this date, research has confirmed the following: 1) SIDS is due to a dysfunction of the cardiac and/or respiratory systems, and 2) the death of the infant is due to hypo-ventilation of the lungs and periods of complete cessation of breathing or apnea. Hypo-ventilation and apnea cause hypo-perfusion of the tissues with necessary oxygen. Ischemia of tissues results and eventually causes death. Research now centers around discovering the cause of infant hypo-ventilation and apnea.

I chose SIDS for my respiratory disease because I had heard about it before, but I never knew that there was a possibility that it had to do with your respiratory system. SIDS is Sudden Infant Death Syndrome. SIDS is common with infants younger than 3 months. It has no explanation yet, but it most commonly occurs to infants when they are sleeping. This is such an awful disease/condition because prior to SIDS the babies are usually healthy and well. Although there is no cause, researchers found that it might be caused by an abnormality in your brain that controls your breathing.

Postnatal respiratory complications among newborns are common. The most commonly reported cause of neonatal respiratory distress is transient tachypnea of the newborn (TTN), with an estimated incidence of 1% to 2% of in all newborns.1

One way which is very important is placing the infant on its back to sleep. This aids in helping the child breathe and keeps the air ways open and clear. Taking everything out the infants crib including sheets, toys and pillows can help prevent SIDS. Anything in the crib has the ability to affect the infants breathing. The infant should be placed in a bare crib on a firm mattress to sleep. Overheating the baby is also a factor that can be prevented by using a sleep sack or the bare minimum of clothing. In order to keep the baby warm you could use a light sheet that only covers the infant’s shoulders to their feet. It is not recommended to place a sheet over the infant’s head. This could also overheat the baby and cause the babies airways to be blocked. The baby should sleep alone at all times. Allowing the infant to sleep with you creates many risk for SIDS to develop. It is also a suffocation risk. There have been many cases where infants were accidentally killed by the parents rolling over on them. An infant should be in his or her crib alone at all times during sleep. Breastfeeding you baby for the first six months is also said to help prevent SIDS. Breastmilk reduces respiratory infections and improves the infant’s coordination in breathing while swallowing. Premature babies must learn how to coordinate breathing and swallowing.

Necrosis of the cells in the small, lower airways occurs, and mucous secretions are increased (Conquest, Cremonesini, & Neill, 2013). Because of the ciliary damage in the infants’ lungs, it is almost impossible for the secretions to be cleared. Bronchiolar level obstruction is caused by these mucosusal secretions, as is desquamation of the dead skin cells and edema (Conquest, Cremonesini, & Neill, 2013). Plugs of soughed, necrotic epithelium and fibrin in the airways will cause partial or total obstruction to airflow, making it very difficult for he infant to exhale which will consequently result in air becoming trapped and will reduce gaseous exchange (Conquest, Cremonesini, & Neill, 2013).

In this article, DiBlasi argues that the conventional method used to provide ventilatory support to preterm neonates with respiratory distress syndrome; nasal continuous positive airway pressure (CPAP) is ineffective. The author bases the claim on the fact that almost half of the infants supported by this technique often develop respiratory failure that warrants invasive ventilatory support and endotracheal intubation that is injurious in nature. According to the author, invasive ventilatory procedures should be avoided to minimize the excessive complications that are usually associated with them.

Sudden infant death syndrome (SIDS) is a mysterious phenomena that has managed to confound science so far. SIDS is defined as the sudden death of any infant or young child that is unexpected by history and in which a thorough post mortem examination fails to demonstrate an adequate cause (Hunt & Brouillette, 1987). It is the leading cause of death in infants in developed countries occurring at a rate of almost 2 per 1000 births. It most often occurs in infants between the ages of one month and eight months with the highest occurrence in the 4-6 month range. Several behaviors have been associated with this syndrome such as prolonged sleep apnea, increased risk of breath inspiration pause, excessive periodic

The innovation of surfactant replacement therapy in the treatment of respiratory distress syndrome has proven to increase the survival and minimize the complications of the premature neonate. Replacing surfactant has lessened time on ventilators, and allowing the neonate and parents an opportunity to grow together earlier outside of intensive care. This paper will discuss the etiology of respiratory distress syndrome type I, the treatment options and nursing care of the neonate during surfactant replacement.

Increases in heart and respiratory rate can lead to increased oxygen consumption and caloric requirements, which will allow for fewer calories available for growth and healing. If stress continues, the initial increase in heart and respiratory rates will cease, and the infant can become bradycardic and apneic, which could lead to a possible hypoxic event and decreased pulmonary function. Premature infants are exceptionally vulnerable because their neurologic systems are still very immature and they cannot selectively limit stimuli or its physiologic impact. During noisy periods, apnea was found most frequently in very preterm babies, and oxygen saturation averaged 90 percent compared to 93 percent during quiet periods (Brown, 2009).

Difficulty of Use IV. Indications for Bubble CPAP in Neonatal Patient a. Neonatal Respiratory Distress Syndrome (RDS) i. Definition of Neonatal RDS ii. Characteristics of RDS in Different Gestational Ages 1. Diagnosis 2.

The placenta is a major source of fetal prostaglandins and its removal after birth allows for the lungs to expand and become metabolically active, where most prostaglandins are degraded. This, in combination with increased pulmonary oxygen tension, in healthy full-term infants, normally causes functional closure of the ductus arteriosus within 15 hours after birth.

The most serious of these being hyaline membrane disease (HMD), most commonly known as RDS or respiratory distress syndrome. Babies that suffer from this condition will tend to find it very difficult to breathe due to the increased surface tension and the ultimate lack of oxygen transported through the body can seriously impair and damage the functions of the brain and other organs of the baby. Surfactant deficiency’s can also sometimes be caused by mutations in a particular gene. An example of this is surfactant protein or SP-B deficiency. This particular dysfunction is hereditary and is caused by a gene mutation on chromes number two. Babies that suffer from tis condition rarely survive past a few months. However there are some procedures that can help babies who may have been born prematurely and have not manufactured enough surfactants to support regular functions. Surfactant replacement therapy and surfactant supplements are amongst the most popular treatments of these types of surfactant deficiencies in newly born

Respiratory distress syndrome (RDS) is a common lung disorder that mostly affects preterm infants. RDS is caused by insufficient surfactant production and structural immaturity of the lungs leading to alveolar collapse. Clinically, RDS presents soon after birth with tachypnea, nasal flaring, grunting, retractions, hypercapnia, and/or an oxygen need. The usual course is clinical worsening followed by recovery in 3 to 5 days as adequate surfactant production occurs. Research in the prevention and treatment of this disease has led to major improvements in the care of preterm infants with RDS and increased survival. However, RDS remains an important cause of morbidity and mortality especially in the most preterm infants. This chapter reviews the most current evidence-based management of RDS, including prevention, delivery room stabilization, respiratory management, and supportive care.